Syndecan-4 is essential for development of concentric myocardial hypertrophy via stretch-induced activation of the calcineurin-NFAT pathway
about
Transmembrane proteoglycans control stretch-activated channels to set cytosolic calcium levels.Syndecan-4 Is an Independent Predictor of All-Cause as Well as Cardiovascular Mortality in Hemodialysis PatientsRANTES/CCL5 mediated-biological effects depend on the syndecan-4/PKCα signaling pathwayThe Soft- and Hard-Heartedness of Cardiac Fibroblasts: Mechanotransduction Signaling Pathways in Fibrosis of the Heart.Contribution of syndecan-4 genetic variants to hypertension, the TAMRISK studySyndecan-4 Regulates Muscle Differentiation and Is Internalized from the Plasma Membrane during Myogenesis.The Heparan Sulfate Proteoglycan Glypican-6 Is Upregulated in the Failing Heart, and Regulates Cardiomyocyte Growth through ERK1/2 Signaling.Syndecan 4 signaling is required for exercise-induced cardiac hypertrophy.Fibronectin contributes to pathological cardiac hypertrophy but not physiological growthParsing good versus bad signaling pathways in the heart: role of calcineurin-nuclear factor of activated T-cells.Protein kinase C, focal adhesions and the regulation of cell migration.Syndecan and integrin interactomes: large complexes in small spaces.Emerging biomarkers for heart failure: an update.Vascular smooth muscle cell in atherosclerosis.Wnt Signaling Cascades and the Roles of Syndecan Proteoglycans.Cardiomyocyte Ca2+ dynamics: clinical perspectives.Syndecans in heart fibrosis.Calcineurin signaling in the heart: The importance of time and place.Syndecan-4: a component of the mechanosensory apparatus of cardiac fibroblasts.Syndecan 4 controls lymphatic vasculature remodeling during mouse embryonic development.Cytoprotective activated protein C averts Nlrp3 inflammasome-induced ischemia-reperfusion injury via mTORC1 inhibition.Increased hypertrophic response with increased mechanical load in skeletal muscles receiving identical activity patterns.Overexpression of integrin α11 induces cardiac fibrosis in mice.Syndecan-4 is a key determinant of collagen cross-linking and passive myocardial stiffness in the pressure-overloaded heart.Lumican is increased in experimental and clinical heart failure, and its production by cardiac fibroblasts is induced by mechanical and proinflammatory stimuli.
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P2860
Syndecan-4 is essential for development of concentric myocardial hypertrophy via stretch-induced activation of the calcineurin-NFAT pathway
description
2011 nî lūn-bûn
@nan
2011 թուականին հրատարակուած գիտական յօդուած
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2011 թվականին հրատարակված գիտական հոդված
@hy
2011年の論文
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2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
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2011年論文
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2011年论文
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name
Syndecan-4 is essential for de ...... f the calcineurin-NFAT pathway
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Syndecan-4 is essential for de ...... f the calcineurin-NFAT pathway
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Syndecan-4 is essential for de ...... f the calcineurin-NFAT pathway
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type
label
Syndecan-4 is essential for de ...... f the calcineurin-NFAT pathway
@ast
Syndecan-4 is essential for de ...... f the calcineurin-NFAT pathway
@en
Syndecan-4 is essential for de ...... f the calcineurin-NFAT pathway
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prefLabel
Syndecan-4 is essential for de ...... f the calcineurin-NFAT pathway
@ast
Syndecan-4 is essential for de ...... f the calcineurin-NFAT pathway
@en
Syndecan-4 is essential for de ...... f the calcineurin-NFAT pathway
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P2093
P2860
P50
P3181
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Syndecan-4 is essential for de ...... f the calcineurin-NFAT pathway
@en
P2093
Alexandra V Finsen
Almira Hasic
Biljana Skrbic
Cathrine R Carlson
Even K Østli
Geir Florholmen
Hilde O Jarstadmarken
Ida G Lunde
Marianne Lyngra
Paul F Goetinck
P2860
P304
P3181
P356
10.1371/JOURNAL.PONE.0028302
P407
P577
2011-01-01T00:00:00Z