An NK Cell Perforin Response Elicited via IL-18 Controls Mucosal Inflammation Kinetics during Salmonella Gut Infection - Test2 - elhamkhani - TriplyDB

An NK Cell Perforin Response Elicited via IL-18 Controls Mucosal Inflammation Kinetics during Salmonella Gut Infection

An NK Cell Perforin Response Elicited via IL-18 Controls Mucosal Inflammation Kinetics during Salmonella Gut Infection

http://www.wikidata.org/entity/Q27313075

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Programmed cell death as a defence against infection.Salmonella infection: Interplay between the bacteria and host immune system.NAIP-NLRC4 Inflammasomes Coordinate Intestinal Epithelial Cell Expulsion with Eicosanoid and IL-18 Release via Activation of Caspase-1 and -8.Epithelial Cell Inflammasomes in Intestinal Immunity and Inflammation.The NLRC4 Inflammasome.Tat-exported peptidoglycan amidase-dependent cell division contributes to Salmonella Typhimurium fitness in the inflamed gut The inflammasomes, immune guardians at defence barriers

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An NK Cell Perforin Response Elicited via IL-18 Controls Mucosal Inflammation Kinetics during Salmonella Gut Infection

http://www.wikidata.org/entity/Q27313075

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2016 nî lūn-bûn

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2016 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2016 թվականի հունիսին հրատարակված գիտական հոդված

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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name

An NK Cell Perforin Response E ...... uring Salmonella Gut Infection

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An NK Cell Perforin Response E ...... uring Salmonella Gut Infection

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An NK Cell Perforin Response E ...... uring Salmonella Gut Infection

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An NK Cell Perforin Response E ...... uring Salmonella Gut Infection

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An NK Cell Perforin Response E ...... uring Salmonella Gut Infection

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An NK Cell Perforin Response E ...... uring Salmonella Gut Infection

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An NK Cell Perforin Response E ...... uring Salmonella Gut Infection

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An NK Cell Perforin Response E ...... uring Salmonella Gut Infection

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An NK Cell Perforin Response E ...... uring Salmonella Gut Infection

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JOURNAL.PPAT.1005723

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An NK Cell Perforin Response E ...... uring Salmonella Gut Infection

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Alexander J Westermann

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Anna A Müller

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Boas Felmy

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Carolin Verbree

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Mikael E Sellin

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Sandra Gadient

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Tamas Dolowschiak

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P2860

The inflammasomes

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

NK cells in mucosal defense against infection

Mucosal Inflammatory Response to Salmonella typhimurium Infection

NADPH oxidase deficient mice develop colitis and bacteremia upon infection with normally avirulent, TTSS-1- and TTSS-2-deficient Salmonella Typhimurium

Salmonella transiently reside in luminal neutrophils in the inflamed gut

A simple screen to identify promoters conferring high levels of phenotypic noise

Granulocytes impose a tight bottleneck upon the gut luminal pathogen population during Salmonella typhimurium colitis

Stromal IFN-γR-signaling modulates goblet cell function during Salmonella Typhimurium infection

Salmonella enterica serovar typhimurium exploits inflammation to compete with the intestinal microbiota

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e1005723

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scholarly article

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10.1371/JOURNAL.PPAT.1005723

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6

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12

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Salomé LeibundGut-Landmann

Wolf-Dietrich Hardt

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2016-06-01T00:00:00Z

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27341123

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4920399

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