Motility and chemotaxis mediate the preferential colonization of gastric injury sites by Helicobacter pylori
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The role of motility and chemotaxis in the bacterial colonization of protected surfaces.Chemodetection and Destruction of Host Urea Allows Helicobacter pylori to Locate the Epithelium.Adhesion and Invasion of Gastric Mucosa Epithelial Cells by Helicobacter pyloriEpithelial Regeneration After Gastric Ulceration Causes Prolonged Cell-Type AlterationsPathobiology of Helicobacter pylori-Induced Gastric Cancer.Small-molecule inhibitors of the pseudaminic acid biosynthetic pathway: targeting motility as a key bacterial virulence factorDecoding the Chemical Language of Motile Bacteria by Using High-Throughput Microfluidic Assays.Co-regulation of Iron Metabolism and Virulence Associated Functions by Iron and XibR, a Novel Iron Binding Transcription Factor, in the Plant Pathogen XanthomonasStaying in Shape: the Impact of Cell Shape on Bacterial Survival in Diverse EnvironmentsHow Helicobacter pylori senses, targets and interacts with the gastric epithelium.Pyrrolidine dithiocarbamate restores gastric damages and suppressive autophagy induced by hydrogen peroxide.Trefoil Factor Peptides and Gastrointestinal Function.The Human Stomach in Health and Disease: Infection Strategies by Helicobacter pylori.Recent Advances in Helicobacter pylori Replication: Possible Implications in Adaptation to a Pathogenic Lifestyle and Perspectives for Drug Design.Role of Flagella in the Pathogenesis of Helicobacter pyloriThe Norepinephrine Metabolite 3,4-Dihydroxymandelic Acid Is Produced by the Commensal Microbiota and Promotes Chemotaxis and Virulence Gene Expression in Enterohemorrhagic Escherichia coli.Why do we still have Helicobacter Pylori in our StomachsColonization, localization, and inflammation: the roles of H. pylori chemotaxis in vivo.Identification of a cheZ-like gene in Azorhizobium caulinodans, a key gene in the control of chemotaxis and colonization of the host plant.The effect of bacterial chemotaxis on host infection and pathogenicity.Pathogenesis of Helicobacter pylori Infection.A computational model for how cells choose temporal or spatial sensing during chemotaxis.Injuries invite ulcer microbe
P2860
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P2860
Motility and chemotaxis mediate the preferential colonization of gastric injury sites by Helicobacter pylori
description
2014 nî lūn-bûn
@nan
2014 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Motility and chemotaxis mediat ...... y sites by Helicobacter pylori
@ast
Motility and chemotaxis mediat ...... y sites by Helicobacter pylori
@en
Motility and chemotaxis mediat ...... y sites by Helicobacter pylori
@nl
type
label
Motility and chemotaxis mediat ...... y sites by Helicobacter pylori
@ast
Motility and chemotaxis mediat ...... y sites by Helicobacter pylori
@en
Motility and chemotaxis mediat ...... y sites by Helicobacter pylori
@nl
prefLabel
Motility and chemotaxis mediat ...... y sites by Helicobacter pylori
@ast
Motility and chemotaxis mediat ...... y sites by Helicobacter pylori
@en
Motility and chemotaxis mediat ...... y sites by Helicobacter pylori
@nl
P2093
P2860
P1433
P1476
Motility and chemotaxis mediat ...... y sites by Helicobacter pylori
@en
P2093
Amy C Engevik
Andrea L Matthis
Chet Closson
Eitaro Aihara
Karen M Ottemann
Marshall H Montrose
Michael A Schumacher
Yana Zavros
P2860
P304
P356
10.1371/JOURNAL.PPAT.1004275
P407
P577
2014-07-01T00:00:00Z