HMGB1 attenuates cardiac remodelling in the failing heart via enhanced cardiac regeneration and miR-206-mediated inhibition of TIMP-3
about
High-mobility Group Box Protein-1, Matrix Metalloproteinases, and Vitamin D in Keloids and Hypertrophic ScarsMicroRNA-206: a potential circulating biomarker candidate for amyotrophic lateral sclerosisAlarmins: awaiting a clinical responseLocal activation of cardiac stem cells for post-myocardial infarction cardiac repairUp-regulated HMGB1 in EAM directly led to collagen deposition by a PKCβ/Erk1/2-dependent pathway: cardiac fibroblast/myofibroblast might be another source of HMGB1Heart structure-specific transcriptomic atlas reveals conserved microRNA-mRNA interactions.HMGB1 in health and disease.microRNA133a targets Foxl2 and promotes differentiation of C2C12 into myogenic progenitor cells.Extracellular high mobility group box 1 plays a role in the effect of bone marrow mononuclear cell transplantation for heart failure.High-mobility group box-1 impairs memory in mice through both toll-like receptor 4 and Receptor for Advanced Glycation End Products.High-Mobility Group Box 1 Promotes Hepatocellular Carcinoma Progression through miR-21-Mediated Matrix Metalloproteinase ActivityHigh-mobility group box 1 induces calcineurin-mediated cell hypertrophy in neonatal rat ventricular myocytesMicroRNA-214 Mediates Isoproterenol-induced Proliferation and Collagen Synthesis in Cardiac Fibroblasts.Differential regulation of extracellular tissue inhibitor of metalloproteinases-3 levels by cell membrane-bound and shed low density lipoprotein receptor-related protein 1.miRNA 206 and miRNA 574-5p are highly expression in coronary artery disease.Extracellular high-mobility group box 1 mediates pressure overload-induced cardiac hypertrophy and heart failure.Identification of cardiac-related circulating microRNA profile in human chronic heart failure.Transcriptional profiling of HMGB1-induced myocardial repair identifies a key role for Notch signalingGenetic comparison of mouse lung telocytes with mesenchymal stem cells and fibroblasts.MicroRNA-206: a promising theranostic marker.Angiotensin II upregulated the expression of microRNA-224 but not microRNA-21 in adult rat cardiac fibroblastsCardiovascular disease and high-mobility group box 1--is a new inflammatory killer in town?The therapeutic potential of miRNAs in cardiac fibrosis: where do we stand?Noncoding RNAs as epigenetic mediators of skeletal muscle regeneration.c-kit(+) cells: the tell-tale heart of cardiac regeneration?Converging pathways involving microRNA-206 and the RNA-binding protein KSRP control post-transcriptionally utrophin A expression in skeletal muscle.The Biological Basis for Cardiac Repair After Myocardial Infarction: From Inflammation to Fibrosis.Stem Cell-Derived Exosomes, Autophagy, Extracellular Matrix Turnover, and miRNAs in Cardiac Regeneration during Stem Cell Therapy.MiR-206 Suppresses the Progression of Coronary Artery Disease by Modulating Vascular Endothelial Growth Factor (VEGF) Expression.High mobility group box 1 protein attenuates myocardial ischemia reperfusion injury via inhibition of the p38 mitogen-activated protein kinase signaling pathway.Contrasting Inflammation Resolution during Atherosclerosis and Post Myocardial Infarction at the Level of Monocyte/Macrophage Phagocytic Clearance.miR-206 inhibits the growth of hepatocellular carcinoma cells via targeting CDK9.Angiotensin II induced differentially expressed microRNAs in adult rat cardiac fibroblasts.Thrombomodulin's lectin-like domain reduces myocardial damage by interfering with HMGB1-mediated TLR2 signalling.Exogenous high-mobility group box 1 protein prevents postinfarction adverse myocardial remodeling through TGF-β/Smad signaling pathway.Functional evolution of cardiac microRNAs in heart development and functions.Influence of middle-distance running on muscular micro RNAs.MicroRNA-206 Protects against Myocardial Ischaemia-Reperfusion Injury in Rats by Targeting Gadd45β.microRNA-206 correlates with left ventricular function after transcatheter aortic valve implantation.HMGB1 Inhibits Apoptosis Following MI and Induces Autophagy via mTORC1 Inhibition.
P2860
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P2860
HMGB1 attenuates cardiac remodelling in the failing heart via enhanced cardiac regeneration and miR-206-mediated inhibition of TIMP-3
description
2011 nî lūn-bûn
@nan
2011 թուականին հրատարակուած գիտական յօդուած
@hyw
2011 թվականին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
HMGB1 attenuates cardiac remod ...... -mediated inhibition of TIMP-3
@ast
HMGB1 attenuates cardiac remod ...... -mediated inhibition of TIMP-3
@en
HMGB1 attenuates cardiac remod ...... -mediated inhibition of TIMP-3
@nl
type
label
HMGB1 attenuates cardiac remod ...... -mediated inhibition of TIMP-3
@ast
HMGB1 attenuates cardiac remod ...... -mediated inhibition of TIMP-3
@en
HMGB1 attenuates cardiac remod ...... -mediated inhibition of TIMP-3
@nl
prefLabel
HMGB1 attenuates cardiac remod ...... -mediated inhibition of TIMP-3
@ast
HMGB1 attenuates cardiac remod ...... -mediated inhibition of TIMP-3
@en
HMGB1 attenuates cardiac remod ...... -mediated inhibition of TIMP-3
@nl
P2093
P2860
P50
P1433
P1476
HMGB1 attenuates cardiac remod ...... -mediated inhibition of TIMP-3
@en
P2093
Anna Di Carlo
Antonella Mangoni
Antonia Germani
Chiara Bertolami
Federica Limana
Grazia Esposito
Maurizio C Capogrossi
Sveva Romani
P2860
P304
P356
10.1371/JOURNAL.PONE.0019845
P407
P577
2011-01-01T00:00:00Z