Arginylation-dependent neural crest cell migration is essential for mouse development - Test2 - elhamkhani - TriplyDB

Arginylation-dependent neural crest cell migration is essential for mouse development

Arginylation-dependent neural crest cell migration is essential for mouse development

http://www.wikidata.org/entity/Q27348001

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The N-end rule pathway Factors controlling cardiac neural crest cell migration Small molecule inhibitors of arginyltransferase regulate arginylation-dependent protein degradation, cell motility, and angiogenesis Arginylation of myosin heavy chain regulates skeletal muscle strength Protein arginylation, a global biological regulator that targets actin cytoskeleton and the muscle The N-end rule pathway and regulation by proteolysis Arginyltransferase is an ATP-independent self-regulating enzyme that forms distinct functional complexes in vivo.Arginylation regulates purine nucleotide biosynthesis by enhancing the activity of phosphoribosyl pyrophosphate synthase Osteoblasts Have a Neural Origin in Heterotopic Ossification.Arginylation-dependent regulation of a proteolytic product of talin is essential for cell-cell adhesion.Characterization of arginylation branch of N-end rule pathway in G-protein-mediated proliferation and signaling of cardiomyocytes A novel spiroindoline targets cell cycle and migration via modulation of microtubule cytoskeleton.Post-translational modification and regulation of actin.Posttranslational arginylation enzyme Ate1 affects DNA mutagenesis by regulating stress response Loss of ATE1-mediated arginylation leads to impaired platelet myosin phosphorylation, clot retraction, and in vivo thrombosis formation.Posttranslational arginylation as a global biological regulator.Post-translational protein arginylation in the normal nervous system and in neurodegeneration.Arginylation regulates myofibrils to maintain heart function and prevent dilated cardiomyopathy.The N-end rule pathway is mediated by a complex of the RING-type Ubr1 and HECT-type Ufd4 ubiquitin ligases.Protein arginylation targets alpha synuclein, facilitates normal brain health, and prevents neurodegeneration Protein arginylation regulates cellular stress response by stabilizing HSP70 and HSP40 transcripts.Analyzing N-terminal Arginylation through the Use of Peptide Arrays and Degradation Assays.Arginyltransferase ATE1 is targeted to the neuronal growth cones and regulates neurite outgrowth during brain development.Arginyltransferase suppresses cell tumorigenic potential and inversely correlates with metastases in human cancers.Talin is cut out for intercellular adhesion.Rapid and dynamic arginylation of the leading edge β- actin is required for cell migration.

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Arginylation-dependent neural crest cell migration is essential for mouse development

http://www.wikidata.org/entity/Q27348001

description

2010 nî lūn-bûn

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2010 թուականի Մարտին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի մարտին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Arginylation-dependent neural crest cell migration is essential for mouse development

@ast

Arginylation-dependent neural crest cell migration is essential for mouse development

@en

Arginylation-dependent neural crest cell migration is essential for mouse development

@nl

type

label

Arginylation-dependent neural crest cell migration is essential for mouse development

@ast

Arginylation-dependent neural crest cell migration is essential for mouse development

@en

Arginylation-dependent neural crest cell migration is essential for mouse development

@nl

prefLabel

Arginylation-dependent neural crest cell migration is essential for mouse development

@ast

Arginylation-dependent neural crest cell migration is essential for mouse development

@en

Arginylation-dependent neural crest cell migration is essential for mouse development

@nl

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Arginylation-dependent neural crest cell migration is essential for mouse development

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Anna Kashina

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Caiying Guo

http://www.w3.org/2001/XMLSchema#string

Fangliang Zhang

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Junling Wang

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N Adrian Leu

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Ralph Bunte

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Satoshi Kurosaka

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Sougata Saha

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Wei He

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P2860

Alternative splicing results in differential expression, activity, and localization of the two forms of arginyl-tRNA-protein transferase, a component of the N-end rule pathway

Fate of the mammalian cardiac neural crest

Global analysis of posttranslational protein arginylation.

Identification of mammalian arginyltransferases that modify a specific subset of protein substrates

Msx2 deficiency in mice causes pleiotropic defects in bone growth and ectodermal organ formation

Menin is required in cranial neural crest for palatogenesis and perinatal viability

Transcription factor AP-2 essential for cranial closure and craniofacial development

A highly efficient recombineering-based method for generating conditional knockout mutations

Fate of the mammalian cranial neural crest during tooth and mandibular morphogenesis

Arginyltransferase regulates alpha cardiac actin function, myofibril formation and contractility during heart development.

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