Upregulation of Major Histocompatibility Complex Class I on Liver Cells by Hepatitis C Virus Core Protein via p53 and TAP1 Impairs Natural Killer Cell Cytotoxicity
about
The transcription factor p53: not a repressor, solely an activatorNatural killer cells in hepatitis C: Current progressNK cell function and receptor diversity in the context of HCV infectionInterferon Response in Hepatitis C Virus (HCV) Infection: Lessons from Cell Culture Systems of HCV InfectionInfluence of HIV and HCV on T cell antigen presentation and challenges in the development of vaccinesPathogenesis of West Nile Virus Infection: a Balance between Virulence, Innate and Adaptive Immunity, and Viral EvasionEffect of Killer Immunoglobulin-Like Receptors in the Response to Combined Treatment in Patients with Chronic Hepatitis C Virus InfectionInfluence of host resistance on viral adaptation: hepatitis C virus as a case studyBiogenesis of functional antigenic peptide transporter TAP requires assembly of pre-existing TAP1 with newly synthesized TAP2Extracellular HCV-core protein induces an immature regulatory phenotype in NK cells: implications for outcome of acute infectionChronically inflamed livers up-regulate expression of inhibitory B7 family members.Hepatitis C virus impairs natural killer cell activity via viral serine protease NS3.Recipients with in utero induction of tolerance upregulated MHC class I in the engrafted donor skin.Increased natural killer cell cytotoxicity and NKp30 expression protects against hepatitis C virus infection in high-risk individuals and inhibits replication in vitro.Inhibition of the HCV core protein on the immune response to HBV surface antigen and on HBV gene expression and replication in vivo.NK cells, innate immunity and hepatitis C infection after liver transplantation.Constitutive but not inducible attenuation of transforming growth factor β signaling increases natural killer cell responses without directly affecting dendritic cells early after persistent viral infection.Cell-to-cell contact with hepatitis C virus-infected cells reduces functional capacity of natural killer cells.Anti-hepatitis C virus T-cell immunity in the context of multiple exposures to the virus.Hepatitis C virus attenuates interferon-induced major histocompatibility complex class I expression and decreases CD8+ T cell effector functions.Role of NK and NKT cells in the immunopathogenesis of HCV-induced hepatitis.IRF-1 and p65 mediate upregulation of constitutive HLA-A antigen expression by hepatocellular carcinoma cells.Viral strategies for evading antiviral cellular immune responses of the host.Molecular signatures associated with HCV-induced hepatocellular carcinoma and liver metastasis.Hepatitis viruses: live and let die.Natural killer cells: multifaceted players with key roles in hepatitis C immunity.Natural killer cell function is intact after direct exposure to infectious hepatitis C virions.Hepatitis C virus (HCV) employs multiple strategies to subvert the host innate antiviral response.The natural killer cell response to HCV infection.Liver natural killer and natural killer T cells: immunobiology and emerging roles in liver diseases.Pseudomonas aeruginosa Cif protein enhances the ubiquitination and proteasomal degradation of the transporter associated with antigen processing (TAP) and reduces major histocompatibility complex (MHC) class I antigen presentation.Emerging roles of p53 and other tumour-suppressor genes in immune regulationThe effect of bovine viral diarrhea virus (BVDV) strains on bovine monocyte-derived dendritic cells (Mo-DC) phenotype and capacity to produce BVDV.Dendritic cells in hepatitis C infection: can they (help) win the battle?Innate immunity and hepatitis C virus infection: a microarray's view.Hepatitis C virus-mediated modulation of cellular immunity.Immune modulation by the hepatitis C virus core protein.Up-regulate HLA class I expression following hepatitis B virus transfection in a hepatocellular carcinoma cell line BEL7405.A synergistic interferon-gamma production is induced by mouse hepatitis virus in interleukin-12 (IL-12)/IL-18-activated natural killer cells and modulated by carcinoembryonic antigen-related cell adhesion molecules (CEACAM) 1a receptor.Partial Activation of natural killer and γδ T cells by classical swine fever viruses is associated with type I interferon elicited from plasmacytoid dendritic cells.
P2860
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P2860
Upregulation of Major Histocompatibility Complex Class I on Liver Cells by Hepatitis C Virus Core Protein via p53 and TAP1 Impairs Natural Killer Cell Cytotoxicity
description
2003 nî lūn-bûn
@nan
2003 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Upregulation of Major Histocom ...... tural Killer Cell Cytotoxicity
@ast
Upregulation of Major Histocom ...... tural Killer Cell Cytotoxicity
@en
Upregulation of Major Histocom ...... tural Killer Cell Cytotoxicity
@nl
type
label
Upregulation of Major Histocom ...... tural Killer Cell Cytotoxicity
@ast
Upregulation of Major Histocom ...... tural Killer Cell Cytotoxicity
@en
Upregulation of Major Histocom ...... tural Killer Cell Cytotoxicity
@nl
prefLabel
Upregulation of Major Histocom ...... tural Killer Cell Cytotoxicity
@ast
Upregulation of Major Histocom ...... tural Killer Cell Cytotoxicity
@en
Upregulation of Major Histocom ...... tural Killer Cell Cytotoxicity
@nl
P2093
P2860
P1433
P1476
Upregulation of Major Histocom ...... tural Killer Cell Cytotoxicity
@en
P2093
Axel Ulsenheimer
Barbara Seliger
Christine S Falk
Jens Encke
Kerstin Herzer
Peter H Krammer
Sören T Eichhorst
P2860
P304
P356
10.1128/JVI.77.15.8299-8309.2003
P577
2003-08-01T00:00:00Z