Spines, plasticity, and cognition in Alzheimer's model mice - Test2 - elhamkhani - TriplyDB

Spines, plasticity, and cognition in Alzheimer's model mice

Spines, plasticity, and cognition in Alzheimer's model mice

http://www.wikidata.org/entity/Q27691328

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Obstructive sleep apnea and cognitive impairment: addressing the blood-brain barrier Reevaluating Metabolism in Alzheimer's Disease from the Perspective of the Astrocyte-Neuron Lactate Shuttle Model From neurodevelopment to neurodegeneration: the interaction of neurofibromin and valosin-containing protein/p97 in regulation of dendritic spine formation Environmental enrichment and brain repair: harnessing the therapeutic effects of cognitive stimulation and physical activity to enhance experience-dependent plasticity Increased dendritic spine density and tau expression are associated with individual differences in steroidal regulation of male sexual behavior.Genetic modulation of soluble Aβ rescues cognitive and synaptic impairment in a mouse model of Alzheimer's disease Mechanisms of Alzheimer's Disease Pathogenesis and Prevention: The Brain, Neural Pathology, N-methyl-D-aspartate Receptors, Tau Protein and Other Risk Factors.Synaptic changes in the dentate gyrus of APP/PS1 transgenic mice revealed by electron microscopy.Textrous!: extracting semantic textual meaning from gene sets.Transcriptome meta-analysis reveals a central role for sex steroids in the degeneration of hippocampal neurons in Alzheimer's disease Evidence for Alzheimer's disease-linked synapse loss and compensation in mouse and human hippocampal CA1 pyramidal neurons.Tau causes synapse loss without disrupting calcium homeostasis in the rTg4510 model of tauopathy Resting state FMRI reveals diminished functional connectivity in a mouse model of amyloidosis.Rescue of tau-induced synaptic transmission pathology by paclitaxel.ZiBuPiYin recipe protects db/db mice from diabetes-associated cognitive decline through improving multiple pathological changes Impairments in experience-dependent scaling and stability of hippocampal place fields limit spatial learning in a mouse model of Alzheimer's disease.Pharmacological Selectivity Within Class I Histone Deacetylases Predicts Effects on Synaptic Function and Memory Rescue.Amyloid plaque formation precedes dendritic spine loss.Amyloid plaque pathogenesis in 5XFAD mouse spinal cord: retrograde transneuronal modulation after peripheral nerve injury Abnormal Population Responses in the Somatosensory Cortex of Alzheimer's Disease Model Mice.Neuregulin-1 attenuates cognitive function impairments in a transgenic mouse model of Alzheimer's disease.β amyloid peptide plaques fail to alter evoked neuronal calcium signals in APP/PS1 Alzheimer's disease mice Synaptic alterations in the rTg4510 mouse model of tauopathy.Altered Synapse Stability in the Early Stages of Tauopathy The intersection of amyloid β and tau in glutamatergic synaptic dysfunction and collapse in Alzheimer's disease.GABAergic neurotransmission and new strategies of neuromodulation to compensate synaptic dysfunction in early stages of Alzheimer's disease.Therapeutic Potential of Secreted Amyloid Precursor Protein APPsα.Therapeutic efficacy of the Ginkgo special extract EGb761® within the framework of the mitochondrial cascade hypothesis of Alzheimer's disease.Viral gene transfer of APPsα rescues synaptic failure in an Alzheimer's disease mouse model.MicroRNA-195 prevents dendritic degeneration and neuron death in rats following chronic brain hypoperfusion.The influence of phospho-τ on dendritic spines of cortical pyramidal neurons in patients with Alzheimer's disease.Cypermethrin Stimulates GSK3β-Dependent Aβ and p-tau Proteins and Cognitive Loss in Young Rats: Reduced HB-EGF Signaling and Downstream Neuroinflammation as Critical Regulators.Alzheimer's disease-related amyloid-β induces synaptotoxicity in human iPS cell-derived neurons.Targeting astrocytes ameliorates neurologic changes in a mouse model of Alzheimer's disease.Chronic γ-secretase inhibition reduces amyloid plaque-associated instability of pre- and postsynaptic structures.Spatial learning impairments in PLB1Triple knock-in Alzheimer mice are task-specific and age-dependent.Amyloid-β dimers in the absence of plaque pathology impair learning and synaptic plasticity.Mouse models of neurodegenerative disease: preclinical imaging and neurovascular component.Effects of curcumin on synapses in APPswe/PS1dE9 mice.Amyloid-Beta and Phosphorylated Tau Accumulations Cause Abnormalities at Synapses of Alzheimer's disease Neurons.

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Spines, plasticity, and cognition in Alzheimer's model mice

http://www.wikidata.org/entity/Q27691328

description

2011 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2011 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年论文

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Spines, plasticity, and cognition in Alzheimer's model mice

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Spines, plasticity, and cognition in Alzheimer's model mice

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Spines, plasticity, and cognition in Alzheimer's model mice.

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Spines, plasticity, and cognition in Alzheimer's model mice

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Spines, plasticity, and cognition in Alzheimer's model mice

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Spines, plasticity, and cognition in Alzheimer's model mice.

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Spines, plasticity, and cognition in Alzheimer's model mice

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Spines, plasticity, and cognition in Alzheimer's model mice

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Spines, plasticity, and cognition in Alzheimer's model mice.

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Neural Plasticity

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Spines, plasticity, and cognition in Alzheimer's model mice

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Knafo S

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Spires-Jones T

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Creative Commons Attribution 3.0 Unported

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Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor protein

Soluble oligomers of the amyloid beta-protein impair synaptic plasticity and behavior

Synapse elimination accompanies functional plasticity in hippocampal neurons

Reversal of long-term dendritic spine alterations in Alzheimer disease models

Tau accumulation causes mitochondrial distribution deficits in neurons in a mouse model of tauopathy and in human Alzheimer's disease brain

Dendritic function of tau mediates amyloid-beta toxicity in Alzheimer's disease mouse models

Naturally secreted oligomers of amyloid beta protein potently inhibit hippocampal long-term potentiation in vivo

Early phenotypic changes in transgenic mice that overexpress different mutants of amyloid precursor protein in brain

Association of missense and 5'-splice-site mutations in tau with the inherited dementia FTDP-17

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10.1155/2012/319836

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2012

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Tara L Spires-Jones

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Alzheimer's disease

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