Characterization of voltage-dependent sodium and calcium channels in mouse pancreatic A- and B-cells.
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The Physiology, Pathology, and Pharmacology of Voltage-Gated Calcium Channels and Their Future Therapeutic PotentialAction potentials and insulin secretion: new insights into the role of Kv channels.Roles of Voltage-Gated Tetrodotoxin-Sensitive Sodium Channels NaV1.3 and NaV1.7 in Diabetes and Painful Diabetic NeuropathyHigh-content screening identifies a role for Na(+) channels in insulin productionA K ATP channel-dependent pathway within alpha cells regulates glucagon release from both rodent and human islets of Langerhans.Multiparameter screening reveals a role for Na+ channels in cytokine-induced β-cell death.Characteristics and functions of {alpha}-amino-3-hydroxy-5-methyl-4-isoxazolepropionate receptors expressed in mouse pancreatic {alpha}-cellsGlucose suppression of glucagon secretion: metabolic and calcium responses from alpha-cells in intact mouse pancreatic islets.Glucose decouples intracellular Ca2+ activity from glucagon secretion in mouse pancreatic islet alpha-cellsIonic mechanisms and Ca2+ dynamics underlying the glucose response of pancreatic β cells: a simulation study.A computational systems analysis of factors regulating α cell glucagon secretion.Glucose and pharmacological modulators of ATP-sensitive K+ channels control [Ca2+]c by different mechanisms in isolated mouse alpha-cellsSodium channel beta1 regulatory subunit deficiency reduces pancreatic islet glucose-stimulated insulin and glucagon secretion.Insulin as a physiological modulator of glucagon secretion.Regulation of insulin synthesis and secretion and pancreatic Beta-cell dysfunction in diabetes.Modeling the pancreatic α-cell: dual mechanisms of glucose suppression of glucagon secretion.β-arrestin-2 is an essential regulator of pancreatic β-cell function under physiological and pathophysiological conditions.Functional identification of islet cell types by electrophysiological fingerprinting.Regulation of calcium in pancreatic α- and β-cells in health and disease.On depolarization-evoked exocytosis as a function of calcium entry: possibilities and pitfalls.Modulation of Ionic Channels and Insulin Secretion by Drugs and Hormones in Pancreatic Beta Cells.TRPM3 channels provide a regulated influx pathway for zinc in pancreatic beta cells.Unperturbed islet α-cell function examined in mouse pancreas tissue slices.Ryanodine receptor activation by Ca v 1.2 is involved in dendritic cell major histocompatibility complex class II surface expressionCyclic GMP kinase I modulates glucagon release from pancreatic α-cells.Molecular mechanisms involved in the non-monotonic effect of bisphenol-a on ca2+ entry in mouse pancreatic β-cells.Insulinotropic effect of high potassium concentration beyond plasma membrane depolarization.CaV1.2 and CaV1.3 channel hyperactivation in mouse islet β cells exposed to type 1 diabetic serum.Na+ current properties in islet α- and β-cells reflect cell-specific Scn3a and Scn9a expression.Sodium channel NaV1.3 is important for enterochromaffin cell excitability and serotonin release.ATP regulates sodium channel kinetics in pancreatic islet beta cells.Pancreatic β-Cell Electrical Activity and Insulin Secretion: Of Mice and Men.Biphasic voltage-dependent inactivation of human NaV 1.3, 1.6 and 1.7 Na+ channels expressed in rodent insulin-secreting cells.KCNJ11 tetramer:ABCC8 tetramer binds 4xATP; closing the channelCalcium Influx through Voltage-gated Calcium ChannelsMathematical modelling of local calcium and regulated exocytosis during inhibition and stimulation of glucagon secretion from pancreatic alpha-cells.Calcium Channels in Postnatal Development of Rat Pancreatic Beta Cells and Their Role in Insulin Secretion.Extracellular factors and immunosuppressive drugs influencing insulin secretion of murine isletsAMPA Receptors Regulate Exocytosis and Insulin Release in Pancreatic β CellsGLP-1 Localisation and Proglucagon Gene Expression in Healthy and Diabetic Mouse Ileum
P2860
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P2860
Characterization of voltage-dependent sodium and calcium channels in mouse pancreatic A- and B-cells.
description
2006 nî lūn-bûn
@nan
2006 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Characterization of voltage-de ...... use pancreatic A- and B-cells.
@ast
Characterization of voltage-de ...... use pancreatic A- and B-cells.
@en
Characterization of voltage-de ...... use pancreatic A- and B-cells.
@nl
type
label
Characterization of voltage-de ...... use pancreatic A- and B-cells.
@ast
Characterization of voltage-de ...... use pancreatic A- and B-cells.
@en
Characterization of voltage-de ...... use pancreatic A- and B-cells.
@nl
altLabel
Characterization of voltage-de ...... ouse pancreatic A- and B-cells
@en
prefLabel
Characterization of voltage-de ...... use pancreatic A- and B-cells.
@ast
Characterization of voltage-de ...... use pancreatic A- and B-cells.
@en
Characterization of voltage-de ...... use pancreatic A- and B-cells.
@nl
P2093
P2860
P3181
P1476
Characterization of voltage-de ...... use pancreatic A- and B-cells.
@en
P2093
Andrea Welling
Franz Hofmann
Sheila Vignali
Veronika Leiss
P2860
P304
P3181
P356
10.1113/JPHYSIOL.2005.102368
P407
P577
2006-05-01T00:00:00Z