TOR signaling is a determinant of cell survival in response to DNA damage. - Test2 - elhamkhani - TriplyDB

TOR signaling is a determinant of cell survival in response to DNA damage.

TOR signaling is a determinant of cell survival in response to DNA damage.

http://www.wikidata.org/entity/Q27938358

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Mechanistic Insights into Molecular Targeting and Combined Modality Therapy for Aggressive, Localized Prostate Cancer Sen1p contributes to genomic integrity by regulating expression of ribonucleotide reductase 1 (RNR1) in Saccharomyces cerevisiae.Identification of novel genes involved in DNA damage response by screening a genome-wide Schizosaccharomyces pombe deletion library A network of conserved damage survival pathways revealed by a genomic RNAi screen.DNA-PK and P38 MAPK: A Kinase Collusion in Alzheimer's Disease?Natural polymorphism in BUL2 links cellular amino acid availability with chronological aging and telomere maintenance in yeast.The ribonucleotide reductase inhibitor, Sml1, is sequentially phosphorylated, ubiquitylated and degraded in response to DNA damage Stage 2 combination testing of rapamycin with cytotoxic agents by the Pediatric Preclinical Testing Program Identification of two novel inhibitors of mTOR signaling pathway based on high content screening.Genome-wide single-cell-level screen for protein abundance and localization changes in response to DNA damage in S. cerevisiae.Mtor-Fanconi Anemia DNA Damage Repair Pathway in Cancer.Target of rapamycin (TOR) in nutrient signaling and growth control Tryptophan-Dependent Control of Colony Formation After DNA Damage via Sea3-Regulated TORC1 Signaling in Saccharomyces cerevisiae.Identification of pathways controlling DNA damage induced mutation in Saccharomyces cerevisiae.Mammalian target of rapamycin contributes to the acquired apoptotic resistance of human mesothelioma multicellular spheroids.mTOR Inhibitors Suppress Homologous Recombination Repair and Synergize with PARP Inhibitors via Regulating SUV39H1 in BRCA-Proficient Triple-Negative Breast Cancer.Regulation of FANCD2 by the mTOR pathway contributes to the resistance of cancer cells to DNA double-strand breaks DNA-dependent protein kinase and DNA repair: relevance to Alzheimer's disease.Regulation of protein synthesis by ionizing radiation.Transcription factor RFX1 is crucial for maintenance of genome integrity in Fusarium graminearum.The Intra-S Checkpoint Responses to DNA Damage.Integrated regulation of PIKK-mediated stress responses by AAA+ proteins RUVBL1 and RUVBL2.The DNA damage checkpoint response to replication stress: A Game of Forks.Emerging roles of radioresistance in prostate cancer metastasis and radiation therapy.Molecularly targeting the PI3K-Akt-mTOR pathway can sensitize cancer cells to radiotherapy and chemotherapy.P53 suppresses ribonucleotide reductase via inhibiting mTORC1.Genome-wide analysis of yeast stress survival and tolerance acquisition to analyze the central trade-off between growth rate and cellular robustness The expanding relevance of nuclear mTOR in carcinogenesis.mTOR is a selective effector of the radiation therapy response in androgen receptor-positive prostate cancer Affected chromosome homeostasis and genomic instability of clonal yeast cultures.TOR complex 2 controls gene silencing, telomere length maintenance, and survival under DNA-damaging conditions.mTOR regulates DNA damage response through NF-κB-mediated FANCD2 pathway in hematopoietic cells.TORC1 kinase and the S-phase cyclin Clb5 collaborate to promote mitotic spindle assembly and DNA replication in S. cerevisiae.mTORC1 and -2 Coordinate Transcriptional and Translational Reprogramming in Resistance to DNA Damage and Replicative Stress in Breast Cancer Cells.Targeting FANCD2 for therapy sensitization.Saccharomyces cerevisiae genes involved in survival of heat shock.DNA damage regulates direct association of TOR kinase with the RNA polymerase II-transcribed HMO1 gene.The mTOR-S6K pathway links growth signalling to DNA damage response by targeting RNF168.Ixr1 Regulates Ribosomal Gene Transcription and Yeast Response to Cisplatin.

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TOR signaling is a determinant of cell survival in response to DNA damage.

http://www.wikidata.org/entity/Q27938358

description

2007 nî lūn-bûn

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2007 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2007 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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name

TOR signaling is a determinant of cell survival in response to DNA damage.

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TOR signaling is a determinant of cell survival in response to DNA damage.

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TOR signaling is a determinant of cell survival in response to DNA damage.

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type

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TOR signaling is a determinant of cell survival in response to DNA damage.

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TOR signaling is a determinant of cell survival in response to DNA damage.

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TOR signaling is a determinant of cell survival in response to DNA damage.

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prefLabel

TOR signaling is a determinant of cell survival in response to DNA damage.

@ast

TOR signaling is a determinant of cell survival in response to DNA damage.

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TOR signaling is a determinant of cell survival in response to DNA damage.

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Molecular and Cellular Biology

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TOR signaling is a determinant of cell survival in response to DNA damage

@en

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Bin Shi

http://www.w3.org/2001/XMLSchema#string

Cynthia S Lancaster

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Hong Guo

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Mary-Ann Bjornsti

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Padma Thimmaiah

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P2860

Two TOR complexes, only one of which is rapamycin sensitive, have distinct roles in cell growth control

Signaling by target of rapamycin proteins in cell growth control

Tor-mediated induction of autophagy via an Apg1 protein kinase complex

TOR signaling in growth and metabolism

TOR controls translation initiation and early G1 progression in yeast

Esc4p, a new target of Mec1p (ATR), promotes resumption of DNA synthesis after DNA damage.

Molecular anatomy and regulation of a stable replisome at a paused eukaryotic DNA replication fork.

Yeast DNA damage-inducible Rnr3 has a very low catalytic activity strongly stimulated after the formation of a cross-talking Rnr1/Rnr3 complex.

CLB5-dependent activation of late replication origins in S. cerevisiae.

Molecular organization of target of rapamycin complex 2.

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10.1128/MCB.00290-07

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