Early and selective loss of neuromuscular synapse subtypes with low sprouting competence in motoneuron diseases
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Stronger is not always better: could a bodybuilding dietary supplement lead to ALS?Ubiquitin-Synaptobrevin Fusion Protein Causes Degeneration of Presynaptic Motor Terminals in MiceFrom animal models to human disease: a genetic approach for personalized medicine in ALSAdducin at the Neuromuscular Junction in Amyotrophic Lateral Sclerosis: Hanging on for Dear LifeEmerging Roles of Filopodia and Dendritic Spines in Motoneuron Plasticity during Development and DiseaseALS Patient Stem Cells for Unveiling Disease Signatures of Motoneuron Susceptibility: Perspectives on the Deadly Mitochondria, ER Stress and Calcium TriadMechanisms of Muscle Denervation in Aging: Insights from a Mouse Model of Amyotrophic Lateral SclerosisTRPM7 and its role in neurodegenerative diseasesMitochondria in motor nerve terminals: function in health and in mutant superoxide dismutase 1 mouse models of familial ALSStem cell transplantation for motor neuron disease: current approaches and future perspectivesA Drosophila model of ALS: human ALS-associated mutation in VAP33A suggests a dominant negative mechanismEarly functional deficit and microglial disturbances in a mouse model of amyotrophic lateral sclerosisGeneration of Functional Neuromuscular Junctions from Human Pluripotent Stem Cell Lines.MicroRNA-206: a potential circulating biomarker candidate for amyotrophic lateral sclerosisEGFR inhibitor erlotinib delays disease progression but does not extend survival in the SOD1 mouse model of ALSPostnatal muscle modification by myogenic factors modulates neuropathology and survival in an ALS mouse modelSpastic paraplegia mutation N256S in the neuronal microtubule motor KIF5A disrupts axonal transport in a Drosophila HSP modelModeling ALS with motor neurons derived from human induced pluripotent stem cellsALS as a distal axonopathy: molecular mechanisms affecting neuromuscular junction stability in the presymptomatic stages of the diseaseNeuronal matrix metalloproteinase-9 is a determinant of selective neurodegenerationElectrophysiologic biomarkers for assessing disease progression and the effect of riluzole in SOD1 G93A ALS miceAge-Related Changes in Pre- and Postsynaptic Partners of the Cholinergic C-Boutons in Wild-Type and SOD1G93A Lumbar MotoneuronsBulbar and speech motor assessment in ALS: challenges and future directions.Overexpression of wild-type androgen receptor in muscle recapitulates polyglutamine disease.miRNA malfunction causes spinal motor neuron disease.A genetic model of amyotrophic lateral sclerosis in zebrafish displays phenotypic hallmarks of motoneuron diseaseDeficits in axonal transport precede ALS symptoms in vivo.Elevated PGC-1α activity sustains mitochondrial biogenesis and muscle function without extending survival in a mouse model of inherited ALSMonitoring peripheral nerve degeneration in ALS by label-free stimulated Raman scattering imaging.Dynamic remodelling of synapses can occur in the absence of the parent cell bodyA conditioning lesion provides selective protection in a rat model of Amyotrophic Lateral Sclerosis.Nerve terminal degeneration is independent of muscle fiber genotype in SOD1 miceDownregulation of genes with a function in axon outgrowth and synapse formation in motor neurones of the VEGFdelta/delta mouse model of amyotrophic lateral sclerosis.Bioenergetic status modulates motor neuron vulnerability and pathogenesis in a zebrafish model of spinal muscular atrophy.Motor neuron vulnerability and resistance in amyotrophic lateral sclerosis.Complement C5a-C5aR1 signalling drives skeletal muscle macrophage recruitment in the hSOD1G93A mouse model of amyotrophic lateral sclerosis.The lack of CuZnSOD leads to impaired neurotransmitter release, neuromuscular junction destabilization and reduced muscle strength in mice.Characterization of intercostal muscle pathology in canine degenerative myelopathy: a disease model for amyotrophic lateral sclerosis.Treadmill gait analysis does not detect motor deficits in animal models of Parkinson's disease or amyotrophic lateral sclerosis.Optimised and rapid pre-clinical screening in the SOD1(G93A) transgenic mouse model of amyotrophic lateral sclerosis (ALS).
P2860
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P2860
Early and selective loss of neuromuscular synapse subtypes with low sprouting competence in motoneuron diseases
description
2000 nî lūn-bûn
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2000 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
Early and selective loss of ne ...... petence in motoneuron diseases
@ast
Early and selective loss of ne ...... petence in motoneuron diseases
@en
Early and selective loss of ne ...... petence in motoneuron diseases
@nl
type
label
Early and selective loss of ne ...... petence in motoneuron diseases
@ast
Early and selective loss of ne ...... petence in motoneuron diseases
@en
Early and selective loss of ne ...... petence in motoneuron diseases
@nl
prefLabel
Early and selective loss of ne ...... petence in motoneuron diseases
@ast
Early and selective loss of ne ...... petence in motoneuron diseases
@en
Early and selective loss of ne ...... petence in motoneuron diseases
@nl
P2093
P3181
P1476
Early and selective loss of ne ...... petence in motoneuron diseases
@en
P2093
P304
P3181
P356
10.1523/JNEUROSCI.20-07-02534.2000
P407
P577
2000-04-01T00:00:00Z