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X-ray Snapshot of the Mechanism of Inactivation of Human Neutrophil Elastase by 1,2,5-Thiadiazolidin-3-one 1,1-Dioxide Derivatives †Effect of plant neutrophil elastase inhibitor on leucocyte migration, adhesion and cytokine release in inflammatory conditionsFreezing the Bioactive Conformation to Boost Potency: The Identification of BAY 85-8501, a Selective and Potent Inhibitor of Human Neutrophil Elastase for Pulmonary DiseasesNeutrophil elastase cleaves epithelial cadherin in acutely injured lung epitheliumUtilization of the 1,2,3,5-thiatriazolidin-3-one 1,1-dioxide scaffold in the design of potential inhibitors of human neutrophil proteinase 3.Circulating endothelial microparticles involved in lung function decline in a rat exposed in cigarette smoke maybe from apoptotic pulmonary capillary endothelial cellsFunctional characterization of polymorphisms in the peptidase inhibitor 3 (elafin) gene and validation of their contribution to risk of acute respiratory distress syndrome.Effects of structure on inhibitory activity in a series of mechanism-based inhibitors of human neutrophil elastase.Neutrophil elastase, proteinase 3, and cathepsin G as therapeutic targets in human diseases.Combination therapy with sivelestat and recombinant human soluble thrombomodulin for ARDS and DIC patients.Sivelestat relieves respiratory distress refractory to dexamethasone in all-trans retinoic acid syndrome: a report of two casesDual function inhibitors of relevance to chronic obstructive pulmonary diseaseMacrophage-specific expression of human lysosomal acid lipase corrects inflammation and pathogenic phenotypes in lal-/- mice.Differentiation syndrome in promyelocytic leukemia: clinical presentation, pathogenesis and treatment.Impact of host proteases on reovirus infection in the respiratory tract.Inactivation of human neutrophil elastase by 1,2,5-thiadiazolidin-3-one 1,1 dioxide-based sulfonamidesCollectins and cationic antimicrobial peptides of the respiratory epithelia.Fibroblast Activation Protein (FAP) Accelerates Collagen Degradation and Clearance from Lungs in Mice.Genetic determinants of reovirus pathogenesis in a murine model of respiratory infectionAlpha-1 antitrypsin reduces severity of pseudomonas pneumonia in mice and inhibits epithelial barrier disruption and pseudomonas invasion of respiratory epithelial cellsIntra-alveolar tissue factor pathway inhibitor is not sufficient to block tissue factor procoagulant activity.Genetic polymorphisms of peptidase inhibitor 3 (elafin) are associated with acute respiratory distress syndrome.Acute respiratory distress syndrome after pulmonary resection.A Comprehensive Review of Punica granatum (Pomegranate) Properties in Toxicological, Pharmacological, Cellular and Molecular Biology Researches.Emerging drugs for acute lung injury.Neutrophil elastase inhibitors: a patent review and potential applications for inflammatory lung diseases (2010 - 2014).Neutrophil Fates in Bronchiectasis and Alpha-1 Antitrypsin Deficiency.Identification of Neutrophil Exocytosis Inhibitors (Nexinhibs), Small Molecule Inhibitors of Neutrophil Exocytosis and Inflammation: DRUGGABILITY OF THE SMALL GTPase Rab27a.The clinical practice guideline for the management of ARDS in Japan.A role for whey acidic protein four-disulfide-core 12 (WFDC12) in the regulation of the inflammatory response in the lung.Neutrophil elastase modulates cytokine expression: contribution to host defense against Pseudomonas aeruginosa-induced pneumonia.Regulation of apoptosis and priming of neutrophil oxidative burst by diisopropyl fluorophosphate.Expression and activity of N-myristoyltransferase in lung inflammation of cattle and its role in neutrophil apoptosisVentilator-induced endothelial activation and inflammation in the lung and distal organs.Escape from the matrix: multiple mechanisms for fibroblast activation in pulmonary fibrosis.Complement C5a is a key mediator of meconium-induced neutrophil activation.Neutrophil elastase activity in acute lung injury and respiratory distress syndrome.The secretory leukocyte protease inhibitor (SLPI) and the secondary granule protein lactoferrin are synthesized in myelocytes, colocalize in subcellular fractions of neutrophils, and are coreleased by activated neutrophils.Modulation of γδ T-cell activation by neutrophil elastase.Deficiency in neutrophil elastase does not impair neutrophil recruitment to inflamed sites.
P2860
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P2860
description
2003 nî lūn-bûn
@nan
2003 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Proteases and lung injury
@ast
Proteases and lung injury
@en
Proteases and lung injury
@nl
type
label
Proteases and lung injury
@ast
Proteases and lung injury
@en
Proteases and lung injury
@nl
prefLabel
Proteases and lung injury
@ast
Proteases and lung injury
@en
Proteases and lung injury
@nl
P2093
P3181
P1476
Proteases and lung injury
@en
P2093
Chung-Wai Chow
Gregory P Downey
Theo J Moraes
P304
P3181
P356
10.1097/01.CCM.0000057842.90746.1E
P407
P433
P577
2003-04-01T00:00:00Z