Functional ATPase activity of p97/valosin-containing protein (VCP) is required for the quality control of endoplasmic reticulum in neuronally differentiated mammalian PC12 cells
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Derlin-2 and Derlin-3 are regulated by the mammalian unfolded protein response and are required for ER-associated degradationValosin-containing protein (VCP/p97) is required for poliovirus replication and is involved in cellular protein secretion pathway in poliovirus infectionAtaxin-3 is a multivalent ligand for the parkin Ubl domainAtaxin-3 interactions with rad23 and valosin-containing protein and its associations with ubiquitin chains and the proteasome are consistent with a role in ubiquitin-mediated proteolysisEndoplasmic Reticulum Stress and Associated ROSInclusion body myopathy-associated mutations in p97/VCP impair endoplasmic reticulum-associated degradationThe AAA-ATPase p97 is essential for outer mitochondrial membrane protein turnoverPositive cooperativity of the p97 AAA ATPase is critical for essential functionsHDAC6-p97/VCP controlled polyubiquitin chain turnoverThe p97/VCP ATPase is critical in muscle atrophy and the accelerated degradation of muscle proteinsInvolvement of the p97-Ufd1-Npl4 complex in the regulated endoplasmic reticulum-associated degradation of inositol 1,4,5-trisphosphate receptorsMolecular components of a cell death pathway activated by endoplasmic reticulum stressInclusion body myopathy associated with Paget disease of bone and frontotemporal dementia is caused by mutant valosin-containing proteinEndotoxin conditioning induces VCP/p97-mediated and inducible nitric-oxide synthase-dependent Tyr284 nitration in protein phosphatase 2A.The slow Wallerian degeneration protein, WldS, binds directly to VCP/p97 and partially redistributes it within the nucleus.VCP associated inclusion body myopathy and paget disease of bone knock-in mouse model exhibits tissue pathology typical of human disease.SNIPER(TACC3) induces cytoplasmic vacuolization and sensitizes cancer cells to Bortezomib.A Mighty "Protein Extractor" of the Cell: Structure and Function of the p97/CDC48 ATPase.Pathogenic VCP/TER94 alleles are dominant actives and contribute to neurodegeneration by altering cellular ATP level in a Drosophila IBMPFD modelA Plastid Protein That Evolved from Ubiquitin and Is Required for Apicoplast Protein Import in Toxoplasma gondii.Valosin-containing protein (VCP) in novel feedback machinery between abnormal protein accumulation and transcriptional suppressionLys11- and Lys48-linked ubiquitin chains interact with p97 during endoplasmic-reticulum-associated degradation.Physical and functional interaction between Dorfin and Valosin-containing protein that are colocalized in ubiquitylated inclusions in neurodegenerative disorders.Liver cytochrome P450 3A endoplasmic reticulum-associated degradation: a major role for the p97 AAA ATPase in cytochrome P450 3A extraction into the cytosol.Neuronal remodeling and apoptosis require VCP-dependent degradation of the apoptosis inhibitor DIAP1.VCP Is an integral component of a novel feedback mechanism that controls intracellular localization of catalase and H2O2 Levels.The complexities of p97 function in health and diseaseTorsinA and torsion dystonia: Unraveling the architecture of the nuclear envelope.Valosin-containing protein gene mutations: cellular phenotypes relevant to neurodegeneration.Exclusion of the unfolded protein response in light-induced retinal degeneration in the canine T4R RHO model of autosomal dominant retinitis pigmentosa.Novel VCP modulators mitigate major pathologies of rd10, a mouse model of retinitis pigmentosa.Mislocalization to the nuclear envelope: an effect of the dystonia-causing torsinA mutation.Pre-B-cell leukemia transcription factor 1 regulates expression of valosin-containing protein, a gene involved in cancer growth.Valosin containing protein associated fronto-temporal lobar degeneration: clinical presentation, pathologic features and pathogenesis.Distinct roles for the AAA ATPases NSF and p97 in the secretory pathway.Misfolded proteins, endoplasmic reticulum stress and neurodegeneration.Protective role of cell division cycle 48 (CDC48) protein against neurodegeneration via ubiquitin-proteasome system dysfunction during zebrafish developmentRegulation of ubiquitin-proteasome system mediated degradation by cytosolic stress.Compartmentalization of superoxide dismutase 1 (SOD1G93A) aggregates determines their toxicity.A progressive translational mouse model of human valosin-containing protein disease: the VCP(R155H/+) mouse
P2860
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P2860
Functional ATPase activity of p97/valosin-containing protein (VCP) is required for the quality control of endoplasmic reticulum in neuronally differentiated mammalian PC12 cells
description
2002 nî lūn-bûn
@nan
2002 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Functional ATPase activity of ...... rentiated mammalian PC12 cells
@ast
Functional ATPase activity of ...... rentiated mammalian PC12 cells
@en
Functional ATPase activity of ...... rentiated mammalian PC12 cells
@nl
type
label
Functional ATPase activity of ...... rentiated mammalian PC12 cells
@ast
Functional ATPase activity of ...... rentiated mammalian PC12 cells
@en
Functional ATPase activity of ...... rentiated mammalian PC12 cells
@nl
prefLabel
Functional ATPase activity of ...... rentiated mammalian PC12 cells
@ast
Functional ATPase activity of ...... rentiated mammalian PC12 cells
@en
Functional ATPase activity of ...... rentiated mammalian PC12 cells
@nl
P2093
P2860
P356
P1476
Functional ATPase activity of ...... rentiated mammalian PC12 cells
@en
P2093
Akira Kakizuka
Keiko Tanaka
Kiyoshi Inoue
Taeko Kobayashi
P2860
P304
P356
10.1074/JBC.M207783200
P407
P577
2002-12-06T00:00:00Z