A chronic inflammatory response dominates the skeletal muscle molecular signature in dystrophin-deficient mdx mice - Test2 - elhamkhani - TriplyDB

A chronic inflammatory response dominates the skeletal muscle molecular signature in dystrophin-deficient mdx mice

A chronic inflammatory response dominates the skeletal muscle molecular signature in dystrophin-deficient mdx mice

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Corticosteroids for the treatment of Duchenne muscular dystrophy Glucocorticoid corticosteroids for Duchenne muscular dystrophy Gene expression comparison of biopsies from Duchenne muscular dystrophy (DMD) and normal skeletal muscle The Dietary Supplement Protandim® Decreases Plasma Osteopontin and Improves Markers of Oxidative Stress in Muscular Dystrophy Mdx Mice SPP1 genotype is a determinant of disease severity in Duchenne muscular dystrophy Osteopontin promotes fibrosis in dystrophic mouse muscle by modulating immune cell subsets and intramuscular TGF-beta The zebrafish candyfloss mutant implicates extracellular matrix adhesion failure in laminin alpha2-deficient congenital muscular dystrophy.Expression profiling of cytokines and related genes in regenerating skeletal muscle after cardiotoxin injection: a role for osteopontin Muscle regeneration in dystrophin-deficient mdx mice studied by gene expression profiling Anti-inflammatory drugs for Duchenne muscular dystrophy: focus on skeletal muscle-releasing factors Nutraceuticals and Their Potential to Treat Duchenne Muscular Dystrophy: Separating the Credible from the Conjecture Use of cell permeable NBD peptides for suppression of inflammation Glucocorticoid-induced leucine zipper (GILZ) and long GILZ inhibit myogenic differentiation and mediate anti-myogenic effects of glucocorticoids P2RX7 purinoceptor: a therapeutic target for ameliorating the symptoms of duchenne muscular dystrophy Hsp72 preserves muscle function and slows progression of severe muscular dystrophy The mdx mouse model as a surrogate for Duchenne muscular dystrophy Altered nuclear dynamics in MDX myofibers.Potent pro-inflammatory and pro-fibrotic molecules, osteopontin and galectin-3, are not major disease modulators of laminin α2 chain-deficient muscular dystrophy.Use of imaging biomarkers to assess perfusion and glucose metabolism in the skeletal muscle of dystrophic mice.Exploiting the full power of temporal gene expression profiling through a new statistical test: application to the analysis of muscular dystrophy data Filtering genes to improve sensitivity in oligonucleotide microarray data analysis.Array lessons from the heart: focus on the genome and transcriptome of cardiomyopathies.Literature-aided meta-analysis of microarray data: a compendium study on muscle development and disease.Transcriptome-scale similarities between mouse and human skeletal muscles with normal and myopathic phenotypes Normalization of oligonucleotide arrays based on the least-variant set of genes.Evaluation of skeletal and cardiac muscle function after chronic administration of thymosin beta-4 in the dystrophin deficient mouse.Transcriptomic analysis of dystrophin RNAi knockdown reveals a central role for dystrophin in muscle differentiation and contractile apparatus organization.TNF inhibits Notch-1 in skeletal muscle cells by Ezh2 and DNA methylation mediated repression: implications in duchenne muscular dystrophy Therapeutic targeting of signaling pathways in muscular dystrophy.A Reduction in Selenoprotein S Amplifies the Inflammatory Profile of Fast-Twitch Skeletal Muscle in the mdx Dystrophic Mouse.Osteopontin, inflammation and myogenesis: influencing regeneration, fibrosis and size of skeletal muscle The effects of MyD88 deficiency on disease phenotype in dysferlin-deficient A/J mice: role of endogenous TLR ligands.A mathematical model of skeletal muscle disease and immune response in the mdx mouse.Fibroblast growth factor-2 enhances proliferation and delays loss of chondrogenic potential in human adult bone-marrow-derived mesenchymal stem cells.Quantitative Proteomics and Immunohistochemistry Reveal Insights into Cellular and Molecular Processes in the Infarct Border Zone One Month after Myocardial Infarction.Overexpression of microRNA-206 in the skeletal muscle from myotonic dystrophy type 1 patients.Green tea extract decreases muscle pathology and NF-kappaB immunostaining in regenerating muscle fibers of mdx mice.Matrix metalloproteinase inhibitor batimastat alleviates pathology and improves skeletal muscle function in dystrophin-deficient mdx mice.TGFBR2 but not SPP1 genotype modulates osteopontin expression in Duchenne muscular dystrophy muscle Co-administration of ibuprofen and nitric oxide is an effective experimental therapy for muscular dystrophy, with immediate applicability to humans.

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P2860

A chronic inflammatory response dominates the skeletal muscle molecular signature in dystrophin-deficient mdx mice

http://www.wikidata.org/entity/Q28217143

description

2002 nî lūn-bûn

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2002 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2002 թվականի փետրվարին հրատարակված գիտական հոդված

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2002年の論文

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name

A chronic inflammatory respons ...... dystrophin-deficient mdx mice

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A chronic inflammatory respons ...... dystrophin-deficient mdx mice

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A chronic inflammatory respons ...... dystrophin-deficient mdx mice

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type

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A chronic inflammatory respons ...... dystrophin-deficient mdx mice

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A chronic inflammatory respons ...... dystrophin-deficient mdx mice

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A chronic inflammatory respons ...... dystrophin-deficient mdx mice

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A chronic inflammatory respons ...... dystrophin-deficient mdx mice

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A chronic inflammatory respons ...... dystrophin-deficient mdx mice

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A chronic inflammatory respons ...... dystrophin-deficient mdx mice

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Human Molecular Genetics

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A chronic inflammatory respons ...... dystrophin-deficient mdx mice

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Anita P Merriam

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Henry J Kaminski

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Jingjin Li

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