Prolonged fluid shear stress induces a distinct set of endothelial cell genes, most specifically lung Krüppel-like factor (KLF2) - Test2 - elhamkhani - TriplyDB

Prolonged fluid shear stress induces a distinct set of endothelial cell genes, most specifically lung Krüppel-like factor (KLF2)

Prolonged fluid shear stress induces a distinct set of endothelial cell genes, most specifically lung Krüppel-like factor (KLF2)

http://www.wikidata.org/entity/Q28217790

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Endothelial argininosuccinate synthetase 1 regulates nitric oxide production and monocyte adhesion under static and laminar shear stress conditions Krüppel-like factor 15 regulates BMPER in endothelial cells Mammalian Krüppel-like factors in health and diseases EKLF and KLF2 have compensatory roles in embryonic beta-globin gene expression and primitive erythropoiesis The aryl hydrocarbon receptor is activated by modified low-density lipoprotein Endothelial Plasticity: Shifting Phenotypes through Force Feedback Regulation of an inflammatory disease: Krüppel-like factors and atherosclerosis Novel paradigms for dialysis vascular access: upstream hemodynamics and vascular remodeling in dialysis access stenosis MicroRNAs in flow-dependent vascular remodelling Novel mechanisms of endothelial mechanotransduction Reversing blood flows act through klf2a to ensure normal valvulogenesis in the developing heart FOXC2 and fluid shear stress stabilize postnatal lymphatic vasculature.Lymph flow regulates collecting lymphatic vessel maturation in vivo.Pulling on my heartstrings: mechanotransduction in cardiac development and function The short and long of noncoding sequences in the control of vascular cell phenotypes Endothelial epigenetics in biomechanical stress: disturbed flow-mediated epigenomic plasticity in vivo and in vitro Site-specific microRNA-92a regulation of Kruppel-like factors 4 and 2 in atherosusceptible endothelium Cigarette smoke extract profoundly suppresses TNFα-mediated proinflammatory gene expression through upregulation of ATF3 in human coronary artery endothelial cells HIF-1α is required for disturbed flow-induced metabolic reprogramming in human and porcine vascular endothelium Regulation of Transcription Factors by Reactive Oxygen Species and Nitric Oxide in Vascular Physiology and Pathology.KLF4 is a key determinant in the development and progression of cerebral cavernous malformations.Biomechanical Regulation of Endothelium-dependent Events Critical for Adaptive Remodeling.Mechanotransduction in vascular physiology and atherogenesis.Optical coherence tomography captures rapid hemodynamic responses to acute hypoxia in the cardiovascular system of early embryos.4D shear stress maps of the developing heart using Doppler optical coherence tomography.Salt overload damages the glycocalyx sodium barrier of vascular endothelium.MicroRNA-mediated integration of haemodynamics and Vegf signalling during angiogenesis.Ethanol exposure alters early cardiac function in the looping heart: a mechanism for congenital heart defects?KLF2 and KLF4 control endothelial identity and vascular integrity The endothelin-1 pathway and the development of cardiovascular defects in the haemodynamically challenged chicken embryo.Gene expression analysis of embryonic stem cells expressing VE-cadherin (CD144) during endothelial differentiation MADS domain transcription factors mediate short-range DNA looping that is essential for target gene expression in Arabidopsis.Gene expression in BMPR2 mutation carriers with and without evidence of pulmonary arterial hypertension suggests pathways relevant to disease penetrance Comprehensive analysis of glomerular mRNA expression of pro- and antithrombotic genes in atypical haemolytic-uremic syndrome (aHUS).Recapitulating physiological and pathological shear stress and oxygen to model vasculature in health and disease.Simvastatin-induced sphingosine 1-phosphate receptor 1 expression is KLF2-dependent in human lung endothelial cells.Hemodynamic shear stress and the endothelium in cardiovascular pathophysiology Fluid shear stress stimulates phosphorylation-dependent nuclear export of HDAC5 and mediates expression of KLF2 and eNOS Sox18 preserves the pulmonary endothelial barrier under conditions of increased shear stress.The Gordon Wilson lecture. Understanding vascular endothelium: a pilgrim's progress. Endothelial dysfunction, biomechanical forces and the pathobiology of atherosclerosis.

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Prolonged fluid shear stress induces a distinct set of endothelial cell genes, most specifically lung Krüppel-like factor (KLF2)

http://www.wikidata.org/entity/Q28217790

description

2002 nî lūn-bûn

@nan

2002 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2002 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2002年の論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年论文

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name

Prolonged fluid shear stress i ...... ung Krüppel-like factor (KLF2)

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Prolonged fluid shear stress i ...... ung Krüppel-like factor (KLF2)

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Prolonged fluid shear stress i ...... ung Krüppel-like factor (KLF2)

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type

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Prolonged fluid shear stress i ...... ung Krüppel-like factor (KLF2)

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Prolonged fluid shear stress i ...... ung Krüppel-like factor (KLF2)

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Prolonged fluid shear stress i ...... ung Krüppel-like factor (KLF2)

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Prolonged fluid shear stress i ...... ung Krüppel-like factor (KLF2)

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Prolonged fluid shear stress i ...... ung Krüppel-like factor (KLF2)

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Prolonged fluid shear stress i ...... ung Krüppel-like factor (KLF2)

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Prolonged fluid shear stress i ...... ung Krüppel-like factor (KLF2)

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Anton J G Horrevoets

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Hans Pannekoek

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Philip G de Groot

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Rob J Dekker

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Ruud D Fontijn

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Simone van Soest

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