about
A gain-of-function mutation in TRPA1 causes familial episodic pain syndromeIon Channels and Oxidative Stress as a Potential Link for the Diagnosis or Treatment of Liver DiseasesPrimary erythromelalgia: a reviewCellular hyper-excitability caused by mutations that alter the activation process of voltage-gated sodium channelsThe ataxia3 mutation in the N-terminal cytoplasmic domain of sodium channel Na(v)1.6 disrupts intracellular traffickingLoss-of-function mutations in sodium channel Nav1.7 cause anosmia.DDESC: Dragon database for exploration of sodium channels in human.A simple technique for the prediction of interacting proteins reveals a direct Brn-3a-androgen receptor interaction.Sodium channel Na v 1.7 immunoreactivity in painful human dental pulp and burning mouth syndrome.Alternative splicing of Na(V)1.7 exon 5 increases the impact of the painful PEPD mutant channel I1461T.Mutations at opposite ends of the DIII/S4-S5 linker of sodium channel Na V 1.7 produce distinct pain disordersA novel Nav1.7 mutation producing carbamazepine-responsive erythromelalgia.Synthesis and analgesic effects of μ-TRTX-Hhn1b on models of inflammatory and neuropathic pain.Ion channel voltage sensors: structure, function, and pathophysiology.Tetrodotoxin (TTX) as a therapeutic agent for pain.A nonsense mutation in the SCN9A gene in congenital insensitivity to painVoltage-gated sodium channels at 60: structure, function and pathophysiology.Evolution: the advantage of 'maladaptive' pain plasticity.Ranolazine attenuates behavioral signs of neuropathic pain.Functional properties and differential neuromodulation of Na(v)1.6 channels.Nav1.7 is the predominant sodium channel in rodent olfactory sensory neurons.Expanding chemical diversity of conotoxins: peptoid-peptide chimeras of the sodium channel blocker μ-KIIIA and its selenopeptide analoguesCommon molecular determinants of tarantula huwentoxin-IV inhibition of Na+ channel voltage sensors in domains II and IVInherited neuronal ion channelopathies: new windows on complex neurological diseases.Inhibition of Navβ4 peptide-mediated resurgent sodium currents in Nav1.7 channels by carbamazepine, riluzole, and anandamide.Sensitization of dural afferents underlies migraine-related behavior following meningeal application of interleukin-6 (IL-6).Novel sodium channel antagonists in the treatment of neuropathic pain.Vector-mediated release of GABA attenuates pain-related behaviors and reduces Na(V)1.7 in DRG neurons.A novel substituted aminoquinoline selectively targets voltage-sensitive sodium channel isoforms and NMDA receptor subtypes and alleviates chronic inflammatory and neuropathic pain.Navβ4 regulates fast resurgent sodium currents and excitability in sensory neuronsshRNA mediated knockdown of Nav1.7 in rat dorsal root ganglion attenuates pain following burn injuryThe role of sodium channels in chronic pain.Axonal sodium-channel bands shape the response to electric stimulation in retinal ganglion cells.Tarantula huwentoxin-IV inhibits neuronal sodium channels by binding to receptor site 4 and trapping the domain ii voltage sensor in the closed configuration.Paroxysmal extreme pain disorder M1627K mutation in human Nav1.7 renders DRG neurons hyperexcitableEfficacy, safety, and tolerability of lacosamide in patients with gain-of-function Nav1.7 mutation-related small fiber neuropathy: study protocol of a randomized controlled trial-the LENSS study.EXPRESS: Voltage-dependent sodium (NaV) channels in group IV sensory afferents.Intracellular signaling in primary sensory neurons and persistent pain.Assessing decreased sensation and increased sensory phenomena in diabetic polyneuropathiesGain-of-function mutations in SCN11A cause familial episodic pain.
P2860
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P2860
description
2007 nî lūn-bûn
@nan
2007 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
name
From genes to pain: Na v 1.7 and human pain disorders
@ast
From genes to pain: Na v 1.7 and human pain disorders
@en
From genes to pain: Na v 1.7 and human pain disorders
@nl
type
label
From genes to pain: Na v 1.7 and human pain disorders
@ast
From genes to pain: Na v 1.7 and human pain disorders
@en
From genes to pain: Na v 1.7 and human pain disorders
@nl
prefLabel
From genes to pain: Na v 1.7 and human pain disorders
@ast
From genes to pain: Na v 1.7 and human pain disorders
@en
From genes to pain: Na v 1.7 and human pain disorders
@nl
P2093
P3181
P1476
From genes to pain: Na v 1.7 and human pain disorders
@en
P2093
Joel A Black
Stephen G Waxman
Sulayman D Dib-Hajj
Theodore R Cummins
P304
P3181
P356
10.1016/J.TINS.2007.08.004
P407
P577
2007-11-01T00:00:00Z