Canonical transient receptor potential channels promote cardiomyocyte hypertrophy through activation of calcineurin signaling - Test2 - elhamkhani - TriplyDB

Canonical transient receptor potential channels promote cardiomyocyte hypertrophy through activation of calcineurin signaling

Canonical transient receptor potential channels promote cardiomyocyte hypertrophy through activation of calcineurin signaling

http://www.wikidata.org/entity/Q28261648

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S165F mutation of junctophilin 2 affects Ca2+ signalling in skeletal muscle RING finger protein RNF207, a novel regulator of cardiac excitation Plasma membrane Ca2+-ATPase isoform 4 antagonizes cardiac hypertrophy in association with calcineurin inhibition in rodents TRP-ing up heart and vessels: canonical transient receptor potential channels and cardiovascular disease Modulation of TRPC5 cation channels by halothane, chloroform and propofol "TRP inflammation" relationship in cardiovascular system Proteomic-based detection of a protein cluster dysregulated during cardiovascular development identifies biomarkers of congenital heart defects Mild hypoxia-induced cardiomyocyte hypertrophy via up-regulation of HIF-1α-mediated TRPC signalling Calmodulin mediates differential sensitivity of CaMKII and calcineurin to local Ca2+ in cardiac myocytes TRPC3 and TRPC6 are essential for angiotensin II-induced cardiac hypertrophy.A pathogenic C terminus-truncated polycystin-2 mutant enhances receptor-activated Ca2+ entry via association with TRPC3 and TRPC7 TRPC3 positively regulates reactive oxygen species driving maladaptive cardiac remodeling.TrpC3 regulates hypertrophy-associated gene expression without affecting myocyte beating or cell size.Microdomain-specific localization of functional ion channels in cardiomyocytes: an emerging concept of local regulation and remodelling.Cyclic GMP/PKG-dependent inhibition of TRPC6 channel activity and expression negatively regulates cardiomyocyte NFAT activation Novel mechanism of cardiac stress modulation by PDE5 inhibition Phosphorylation of TRPC6 channels at Thr69 is required for anti-hypertrophic effects of phosphodiesterase 5 inhibition TRPC channels are necessary mediators of pathologic cardiac hypertrophy.TRPC1 channels are critical for hypertrophic signaling in the heart.Transient receptor potential channels contribute to pathological structural and functional remodeling after myocardial infarction Roles of cGMP-dependent protein kinase I (cGKI) and PDE5 in the regulation of Ang II-induced cardiac hypertrophy and fibrosis.TRPC3 contributes to regulation of cardiac contractility and arrhythmogenesis by dynamic interaction with NCX1.Molecular determinants of cardiac fibroblast electrical function and therapeutic implications for atrial fibrillation.Mechanosensitive channels in striated muscle and the cardiovascular system: not quite a stretch anymore.Inhibition of transient receptor potential channel 5 reverses 5-Fluorouracil resistance in human colorectal cancer cells.Selective and direct inhibition of TRPC3 channels underlies biological activities of a pyrazole compound.Three 4-letter words of hypertension-related cardiac hypertrophy: TRPC, mTOR, and HDAC.Expression and localization of TRPC proteins in rat ventricular myocytes at various developmental stages.Role of TRP channels in the cardiovascular system.Dysfunctional ryanodine receptor and cardiac hypertrophy: role of signaling molecules PKC-dependent coupling of calcium permeation through transient receptor potential canonical 3 (TRPC3) to calcineurin signaling in HL-1 myocytes The transient receptor potential (TRP) channel TRPC3 TRP domain and AMP-activated protein kinase binding site are required for TRPC3 activation by erythropoietin.Plasma membrane mechanical stress activates TRPC5 channels.Evidence of a Role for Fibroblast Transient Receptor Potential Canonical 3 Ca2+ Channel in Renal Fibrosis Hormonal signaling and signal pathway crosstalk in the control of myometrial calcium dynamics.Activation of transient receptor potential canonical 3 (TRPC3)-mediated Ca2+ entry by A1 adenosine receptor in cardiomyocytes disturbs atrioventricular conduction TRPC4α and TRPC4β Similarly Affect Neonatal Cardiomyocyte Survival during Chronic GPCR Stimulation.Evidence for functional expression of TRPM7 channels in human atrial myocytes.STIM1 elevation in the heart results in aberrant Ca²⁺ handling and cardiomyopathy Transient receptor potential channel TRPC5 is essential for P-glycoprotein induction in drug-resistant cancer cells.Store-operated Ca2+ entry supports contractile function in hearts of hibernators.

P2860

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P2860

Canonical transient receptor potential channels promote cardiomyocyte hypertrophy through activation of calcineurin signaling

http://www.wikidata.org/entity/Q28261648

description

2006 nî lūn-bûn

@nan

2006 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2006 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

Canonical transient receptor p ...... ation of calcineurin signaling

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Canonical transient receptor p ...... ation of calcineurin signaling

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Canonical transient receptor p ...... ation of calcineurin signaling

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Canonical transient receptor p ...... ation of calcineurin signaling

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Canonical transient receptor p ...... ation of calcineurin signaling

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Canonical transient receptor p ...... ation of calcineurin signaling

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Canonical transient receptor p ...... ation of calcineurin signaling

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Canonical transient receptor p ...... ation of calcineurin signaling

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Canonical transient receptor p ...... ation of calcineurin signaling

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Journal of Biological Chemistry

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Canonical transient receptor p ...... ation of calcineurin signaling

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David B Hood

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Eric N Olson

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Erik W Bush

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Joseph A Chapo

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Michael R Bristow

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Philip J Papst

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Timothy A McKinsey

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Wayne Minobe

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33487-96

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scholarly article

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2588808

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10.1074/JBC.M605536200

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44

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281

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2006-11-03T00:00:00Z

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16950785

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