Nuclear factor of activated T cells c1 induces osteoclast-associated receptor gene expression during tumor necrosis factor-related activation-induced cytokine-mediated osteoclastogenesis - Test2 - elhamkhani - TriplyDB

Nuclear factor of activated T cells c1 induces osteoclast-associated receptor gene expression during tumor necrosis factor-related activation-induced cytokine-mediated osteoclastogenesis

Nuclear factor of activated T cells c1 induces osteoclast-associated receptor gene expression during tumor necrosis factor-related activation-induced cytokine-mediated osteoclastogenesis

http://www.wikidata.org/entity/Q28267970

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Identification of LRRc17 as a negative regulator of receptor activator of NF-kappaB ligand (RANKL)-induced osteoclast differentiation MicroRNAs in Osteoclastogenesis and Function: Potential Therapeutic Targets for Osteoporosis Signaling Pathways in Osteoclast Differentiation Osteoimmunology: Major and Costimulatory Pathway Expression Associated with Chronic Inflammatory Induced Bone Loss Regulation of NFATc1 in Osteoclast Differentiation Pyrroloquinoline quinine inhibits RANKL-mediated expression of NFATc1 in part via suppression of c-Fos in mouse bone marrow cells and inhibits wear particle-induced osteolysis in mice Molecules and signaling pathways involved in the expression of OC-STAMP during osteoclastogenesis Role of CrkII Signaling in RANKL-Induced Osteoclast Differentiation and Function.NFATc1 in mice represses osteoprotegerin during osteoclastogenesis and dissociates systemic osteopenia from inflammation in cherubism.Deletion of CD74, a putative MIF receptor, in mice enhances osteoclastogenesis and decreases bone mass Id1 represses osteoclast-dependent transcription and affects bone formation and hematopoiesis.Promising bone-related therapeutic targets for rheumatoid arthritis.The role of calcium release activated calcium channels in osteoclast differentiation.Negative feedback control of osteoclast formation through ubiquitin-mediated down-regulation of NFATc1.TGF-β inducible early gene 1 regulates osteoclast differentiation and survival by mediating the NFATc1, AKT, and MEK/ERK signaling pathways Transcriptional regulation of bone and joint remodeling by NFAT Lhx2 regulates bone remodeling in mice by modulating RANKL signaling in osteoclasts.Mitf regulates osteoclastogenesis by modulating NFATc1 activity.2-Methoxystypandrone represses RANKL-mediated osteoclastogenesis by down-regulating formation of TRAF6-TAK1 signalling complexes Angelica sinensis extract inhibits RANKL-mediated osteoclastogenesis by down-regulated the expression of NFATc1 in mouse bone marrow cells (+)-Vitisin A inhibits osteoclast differentiation by preventing TRAF6 ubiquitination and TRAF6-TAK1 formation to suppress NFATc1 activation Activation of liver X receptor (LXR) inhibits receptor activator of nuclear factor κB ligand (RANKL)-induced osteoclast differentiation in an LXRβ-dependent mechanism Id helix-loop-helix proteins negatively regulate TRANCE-mediated osteoclast differentiation Orai1-mediated calcium entry plays a critical role in osteoclast differentiation and function by regulating activation of the transcription factor NFATc1 Gene disruption of the calcium channel Orai1 results in inhibition of osteoclast and osteoblast differentiation and impairs skeletal development.Signaling networks that control the lineage commitment and differentiation of bone cells.NRROS Negatively Regulates Osteoclast Differentiation by Inhibiting RANKL-Mediated NF-N:B and Reactive Oxygen Species Pathways.Plasma membrane calcium ATPase regulates bone mass by fine-tuning osteoclast differentiation and survival.Osteoclast fusion and regulation by RANKL-dependent and independent factors.The Influence of DNA Methylation on Bone Cells.Early estrogen-induced gene 1, a novel RANK signaling component, is essential for osteoclastogenesis Regulators of G protein signaling 12 promotes osteoclastogenesis in bone remodeling and pathological bone loss.The immunoreceptor tyrosine-based activation motif (ITAM) -related factors are increased in synovial tissue and vasculature of rheumatoid arthritic joints.Sirt6 cooperates with Blimp1 to positively regulate osteoclast differentiation.STAT5 is a key transcription factor for IL-3-mediated inhibition of RANKL-induced osteoclastogenesis NFATc1 induces osteoclast fusion via up-regulation of Atp6v0d2 and the dendritic cell-specific transmembrane protein (DC-STAMP).GATA4 negatively regulates bone sialoprotein expression in osteoblasts.NADPH oxidase gp91(phox) contributes to RANKL-induced osteoclast differentiation by upregulating NFATc1 The transmembrane adaptor protein, linker for activation of T cells (LAT), regulates RANKL-induced osteoclast differentiation.Selective induction of P2Y14 receptor by RANKL promotes osteoclast formation.

P2860

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P2860

Nuclear factor of activated T cells c1 induces osteoclast-associated receptor gene expression during tumor necrosis factor-related activation-induced cytokine-mediated osteoclastogenesis

http://www.wikidata.org/entity/Q28267970

description

2005 nî lūn-bûn

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2005 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2005 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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name

Nuclear factor of activated T ...... ne-mediated osteoclastogenesis

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Nuclear factor of activated T ...... ne-mediated osteoclastogenesis

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Nuclear factor of activated T ...... ne-mediated osteoclastogenesis

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Nuclear factor of activated T ...... ne-mediated osteoclastogenesis

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Nuclear factor of activated T ...... ne-mediated osteoclastogenesis

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Nuclear factor of activated T ...... ne-mediated osteoclastogenesis

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Nuclear factor of activated T ...... ne-mediated osteoclastogenesis

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Nuclear factor of activated T ...... ne-mediated osteoclastogenesis

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Nuclear factor of activated T ...... ne-mediated osteoclastogenesis

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David E Fisher

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Hye-Mi Jin

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Hyun Kook

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Jung Ha Kim

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Junwon Lee

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Kabsun Kim

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Kyung Keun Kim

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Nacksung Kim

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Seoung-Hoon Lee

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Yongwon Choi

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