Excitotoxic neuronal death and the pathogenesis of Huntington's disease
about
Domoic acid toxicologic pathology: a reviewCorticostriatal Dysfunction in Huntington's Disease: The BasicsLevodopa-Induced Dyskinesia Is Related to Indirect Pathway Medium Spiny Neuron Excitotoxicity: A Hypothesis Based on an Unexpected FindingDeveloping stem cell therapies for juvenile and adult-onset Huntington's diseaseRole of cerebral cortex in the neuropathology of Huntington's diseaseN-Acetylcysteine improves mitochondrial function and ameliorates behavioral deficits in the R6/1 mouse model of Huntington's diseaseIntrastriatal transplantation of adenovirus-generated induced pluripotent stem cells for treating neuropathological and functional deficits in a rodent model of Huntington's disease.High-resolution respirometry of fine-needle muscle biopsies in pre-manifest Huntington's disease expansion mutation carriers shows normal mitochondrial respiratory function.Identification of translational activators of glial glutamate transporter EAAT2 through cell-based high-throughput screening: an approach to prevent excitotoxicity.Differential vulnerability of neurons in Huntington's disease: the role of cell type-specific features.Developmental regulation and neuroprotective effects of striatal tonic GABAA currentsEarly atrophy of pallidum and accumbens nucleus in Huntington's disease.Treatment with a herbal formula B401 enhances neuroprotection and angiogenesis in the R6/2 mouse model of Huntington's disease.Nuclear translocation of AMPK-alpha1 potentiates striatal neurodegeneration in Huntington's disease.Treadmill exercise improves short-term memory by enhancing hippocampal cell proliferation in quinolinic acid-induced Huntington's disease rats.Exploratory 7-Tesla magnetic resonance spectroscopy in Huntington's disease provides in vivo evidence for impaired energy metabolism.HD iPSC-derived neural progenitors accumulate in culture and are susceptible to BDNF withdrawal due to glutamate toxicity.Reductions in behavioral deficits and neuropathology in the R6/2 mouse model of Huntington's disease following transplantation of bone-marrow-derived mesenchymal stem cells is dependent on passage number.Association study indicates a protective role of phosphatidylinositol-4-phosphate-5-kinase against tardive dyskinesia.Treadmill exercise enhances spatial learning ability through suppressing hippocampal apoptosis in Huntington's disease ratsThe nervous system and metabolic dysregulation: emerging evidence converges on ketogenic diet therapy.Up-regulation of GLT1 reverses the deficit in cortically evoked striatal ascorbate efflux in the R6/2 mouse model of Huntington's disease.Neuronal cell death in neurodegenerative diseases: recurring themes around protein handling.Using Actiwatch to monitor circadian rhythm disturbance in Huntington' disease: A cautionary note.Brain metabolite alterations and cognitive dysfunction in early Huntington's disease.The Isotropic Fractionator as a Tool for Quantitative Analysis in Central Nervous System DiseasesOxidative stress is related to the deleterious effects of heme oxygenase-1 in an in vivo neuroinflammatory rat model.Characterization of forebrain neurons derived from late-onset Huntington's disease human embryonic stem cell lines.Neuroprotection against neonatal hypoxia/ischemia-induced cerebral cell death by prevention of calpain-mediated mGluR1alpha truncation.TRiC subunits enhance BDNF axonal transport and rescue striatal atrophy in Huntington's diseaseMembrane compartments and purinergic signalling: P2X receptors in neurodegenerative and neuroinflammatory events.Transplantation of umbilical cord-derived mesenchymal stem cells into the striata of R6/2 mice: behavioral and neuropathological analysis.Amyotrophic lateral sclerosis (ALS): three letters that change the people's life. For ever.Induced Pluripotent Stem Cells in Huntington's Disease: Disease Modeling and the Potential for Cell-Based Therapy.Basic mechanisms of neurodegeneration: a critical update.Hypoxia inducible factor-1 as a target for neurodegenerative diseases.Corticostriatal dysfunction and glutamate transporter 1 (GLT1) in Huntington's disease: interactions between neurons and astrocytes.New insights into the mechanism of drug-induced dyskinesia.Corticostriatal circuit dysfunction in Huntington's disease: intersection of glutamate, dopamine and calcium.Astrocytes are key but indirect contributors to the development of the symptomatology and pathophysiology of Huntington's disease.
P2860
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P2860
Excitotoxic neuronal death and the pathogenesis of Huntington's disease
description
2008 nî lūn-bûn
@nan
2008 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Excitotoxic neuronal death and the pathogenesis of Huntington's disease
@ast
Excitotoxic neuronal death and the pathogenesis of Huntington's disease
@en
Excitotoxic neuronal death and the pathogenesis of Huntington's disease
@nl
type
label
Excitotoxic neuronal death and the pathogenesis of Huntington's disease
@ast
Excitotoxic neuronal death and the pathogenesis of Huntington's disease
@en
Excitotoxic neuronal death and the pathogenesis of Huntington's disease
@nl
prefLabel
Excitotoxic neuronal death and the pathogenesis of Huntington's disease
@ast
Excitotoxic neuronal death and the pathogenesis of Huntington's disease
@en
Excitotoxic neuronal death and the pathogenesis of Huntington's disease
@nl
P2093
P3181
P1476
Excitotoxic neuronal death and the pathogenesis of Huntington's disease
@en
P2093
Ana María Estrada Sánchez
Jana Mejía-Toiber
Lourdes Massieu
P304
P3181
P356
10.1016/J.ARCMED.2007.11.011
P407
P577
2008-04-01T00:00:00Z