Mutations in the glutamate transporter EAAT2 gene do not cause abnormal EAAT2 transcripts in amyotrophic lateral sclerosis
about
Association study of polymorphisms in the excitatory amino acid transporter 2 gene (SLC1A2) with schizophreniaAmyotrophic lateral sclerosis-linked glutamate transporter mutant has impaired glutamate clearance capacityMechanisms of neurodegeneration in amyotrophic lateral sclerosisAlternative splicing of exon 10 in the tau gene as a target for treatment of tauopathies.Neurotoxic mechanisms of degeneration in motor neuron diseases.Glutamate transporters and the excitotoxic path to motor neuron degeneration in amyotrophic lateral sclerosis.Familial amyotrophic lateral sclerosis.A phase II trial of talampanel in subjects with amyotrophic lateral sclerosis.The role of glutamate transporters in neurodegenerative diseases and potential opportunities for intervention.Excitotoxicity and amyotrophic lateral sclerosis.Clinical features and a mutation with late onset of limb girdle muscular dystrophy 2B.Metabolism, cell surface organization, and disease.An autopsy case of frontotemporal lobar degeneration with the appearance of fused in sarcoma inclusions (basophilic inclusion body disease) clinically presenting corticobasal syndrome.Genotype-phenotype relationships in familial amyotrophic lateral sclerosis with FUS/TLS mutations in Japan.Wnt signaling induces GLT-1 expression in rat C6 glioma cells.Posttranslational nitration of tyrosine residues modulates glutamate transmission and contributes to N-methyl-D-aspartate-mediated thermal hyperalgesia.Increases in cortical glutamate concentrations in transgenic amyotrophic lateral sclerosis mice are attenuated by creatine supplementation.No occurrence of the glutamate transporter EAAT2 A206G polymorphism in schizophrenic subjects.Tardive decrease of astrocytic glutamate transporter protein in transgenic mice with ALS-linked mutant SOD1.Expression and distribution of 'high affinity' glutamate transporters GLT1, GLAST, EAAC1 and of GCPII in the rat peripheral nervous system.FUS/TLS-immunoreactive neuronal and glial cell inclusions increase with disease duration in familial amyotrophic lateral sclerosis with an R521C FUS/TLS mutation.EAAT2 and the Molecular Signature of Amyotrophic Lateral Sclerosis.
P2860
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P2860
Mutations in the glutamate transporter EAAT2 gene do not cause abnormal EAAT2 transcripts in amyotrophic lateral sclerosis
description
1998 nî lūn-bûn
@nan
1998 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
Mutations in the glutamate tra ...... amyotrophic lateral sclerosis
@ast
Mutations in the glutamate tra ...... amyotrophic lateral sclerosis
@en
Mutations in the glutamate tra ...... amyotrophic lateral sclerosis
@nl
type
label
Mutations in the glutamate tra ...... amyotrophic lateral sclerosis
@ast
Mutations in the glutamate tra ...... amyotrophic lateral sclerosis
@en
Mutations in the glutamate tra ...... amyotrophic lateral sclerosis
@nl
prefLabel
Mutations in the glutamate tra ...... amyotrophic lateral sclerosis
@ast
Mutations in the glutamate tra ...... amyotrophic lateral sclerosis
@en
Mutations in the glutamate tra ...... amyotrophic lateral sclerosis
@nl
P2093
P356
P1433
P1476
Mutations in the glutamate tra ...... amyotrophic lateral sclerosis
@en
P2093
B A Geller
B A Hosler
J D Rothstein
T L Munsat
P304
P356
10.1002/ANA.410430514
P407
P577
1998-05-01T00:00:00Z