DNA copy number alterations and expression of relevant genes in triple-negative breast cancer - Test2 - elhamkhani - TriplyDB

DNA copy number alterations and expression of relevant genes in triple-negative breast cancer

DNA copy number alterations and expression of relevant genes in triple-negative breast cancer

http://www.wikidata.org/entity/Q28271168

about

WW domain-mediated interaction with Wbp2 is important for the oncogenic property of TAZ Genomic amplification and oncogenic properties of the GASC1 histone demethylase gene in breast cancer Selective genomic copy number imbalances and probability of recurrence in early-stage breast cancer An integrative multi-dimensional genetic and epigenetic strategy to identify aberrant genes and pathways in cancer.Integrative molecular profiling of triple negative breast cancers identifies amplicon drivers and potential therapeutic targets.Genomic profiling of isolated circulating tumor cells from metastatic breast cancer patients.Novel therapeutic strategies in the treatment of triple-negative breast cancer.RNA interference (RNAi) screening approach identifies agents that enhance paclitaxel activity in breast cancer cells.Identification and functional analysis of 9p24 amplified genes in human breast cancer.Molecular characterisation of cell line models for triple-negative breast cancers.Characteristics of triple-negative breast cancer Dual-color fluorescence in situ hybridization reveals an association of chromosome 8q22 but not 8p21 imbalance with high grade invasive breast carcinoma.Concurrent gene signatures for han chinese breast cancers Nuclear factor I/B is an oncogene in small cell lung cancer Genome-wide copy number alterations in subtypes of invasive breast cancers in young white and African American women.Genetic characterization of breast cancer and implications for clinical management 17p12 deletion in breast cancer predicts resistance to neoadjuvant chemotherapy.Structural and functional insights into the TEAD-YAP complex in the Hippo signaling pathway.bc-GenExMiner 3.0: new mining module computes breast cancer gene expression correlation analyses Emerging roles of TEAD transcription factors and its coactivators in cancers RNF138 interacts with RAD51D and is required for DNA interstrand crosslink repair and maintaining chromosome integrity Transcription Factor NFIB Is a Driver of Small Cell Lung Cancer Progression in Mice and Marks Metastatic Disease in Patients.Nuclear factor one B (NFIB) encodes a subtype-specific tumour suppressor in glioblastoma.DNA amplifications in breast cancer: genotypic-phenotypic correlations.Beyond triple-negative breast cancer: the need to define new subtypes.The Hippo pathway in biological control and cancer development.TGF-beta-dependent and -independent roles of STRAP in cancer.The YAP and TAZ transcription co-activators: key downstream effectors of the mammalian Hippo pathway.Understanding the biology of triple-negative breast cancer.NFIB is a potential target for estrogen receptor-negative breast cancers.The oncogenic role of GASC1 in chemically induced mouse skin cancer.Nuclear Factor I/B: A Master Regulator of Cell Differentiation with Paradoxical Roles in Cancer.Exploring phenotype patterns of breast cancer within somatic mutations: a modicum in the intrinsic code.TEADs mediate nuclear retention of TAZ to promote oncogenic transformation.Post-Transcriptional Regulation of the GASC1 Oncogene with Active Tumor-Targeted siRNA-Nanoparticles.The prognostic impacts of TEA domain (TEAD) transcription factor polymorphisms in Chinese hepatocellular carcinoma patients.Molecular evidence in support of the neoplastic and precursor nature of microglandular adenosis.Tumours of the lacrimal gland. Epidemiological, clinical and genetic characteristics.Hypoxia-induced protein CAIX is associated with somatic loss of BRCA1 protein and pathway activity in triple negative breast cancer.Regulation of the Hippo Pathway Transcription Factor TEAD.

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P2860

DNA copy number alterations and expression of relevant genes in triple-negative breast cancer

http://www.wikidata.org/entity/Q28271168

description

2008 nî lūn-bûn

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2008 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի հունիսին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

DNA copy number alterations an ...... triple-negative breast cancer

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DNA copy number alterations an ...... triple-negative breast cancer

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DNA copy number alterations an ...... triple-negative breast cancer

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type

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DNA copy number alterations an ...... triple-negative breast cancer

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DNA copy number alterations an ...... triple-negative breast cancer

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DNA copy number alterations an ...... triple-negative breast cancer

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DNA copy number alterations an ...... triple-negative breast cancer

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DNA copy number alterations an ...... triple-negative breast cancer

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DNA copy number alterations an ...... triple-negative breast cancer

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Genes, Chromosomes and Cancer

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DNA copy number alterations an ...... triple-negative breast cancer

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Eun-Mi Jung

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In-Ae Park

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Jason Jongho Kang

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Ji-Yeon Bae

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Jihyoung Cho

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Jong Won Lee

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Ki-Tae Hwang

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Monica Nicolau

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Song-Ju Yang

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Wonshik Han

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P2860

Human SPF45, a splicing factor, has limited expression in normal tissues, is overexpressed in many tumors, and can confer a multidrug-resistant phenotype to cells

Expression of cytokeratins 17 and 5 identifies a group of breast carcinomas with poor clinical outcome

A novel amplicon at 9p23 - 24 in squamous cell carcinoma of the esophagus that lies proximal to GASC1 and harbors NFIB.

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scholarly article

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10.1002/GCC.20550

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6

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47

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Stefanie S Jeffrey

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18314908

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