Minireview. Kainic acid as a tool for the study of temporal lobe epilepsy
about
N-copine: a novel two C2-domain-containing protein with neuronal activity-regulated expressionInterictal spike frequency varies with ovarian cycle stage in a rat model of epilepsyQ/R editing of the rat GluR5 and GluR6 kainate receptors in vivo and in vitro: evidence for independent developmental, pathological and cellular regulationElectrophysiological evidence of monosynaptic excitatory transmission between granule cells after seizure-induced mossy fiber sproutingSurvival of dentate hilar mossy cells after pilocarpine-induced seizures and their synchronized burst discharges with area CA3 pyramidal cellsInteractions between inflammation, sex steroids, and Alzheimer's disease risk factorsAnticonvulsant activity of B2, an adenosine analog, on chemical convulsant-induced seizuresKainate receptors and rhythmic activity in neuronal networks: hippocampal gamma oscillations as a toolDomoic acid epileptic diseaseClassic hippocampal sclerosis and hippocampal-onset epilepsy produced by a single “cryptic” episode of focal hippocampal excitation in awake ratsThe GluK4 kainate receptor subunit regulates memory, mood, and excitotoxic neurodegeneration.Genetic determinants of susceptibility to excitotoxic cell death: implications for gene targeting approaches.Epileptogenic effects of status epilepticus.Lack of kainic acid-induced gamma oscillations predicts subsequent CA1 excitotoxic cell death.Experimental models of chronic focal epilepsy: a critical review of four models.The anti-epileptic drug levetiracetam reverses the inhibition by negative allosteric modulators of neuronal GABA- and glycine-gated currentsAutomated identification of multiple seizure-related and interictal epileptiform event types in the EEG of micePlasticity, synaptic strength, and epilepsy: what can we learn from ultrastructural data?Changes in nitric oxide synthesis and epileptic activity in the contralateral hippocampus of rats following intrahippocampal kainate injection.Neuroprotection by glutamate receptor antagonists against seizure-induced excitotoxic cell death in the aging brain.Ligands for ionotropic glutamate receptorsMossy fiber plasticity and enhanced hippocampal excitability, without hippocampal cell loss or altered neurogenesis, in an animal model of prolonged febrile seizuresPretreatment of Guinea pigs with galantamine prevents immediate and delayed effects of soman on inhibitory synaptic transmission in the hippocampus.A-to-I RNA editing and human disease.Hsp70 and its molecular role in nervous system diseasesProgressive myoclonic epilepsy-associated gene KCTD7 is a regulator of potassium conductance in neurons.Seizure-induced reduction in PIP3 levels contributes to seizure-activity and is rescued by valproic acid.Spontaneous and evoked high-frequency oscillations in the tetanus toxin model of epilepsy.Is neuronal death required for seizure-induced epileptogenesis in the immature brain?Two populations of kainate receptors with separate signaling mechanisms in hippocampal interneuronsStatus epilepticus decreases glutamate receptor 2 mRNA and protein expression in hippocampal pyramidal cells before neuronal deathNeurological disease rises from ocean to bring model for human epilepsy to life.Age-dependent changes in intrinsic neuronal excitability in subiculum after status epilepticus.Inflammatory mechanisms associated with brain damage induced by kainic acid with special reference to the interleukin-1 system.The influence of epileptic neuropathology and prior peripheral immunity on CNS transduction by rAAV2 and rAAV5.A novel seizure-induced synaptotagmin gene identified by differential display.Defining "epileptogenesis" and identifying "antiepileptogenic targets" in animal models of acquired temporal lobe epilepsy is not as simple as it might seem.Sequel of spontaneous seizures after kainic acid-induced status epilepticus and associated neuropathological changes in the subiculum and entorhinal cortexSelective death of hippocampal CA3 pyramidal cells with mossy fiber afferents after CRH-induced status epilepticus in infant ratsA postnatal peak in microglial development in the mouse hippocampus is correlated with heightened sensitivity to seizure triggers.
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Minireview. Kainic acid as a tool for the study of temporal lobe epilepsy
description
1981 nî lūn-bûn
@nan
1981 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
1981 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
1981年の論文
@ja
1981年論文
@yue
1981年論文
@zh-hant
1981年論文
@zh-hk
1981年論文
@zh-mo
1981年論文
@zh-tw
1981年论文
@wuu
name
Minireview. Kainic acid as a tool for the study of temporal lobe epilepsy
@ast
Minireview. Kainic acid as a tool for the study of temporal lobe epilepsy
@en
Minireview. Kainic acid as a tool for the study of temporal lobe epilepsy
@nl
type
label
Minireview. Kainic acid as a tool for the study of temporal lobe epilepsy
@ast
Minireview. Kainic acid as a tool for the study of temporal lobe epilepsy
@en
Minireview. Kainic acid as a tool for the study of temporal lobe epilepsy
@nl
prefLabel
Minireview. Kainic acid as a tool for the study of temporal lobe epilepsy
@ast
Minireview. Kainic acid as a tool for the study of temporal lobe epilepsy
@en
Minireview. Kainic acid as a tool for the study of temporal lobe epilepsy
@nl
P3181
P1433
P1476
Minireview. Kainic acid as a tool for the study of temporal lobe epilepsy
@en
P2093
J V Nadler
P304
P3181
P356
10.1016/0024-3205(81)90659-7
P407
P577
1981-11-16T00:00:00Z