about
Virulence factors in Escherichia coli urinary tract infectionAlpha-hemolysin from Escherichia coli uses endogenous amplification through P2X receptor activation to induce hemolysis.RegA, an AraC-like protein, is a global transcriptional regulator that controls virulence gene expression in Citrobacter rodentium.Bacteria differentially induce degradation of Bcl-xL, a survival protein, by human plateletsEscherichia coli alpha-hemolysin triggers shrinkage of erythrocytes via K(Ca)3.1 and TMEM16A channels with subsequent phosphatidylserine exposure.Bacterial RTX toxins allow acute ATP release from human erythrocytes directly through the toxin pore.Vibrio cholerae tolC is required for bile resistance and colonization.Python erythrocytes are resistant to α-hemolysin from Escherichia coli.Inactivation of host Akt/protein kinase B signaling by bacterial pore-forming toxins.Multidrug-resistance efflux pumps - not just for resistance.Shewanella and Photobacterium spp. in oysters and seawater from the Delaware Bay.P2X receptor-dependent erythrocyte damage by α-hemolysin from Escherichia coli triggers phagocytosis by THP-1 cells.Origins and virulence mechanisms of uropathogenic Escherichia coliInhibition of P2X Receptors Protects Human Monocytes against Damage by Leukotoxin from Aggregatibacter actinomycetemcomitans and α-Hemolysin from Escherichia coliLoop Diuretics Diminish Hemolysis Induced by α-Hemolysin from Escherichia coli.P2X1, P2X4, and P2X7 Receptor Knock Out Mice Expose Differential Outcome of Sepsis Induced by α-Haemolysin Producing Escherichia coli[Ca2+]i Oscillations and IL-6 Release Induced by α-Hemolysin from Escherichia coli Require P2 Receptor Activation in Renal Epithelia.Putative identification of an amphipathic alpha-helical sequence in hemolysin of Escherichia coli (HlyA) involved in transmembrane pore formation.Zinc treatment is efficient against Escherichia coli α-haemolysin-induced intestinal leakage in mice.Effects of Escherichia coli hemolysin on endothelial cell function.Genome-Wide Analysis of Oceanimonas sp. GK1 Isolated from Gavkhouni Wetland (Iran) Demonstrates Presence of Genes for Virulence and Pathogenicity.Evolved Aztreonam Resistance Is Multifactorial and Can Produce Hypervirulence in Pseudomonas aeruginosa.
P2860
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P2860
description
1988 nî lūn-bûn
@nan
1988 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
1988 թվականի հունիսին հրատարակված գիտական հոդված
@hy
1988年の論文
@ja
1988年論文
@yue
1988年論文
@zh-hant
1988年論文
@zh-hk
1988年論文
@zh-mo
1988年論文
@zh-tw
1988年论文
@wuu
name
The hemolysin of Escherichia coli
@ast
The hemolysin of Escherichia coli
@en
The hemolysin of Escherichia coli
@nl
type
label
The hemolysin of Escherichia coli
@ast
The hemolysin of Escherichia coli
@en
The hemolysin of Escherichia coli
@nl
prefLabel
The hemolysin of Escherichia coli
@ast
The hemolysin of Escherichia coli
@en
The hemolysin of Escherichia coli
@nl
P2093
P2860
P356
P1476
The hemolysin of Escherichia coli
@en
P2093
P2860
P2888
P304
P356
10.1007/BF00144740
P407
P577
1988-06-01T00:00:00Z
P6179
1005106271