MicroRNA miR-133 represses HERG K+ channel expression contributing to QT prolongation in diabetic hearts - Test2 - elhamkhani - TriplyDB

MicroRNA miR-133 represses HERG K+ channel expression contributing to QT prolongation in diabetic hearts

MicroRNA miR-133 represses HERG K+ channel expression contributing to QT prolongation in diabetic hearts

http://www.wikidata.org/entity/Q28291882

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High glucose suppresses human islet insulin biosynthesis by inducing miR-133a leading to decreased polypyrimidine tract binding protein-expression MicroRNAs in cardiomyocyte development MicroRNAs 1, 133, and 206: critical factors of skeletal and cardiac muscle development, function, and disease miR-605 joins p53 network to form a p53:miR-605:Mdm2 positive feedback loop in response to stress Subclinical Detection of Diabetic Cardiomyopathy with MicroRNAs: Challenges and Perspectives MicroRNAs as pharmacological targets in diabetes Human genetics of diabetic vascular complications Dedifferentiation and proliferation of mammalian cardiomyocytes MicroRNA-1 downregulation by propranolol in a rat model of myocardial infarction: a new mechanism for ischaemic cardioprotection NFATc4 is negatively regulated in miR-133a-mediated cardiomyocyte hypertrophic repression Down-regulation of miR-1/miR-133 contributes to re-expression of pacemaker channel genes HCN2 and HCN4 in hypertrophic heart Differential expression of dicer, miRNAs, and inflammatory markers in diabetic Ins2+/- Akita hearts The cardiac transcription network modulated by Gata4, Mef2a, Nkx2.5, Srf, histone modifications, and microRNAs miR-24 regulates menin in the endocrine pancreas MicroRNA-223 regulates Glut4 expression and cardiomyocyte glucose metabolism.Influence of diabetes mellitus on outcome in patients over 40 years of age with the long QT syndrome Right into the heart of microRNA-133a.MicroRNA-133a protects against myocardial fibrosis and modulates electrical repolarization without affecting hypertrophy in pressure-overloaded adult hearts MicroRNA-133a regulates DNA methylation in diabetic cardiomyocytes.MicroRNAs and metabolic disorders - where are we heading?Elevated expression levels of miR-143/5 in saphenous vein smooth muscle cells from patients with Type 2 diabetes drive persistent changes in phenotype and function.MicroRNA regulatory networks in cardiovascular development Small engine, big power: microRNAs as regulators of cardiac diseases and regeneration.Strategies to deliver microRNAs as potential therapeutics in the treatment of cardiovascular pathology.MicroRNAs: Meta-controllers of gene expression in synaptic activity emerge as genetic and diagnostic markers of human disease Toward microRNA-based therapeutics for heart disease: the sense in antisense.A single anti-microRNA antisense oligodeoxyribonucleotide (AMO) targeting multiple microRNAs offers an improved approach for microRNA interference MicroRNAs and cardiovascular diseases.An intragenic SRF-dependent regulatory motif directs cardiac-specific microRNA-1-1/133a-2 expression.Diabetes complications: the microRNA perspective.MicroRNAs in β-cell biology, insulin resistance, diabetes and its complications Identification of New SRF Binding Sites in Genes Modulated by SRF Over-Expression in Mouse Hearts NF-kappaB P50/P65 hetero-dimer mediates differential regulation of CD166/ALCAM expression via interaction with micoRNA-9 after serum deprivation, providing evidence for a novel negative auto-regulatory loop.Cardiac ventricular repolarization reserve: a principle for understanding drug-related proarrhythmic risk.Expression of circulating microRNA-1 and microRNA-133 in pediatric patients with tachycardia miR-146a-Mediated extracellular matrix protein production in chronic diabetes complications.ASK1 modulates the expression of microRNA Let7A in microglia under high glucose in vitro condition.Serum miR-210 Contributes to Tumor Detection, Stage Prediction and Dynamic Surveillance in Patients with Bladder Cancer.MiR-21 promotes intrahepatic cholangiocarcinoma proliferation and growth in vitro and in vivo by targeting PTPN14 and PTEN.MicroRNAs: powerful new regulators of heart disease and provocative therapeutic targets.

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P2860

MicroRNA miR-133 represses HERG K+ channel expression contributing to QT prolongation in diabetic hearts

http://www.wikidata.org/entity/Q28291882

description

2007 nî lūn-bûn

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2007 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2007 թվականի ապրիլին հրատարակված գիտական հոդված

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2007年の論文

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年學術文章

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name

MicroRNA miR-133 represses HER ...... rolongation in diabetic hearts

@ast

MicroRNA miR-133 represses HER ...... rolongation in diabetic hearts

@en

MicroRNA miR-133 represses HER ...... rolongation in diabetic hearts

@nl

type

label

MicroRNA miR-133 represses HER ...... rolongation in diabetic hearts

@ast

MicroRNA miR-133 represses HER ...... rolongation in diabetic hearts

@en

MicroRNA miR-133 represses HER ...... rolongation in diabetic hearts

@nl

prefLabel

MicroRNA miR-133 represses HER ...... rolongation in diabetic hearts

@ast

MicroRNA miR-133 represses HER ...... rolongation in diabetic hearts

@en

MicroRNA miR-133 represses HER ...... rolongation in diabetic hearts

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Journal of Biological Chemistry

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MicroRNA miR-133 represses HER ...... rolongation in diabetic hearts

@en

P2093

Baofeng Yang

http://www.w3.org/2001/XMLSchema#string

Huixian Lin

http://www.w3.org/2001/XMLSchema#string

Jiening Xiao

http://www.w3.org/2001/XMLSchema#string

Ning Wang

http://www.w3.org/2001/XMLSchema#string

Xiaobin Luo

http://www.w3.org/2001/XMLSchema#string

Yanjie Lu

http://www.w3.org/2001/XMLSchema#string

Ying Zhang

http://www.w3.org/2001/XMLSchema#string

Yiqiang Zhang

http://www.w3.org/2001/XMLSchema#string

Zhiguo Wang

http://www.w3.org/2001/XMLSchema#string

P2860

Principles of microRNA-target recognition

The role of microRNA-1 and microRNA-133 in skeletal muscle proliferation and differentiation

Antisense inhibition of human miRNAs and indications for an involvement of miRNA in cell growth and apoptosis

The functions of animal microRNAs

Silencing of microRNAs in vivo with 'antagomirs'

Serum response factor regulates a muscle-specific microRNA that targets Hand2 during cardiogenesis

A case-control analysis of intra-arterial urokinase thrombolysis in acute cardioembolic stroke.

Prolonged QTc interval predicts mortality in patients with Type 1 diabetes mellitus.

Cardiac IKr channels minimally comprise hERG 1a and 1b subunits.

QT interval abnormalities are often present at diagnosis in diabetes and are better predictors of cardiac death than ankle brachial pressure index and autonomic function tests

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3256585

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10.1074/JBC.C700015200

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17

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282

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2007-04-27T00:00:00Z

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17344217

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