Suppression of Wnt signaling by the green tea compound (-)-epigallocatechin 3-gallate (EGCG) in invasive breast cancer cells. Requirement of the transcriptional repressor HBP1 - Test2 - elhamkhani - TriplyDB

Suppression of Wnt signaling by the green tea compound (-)-epigallocatechin 3-gallate (EGCG) in invasive breast cancer cells. Requirement of the transcriptional repressor HBP1

Suppression of Wnt signaling by the green tea compound (-)-epigallocatechin 3-gallate (EGCG) in invasive breast cancer cells. Requirement of the transcriptional repressor HBP1

http://www.wikidata.org/entity/Q28298718

about

The role of nutraceuticals in the regulation of Wnt and Hedgehog signaling in cancer Suppressive Effects of Tea Catechins on Breast Cancer Polyphenol Compound as a Transcription Factor Inhibitor Phytochemicals as Innovative Therapeutic Tools against Cancer Stem Cells Flavonoids and Wnt/β-catenin signaling: potential role in colorectal cancer therapies Emerging role of the β-catenin-PPARγ axis in the pathogenesis of colorectal cancer Targeting CSC-related miRNAs for cancer therapy by natural agents Chemoprevention in gastrointestinal physiology and disease. Natural products and microbiome The Anti-Cancer Effect of Polyphenols against Breast Cancer and Cancer Stem Cells: Molecular Mechanisms Sustained proliferation in cancer: Mechanisms and novel therapeutic targets The tumor suppressor protein HBP1 is a novel c-myc-binding protein that negatively regulates c-myc transcriptional activity.Research resource: A genome-wide study identifies potential new target genes for POU1F1 Cancer stem cells: a novel paradigm for cancer prevention and treatment.Epigallocatechin-3-gallate induces mesothelioma cell death via H2 O2 -dependent T-type Ca2+ channel opening.Green tea compounds in breast cancer prevention and treatment Polyphenon E inhibits the growth of human Barrett's and aerodigestive adenocarcinoma cells by suppressing cyclin D1 expression.EGCG enhances the therapeutic potential of gemcitabine and CP690550 by inhibiting STAT3 signaling pathway in human pancreatic cancer.Novel targets for prostate cancer chemoprevention.HBP1 promoter methylation augments the oncogenic β-catenin to correlate with prognosis in NSCLC.The green tea component EGCG inhibits RNA polymerase III transcription The Wnt signaling pathway: implications for therapy in osteosarcoma.Fluorinated N,N-dialkylaminostilbenes for Wnt pathway inhibition and colon cancer repression.Green tea polyphenol EGCG suppresses Wnt/β-catenin signaling by promoting GSK-3β- and PP2A-independent β-catenin phosphorylation/degradation.Whole blueberry powder modulates the growth and metastasis of MDA-MB-231 triple negative breast tumors in nude mice Tea polyphenols for health promotion Can we safely target the WNT pathway?Implications of cancer stem cell theory for cancer chemoprevention by natural dietary compounds.Dietary compound isoliquiritigenin prevents mammary carcinogenesis by inhibiting breast cancer stem cells through WIF1 demethylation.Cancer stem cells: potential target for bioactive food components MicroRNA-221/222 confers breast cancer fulvestrant resistance by regulating multiple signaling pathways Modulation of Wnt/β-catenin signaling pathway by bioactive food components.Real-time imaging of beta-catenin dynamics in cells and living mice.Natural products as potential cancer therapy enhancers: A preclinical update.The evolving roles of canonical WNT signaling in stem cells and tumorigenesis: implications in targeted cancer therapies Cancer and diet: How are they related?The hallmarks of premalignant conditions: a molecular basis for cancer prevention Pathways to breast cancer recurrence.Bisleuconothine A, a bisindole alkaloid, inhibits colorectal cancer cell in vitro and in vivo targeting Wnt signaling.Epigallocatechin-3-gallate targets cancer stem-like cells and enhances 5-fluorouracil chemosensitivity in colorectal cancer.Novel strategies targeting cancer stem cells through phytochemicals and their analogs

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P2860

Suppression of Wnt signaling by the green tea compound (-)-epigallocatechin 3-gallate (EGCG) in invasive breast cancer cells. Requirement of the transcriptional repressor HBP1

http://www.wikidata.org/entity/Q28298718

description

2006 nî lūn-bûn

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2006 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2006 թվականի ապրիլին հրատարակված գիտական հոդված

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2006年の論文

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Suppression of Wnt signaling b ...... transcriptional repressor HBP1

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Suppression of Wnt signaling b ...... transcriptional repressor HBP1

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Suppression of Wnt signaling b ...... transcriptional repressor HBP1

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Suppression of Wnt signaling b ...... transcriptional repressor HBP1

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Suppression of Wnt signaling b ...... transcriptional repressor HBP1

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Suppression of Wnt signaling b ...... transcriptional repressor HBP1

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Suppression of Wnt signaling b ...... transcriptional repressor HBP1

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Suppression of Wnt signaling b ...... transcriptional repressor HBP1

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Suppression of Wnt signaling b ...... transcriptional repressor HBP1

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Suppression of Wnt signaling b ...... transcriptional repressor HBP1

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Amy S Yee

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David E Wazer

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Ian C Summerhayes

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K Eric Paulson

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Kimberly M Rieger-Christ

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Xiaowei Zhang

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P2860

Identification of c-MYC as a target of the APC pathway

HBP1: a HMG box transcriptional repressor that is targeted by the retinoblastoma family

Human HMG box transcription factor HBP1: a role in hCD2 LCR function

Pin1 is overexpressed in breast cancer and cooperates with Ras signaling in increasing the transcriptional activity of c-Jun towards cyclin D1.

Negative regulation of the Wnt-beta-catenin pathway by the transcriptional repressor HBP1

HMG box transcriptional repressor HBP1 maintains a proliferation barrier in differentiated liver tissue

Involvement of retinoblastoma protein and HBP1 in histone H1(0) gene expression

HBP1 repression of the p47phox gene: cell cycle regulation via the NADPH oxidase

The prolyl isomerase Pin1 in breast development and cancer

The Wnt signaling pathway in development and disease

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