Reevaluation of the virulence phenotype of the inv yadA double mutants of Yersinia pseudotuberculosis
about
Oligomeric coiled-coil adhesin YadA is a double-edged swordThe Yersinia adhesin YadA collagen-binding domain structure is a novel left-handed parallel beta-rollYersinia pestis, the cause of plague, is a recently emerged clone of Yersinia pseudotuberculosisYersinia infection tools-characterization of structure and function of adhesinsThe virulence plasmid of Yersinia, an antihost genomeFirst analysis of a bacterial collagen-binding protein with collagen Toolkits: promiscuous binding of YadA to collagens may explain how YadA interferes with host processesApically exposed, tight junction-associated beta1-integrins allow binding and YopE-mediated perturbation of epithelial barriers by wild-type Yersinia bacteria.M-cell surface beta1 integrin expression and invasin-mediated targeting of Yersinia pseudotuberculosis to mouse Peyer's patch M cellsInteractions between periodontal bacteria and human oral epithelial cells: Fusobacterium nucleatum adheres to and invades epithelial cells.Yersinia pestis pFra shows biovar-specific differences and recent common ancestry with a Salmonella enterica serovar Typhi plasmidIdentification of a domain in Yersinia virulence factor YadA that is crucial for extracellular matrix-specific cell adhesion and uptakeIntranasal inoculation of mice with Yersinia pseudotuberculosis causes a lethal lung infection that is dependent on Yersinia outer proteins and PhoP.Pathogenesis of Y. enterocolitica and Y. pseudotuberculosis in Human Yersiniosis.A multi-omic systems approach to elucidating Yersinia virulence mechanisms.Adhesins and host serum factors drive Yop translocation by yersinia into professional phagocytes during animal infection.Yersinia pseudotuberculosis uses Ail and YadA to circumvent neutrophils by directing Yop translocation during lung infection.The YadA protein of Yersinia pseudotuberculosis mediates high-efficiency uptake into human cells under environmental conditions in which invasin is repressed.Commensal bacteria can enter colonic epithelial cells and induce proinflammatory cytokine secretion: a possible pathogenic mechanism of ulcerative colitisInfluence of the Cpx extracytoplasmic-stress-responsive pathway on Yersinia sp.-eukaryotic cell contact.
P2860
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P2860
Reevaluation of the virulence phenotype of the inv yadA double mutants of Yersinia pseudotuberculosis
description
1997 nî lūn-bûn
@nan
1997 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
1997 թվականի հունվարին հրատարակված գիտական հոդված
@hy
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
name
Reevaluation of the virulence ...... of Yersinia pseudotuberculosis
@ast
Reevaluation of the virulence ...... of Yersinia pseudotuberculosis
@en
Reevaluation of the virulence ...... of Yersinia pseudotuberculosis
@nl
type
label
Reevaluation of the virulence ...... of Yersinia pseudotuberculosis
@ast
Reevaluation of the virulence ...... of Yersinia pseudotuberculosis
@en
Reevaluation of the virulence ...... of Yersinia pseudotuberculosis
@nl
prefLabel
Reevaluation of the virulence ...... of Yersinia pseudotuberculosis
@ast
Reevaluation of the virulence ...... of Yersinia pseudotuberculosis
@en
Reevaluation of the virulence ...... of Yersinia pseudotuberculosis
@nl
P2860
P1476
Reevaluation of the virulence ...... of Yersinia pseudotuberculosis
@en
P2093
P2860
P304
P407
P577
1997-01-01T00:00:00Z