Activation of NF-kappa B is required for hypertrophic growth of primary rat neonatal ventricular cardiomyocytes
about
Thioredoxin and ventricular remodelingKinetic mRNA Profiling in a Rat Model of Left-Ventricular Hypertrophy Reveals Early Expression of Chemokines and Their ReceptorsA-kinase-anchoring protein-Lbc anchors IκB kinase β to support interleukin-6-mediated cardiomyocyte hypertrophyMyotrophin/V-1, a protein up-regulated in the failing human heart and in postnatal cerebellum, converts NFkappa B p50-p65 heterodimers to p50-p50 and p65-p65 homodimersFAK-related nonkinase attenuates hypertrophy induced by angiotensin-II in cultured neonatal rat cardiac myocytesSirtuin 6 protects cardiomyocytes from hypertrophy in vitro via inhibition of NF-κB-dependent transcriptional activityCardioprotective effects of peroxisome proliferator activated receptor gamma activators on acute myocarditis: anti-inflammatory actions associated with nuclear factor kappaB blockadeNuclear factor-kappaB activation leads to down-regulation of fatty acid oxidation during cardiac hypertrophyRepression of miR-142 by p300 and MAPK is required for survival signalling via gp130 during adaptive hypertrophyThe dietary compound curcumin inhibits p300 histone acetyltransferase activity and prevents heart failure in ratsHyperosmotic stress activates p65/RelB NFkappaB in cultured cardiomyocytes with dichotomic actions on caspase activation and cell deathNuclear co-translocation of myotrophin and p65 stimulates myocyte growth. Regulation by myotrophin hairpin loopsLosartan reduced connexin43 expression in left ventricular myocardium of spontaneously hypertensive ratsIL-33 and ST2 comprise a critical biomechanically induced and cardioprotective signaling systemHeat shock protein 90 regulates IκB kinase complex and NF-κB activation in angiotensin II-induced cardiac cell hypertrophy.Transcriptome profiling of peripheral blood cells identifies potential biomarkers for doxorubicin cardiotoxicity in a rat modelRedox regulation, NF-kappaB, and atrial fibrillation.Autophagy and the ubiquitin-proteasome system in cardiac dysfunction.Superoxide scavenging and Akt inhibition in myocardium ameliorate pressure overload-induced NF-κB activation and cardiac hypertrophy.Molecular basis of lysophosphatidic acid-induced NF-κB activationThyroid Hormone-Regulated Cardiac microRNAs are Predicted to Suppress Pathological Hypertrophic Signaling.beta-Arrestin inhibits NF-kappaB activity by means of its interaction with the NF-kappaB inhibitor IkappaBalpha.MicroRNA-301a mediated regulation of Kv4.2 in diabetes: identification of key modulators.Role of the ubiquitin-proteasome system in cardiac dysfunction of adipose triglyceride lipase-deficient miceCardiomyocyte p65 nuclear factor-κB is necessary for compensatory adaptation to pressure overloadPotential role of nuclear factor κB in diabetic cardiomyopathy.Prevention and reversal of cardiac hypertrophy by soluble epoxide hydrolase inhibitors.AdipoR1/APPL1 potentiates the protective effects of globular adiponectin on angiotensin II-induced cardiac hypertrophy and fibrosis in neonatal rat atrial myocytes and fibroblastsAdiponectin mediates cardioprotection in oxidative stress-induced cardiac myocyte remodeling.Tissue kallikrein protects against pressure overload-induced cardiac hypertrophy through kinin B2 receptor and glycogen synthase kinase-3beta activationCARMA3 deficiency abrogates G protein-coupled receptor-induced NF-{kappa}B activation.Re-employment of developmental transcription factors in adult heart diseasePeroxisome proliferator-activated receptors and cardiovascular remodeling.Involvement of nuclear factor κB (NF-κB) signaling pathway in regulation of cardiac G protein-coupled receptor kinase 5 (GRK5) expression.Interaction between NFκB and NFAT coordinates cardiac hypertrophy and pathological remodeling.Activation of AP-1 contributes to the beta-adrenoceptor-mediated myocardial induction of interleukin-6.Activated vitamin D attenuates left ventricular abnormalities induced by dietary sodium in Dahl salt-sensitive animals.Myocardial NF-κB activation is essential for zebrafish heart regeneration.TAK1 Regulates Myocardial Response to Pathological Stress via NFAT, NFκB, and Bnip3 Pathways.A unique microRNA profile in end-stage heart failure indicates alterations in specific cardiovascular signaling networks.
P2860
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P2860
Activation of NF-kappa B is required for hypertrophic growth of primary rat neonatal ventricular cardiomyocytes
description
2001 nî lūn-bûn
@nan
2001 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Activation of NF-kappa B is re ...... tal ventricular cardiomyocytes
@ast
Activation of NF-kappa B is re ...... tal ventricular cardiomyocytes
@en
Activation of NF-kappa B is re ...... tal ventricular cardiomyocytes
@nl
type
label
Activation of NF-kappa B is re ...... tal ventricular cardiomyocytes
@ast
Activation of NF-kappa B is re ...... tal ventricular cardiomyocytes
@en
Activation of NF-kappa B is re ...... tal ventricular cardiomyocytes
@nl
prefLabel
Activation of NF-kappa B is re ...... tal ventricular cardiomyocytes
@ast
Activation of NF-kappa B is re ...... tal ventricular cardiomyocytes
@en
Activation of NF-kappa B is re ...... tal ventricular cardiomyocytes
@nl
P2093
P2860
P3181
P356
P1476
Activation of NF-kappa B is re ...... tal ventricular cardiomyocytes
@en
P2093
P2860
P304
P3181
P356
10.1073/PNAS.111155798
P407
P577
2001-06-05T00:00:00Z