Effects of nitric oxide synthase inhibitors on murine infection with Mycobacterium tuberculosis - Test2 - elhamkhani - TriplyDB

Effects of nitric oxide synthase inhibitors on murine infection with Mycobacterium tuberculosis

Effects of nitric oxide synthase inhibitors on murine infection with Mycobacterium tuberculosis

http://www.wikidata.org/entity/Q28367637

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1,25-Dihydroxyvitamin D3 induces nitric oxide synthase and suppresses growth of Mycobacterium tuberculosis in a human macrophage-like cell line Theiler's virus infection: a model for multiple sclerosis Reactive oxygen and nitrogen intermediates in the relationship between mammalian hosts and microbial pathogens The emerging role of gasotransmitters in the pathogenesis of tuberculosis Redox homeostasis in mycobacteria: the key to tuberculosis control?The Role of Nitric Oxide in Mycobacterial Infections TB drug development: immunology at the table Tuberculosis: latency and reactivation Modulation of Mycobacterium bovis-specific responses of bovine peripheral blood mononuclear cells by 1,25-dihydroxyvitamin D(3)DevS, a heme-containing two-component oxygen sensor of Mycobacterium tuberculosis Deletion of the Mycobacterium tuberculosis pknH gene confers a higher bacillary load during the chronic phase of infection in BALB/c mice Function and regulation of class I ribonucleotide reductase-encoding genes in mycobacteria Truncated hemoglobin HbN protects Mycobacterium bovis from nitric oxide Response of Mycobacterium tuberculosis to reactive oxygen and nitrogen intermediates Response to reactive nitrogen intermediates in Mycobacterium tuberculosis: induction of the 16-kilodalton alpha-crystallin homolog by exposure to nitric oxide donors Cholesterol catabolism by Mycobacterium tuberculosis requires transcriptional and metabolic adaptations Mycobacterial persistence requires the utilization of host cholesterol Regulation of the Mycobacterium tuberculosis hypoxic response gene encoding alpha -crystallin Proteasomal control of cytokinin synthesis protects Mycobacterium tuberculosis against nitric oxide A cooperative oxygen-binding hemoglobin from Mycobacterium tuberculosis Reengineering redox sensitive GFP to measure mycothiol redox potential of Mycobacterium tuberculosis during infection Mycobacteria counteract a TLR-mediated nitrosative defense mechanism in a zebrafish infection model Transient loss of resistance to pulmonary tuberculosis in p47(phox-/-) mice Genetic requirements for mycobacterial survival during infection Identification of nitric oxide synthase as a protective locus against tuberculosis Macrophage migration inhibitory factor (MIF) is a critical mediator of the innate immune response to Mycobacterium tuberculosis.Convergence of IL-1beta and VDR activation pathways in human TLR2/1-induced antimicrobial responses Role of inducible nitric oxide synthase in resistance to Mycobacterium leprae in mice The Mycobacterium tuberculosis cytochrome P450 system.Therapeutic effects of nitric oxide inhibition during experimental fecal peritonitis: role of interleukin-10 and monocyte chemoattractant protein 1.Protective role of nitric oxide in Staphylococcus aureus infection in mice.Neither interleukin-6 nor inducible nitric oxide synthase is required for clearance of Chlamydia trachomatis from the murine genital tract epithelium.A tumor necrosis factor mimetic peptide activates a murine macrophage cell line to inhibit mycobacterial growth in a nitric oxide-dependent fashion CD4 T cells promote rather than control tuberculosis in the absence of PD-1-mediated inhibition Acute immune response to Mycobacterium massiliense in C57BL/6 and BALB/c mice.Differential effects of total and partial neutralization of tumor necrosis factor on cell-mediated immunity to Mycobacterium bovis BCG infection.Inhaled nitric oxide treatment of patients with pulmonary tuberculosis evidenced by positive sputum smears.Survival of Mycobacterium avium and Mycobacterium tuberculosis in acidified vacuoles of murine macrophages T-Cell hyporesponsiveness induced by activated macrophages through nitric oxide production in mice infected with Mycobacterium tuberculosis.Glucose 6-phosphate dehydrogenase is required for Salmonella typhimurium virulence and resistance to reactive oxygen and nitrogen intermediates.

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P2860

Effects of nitric oxide synthase inhibitors on murine infection with Mycobacterium tuberculosis

http://www.wikidata.org/entity/Q28367637

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1995 nî lūn-bûn

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1995 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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1995 թվականի փետրվարին հրատարակված գիտական հոդված

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1995年の論文

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1995年論文

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1995年論文

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1995年論文

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1995年論文

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1995年論文

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1995年论文

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Effects of nitric oxide syntha ...... ith Mycobacterium tuberculosis

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Effects of nitric oxide syntha ...... ith Mycobacterium tuberculosis

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Effects of nitric oxide syntha ...... ith Mycobacterium tuberculosis

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Effects of nitric oxide syntha ...... ith Mycobacterium tuberculosis

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Effects of nitric oxide syntha ...... ith Mycobacterium tuberculosis

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Effects of nitric oxide syntha ...... ith Mycobacterium tuberculosis

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Infection and Immunity

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Effects of nitric oxide syntha ...... ith Mycobacterium tuberculosis

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B R Bloom

http://www.w3.org/2001/XMLSchema#string

D Carroll

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J Chan

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J Flynn

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P2860

Neutralization of gamma interferon and tumor necrosis factor alpha blocks in vivo synthesis of nitrogen oxides from L-arginine and protection against Francisella tularensis infection in Mycobacterium bovis BCG-treated mice

Induction of nitric oxide synthase protects against malaria in mice exposed to irradiated Plasmodium berghei infected mosquitoes: involvement of interferon gamma and CD8+ T cells

Multiple defects of immune cell function in mice with disrupted interferon-gamma genes

An essential role for interferon gamma in resistance to Mycobacterium tuberculosis infection

Killing of virulent Mycobacterium tuberculosis by reactive nitrogen intermediates produced by activated murine macrophages

Disseminated tuberculosis in interferon gamma gene-disrupted mice

Nitric oxide as a secretory product of mammalian cells

The 1991 Ulf von Euler Lecture. The L-arginine: nitric oxide pathway.

Nitric oxide produced during murine listeriosis is protective

Mice that lack the interferon-gamma receptor have profoundly altered responses to infection with Bacillus Calmette-Guérin and subsequent challenge with lipopolysaccharide.

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736-40

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201208

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2

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63

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1995-02-01T00:00:00Z

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7529749

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Mycobacterium tuberculosis

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173063

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