Evaluation of the association between persistent organic pollutants (POPs) and diabetes in epidemiological studies: a national toxicology program workshop review - Test2 - elhamkhani - TriplyDB

Evaluation of the association between persistent organic pollutants (POPs) and diabetes in epidemiological studies: a national toxicology program workshop review

Evaluation of the association between persistent organic pollutants (POPs) and diabetes in epidemiological studies: a national toxicology program workshop review

http://www.wikidata.org/entity/Q28385151

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Urinary polycyclic aromatic hydrocarbon biomarkers and diabetes mellitus Insulin resistance and environmental pollutants: experimental evidence and future perspectives Environmental chemicals and type 2 diabetes: an updated systematic review of the epidemiologic evidence EDC-2: The Endocrine Society's Second Scientific Statement on Endocrine-Disrupting Chemicals Association of dioxin and other persistent organic pollutants (POPs) with diabetes: epidemiological evidence and new mechanisms of beta cell dysfunction Occupational and environmental exposure to pesticides and cytokine pathways in chronic diseases (Review)Dose-response relationship between serum 2,3,7,8-tetrachlorodibenzo-p-dioxin and diabetes mellitus: a meta-analysis Can exposure to environmental chemicals increase the risk of diabetes type 1 development?Persistent organic pollutants as risk factors for type 2 diabetes Serum Levels of Persistent Organic Pollutants and Insulin Secretion among Children Age 7-9 Years: A Prospective Cohort Study Association between chlorinated pesticides in the serum of prepubertal Russian boys and longitudinal biomarkers of metabolic function Persistent organic pollutants and biomarkers of diabetes risk in a cohort of Great Lakes sport caught fish consumers Prioritizing Environmental Chemicals for Obesity and Diabetes Outcomes Research: A Screening Approach Using ToxCast™ High-Throughput Data Metabolism Disrupting Chemicals and Metabolic Disorders Fruit and vegetable intake, as reflected by serum carotenoid concentrations, predicts reduced probability of polychlorinated biphenyl-associated risk for type 2 diabetes: National Health and Nutrition Examination Survey 2003-2004 Chlorinated persistent organic pollutants, obesity, and type 2 diabetes Environmental endocrine disruption of energy metabolism and cardiovascular risk.2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD)-elicited effects on bile acid homeostasis: Alterations in biosynthesis, enterohepatic circulation, and microbial metabolism.Perinatal exposure of mice to the pesticide DDT impairs energy expenditure and metabolism in adult female offspring.Analysis of polychlorinated biphenyls and organochlorine pesticides in archived dried blood spots and its application to track temporal trends of environmental chemicals in newborns.Persistent organic pollutants and obesity: are they potential mechanisms for breast cancer promotion?Perfluorooctanoic acid exposure for 28 days affects glucose homeostasis and induces insulin hypersensitivity in mice Aryl hydrocarbon receptor deficiency protects mice from diet-induced adiposity and metabolic disorders through increased energy expenditure.Number of Persistent Organic Pollutants Detected at High Concentrations in Blood Samples of the United States Population.Persistent organic pollutants and non-alcoholic fatty liver disease in morbidly obese patients: a cohort study.Inulin Supplementation Lowered the Metabolic Defects of Prolonged Exposure to Chlorpyrifos from Gestation to Young Adult Stage in Offspring Rats.Serum concentrations of polychlorinated biphenyls (PCBs) in participants of the Anniston Community Health Survey Pyruvate Kinase Isoform Switching and Hepatic Metabolic Reprogramming by the Environmental Contaminant 2,3,7,8-Tetrachlorodibenzo-p-Dioxin Environmental exposure to BDE47 is associated with increased diabetes prevalence: Evidence from community-based case-control studies and an animal experiment.A Systems Biology Approach Reveals Converging Molecular Mechanisms that Link Different POPs to Common Metabolic Diseases 2,3,7,8-Tetrachlorodibenzo-p-Dioxin Alters Lipid Metabolism and Depletes Immune Cell Populations in the Jejunum of C57BL/6 Mice New approaches in human health risk assessment.The Effects of Endocrine Disruptors on Adipogenesis and Osteogenesis in Mesenchymal Stem Cells: A Review.A computational approach to chemical etiologies of diabetes.Lean Fish Consumption Is Associated with Beneficial Changes in the Metabolic Syndrome Components: A 13-Year Follow-Up Study from the Norwegian Tromsø Study.Bisphenol A (BPA) aggravates multiple low-dose streptozotocin-induced Type 1 diabetes in C57BL/6 mice.Methodological issues in human studies of endocrine disrupting chemicals.Impact of nutrition on pollutant toxicity: an update with new insights into epigenetic regulation.Food contaminants and programming of type 2 diabetes: recent findings from animal studies.Lipidomic Evaluation of Aryl Hydrocarbon Receptor-Mediated Hepatic Steatosis in Male and Female Mice Elicited by 2,3,7,8-Tetrachlorodibenzo-p-dioxin.

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Evaluation of the association between persistent organic pollutants (POPs) and diabetes in epidemiological studies: a national toxicology program workshop review

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An Environment-Wide Association Study (EWAS) on type 2 diabetes mellitus

Reduction in the Incidence of Type 2 Diabetes with Lifestyle Intervention or Metformin

A strong dose-response relation between serum concentrations of persistent organic pollutants and diabetes results from the National Health and Examination Survey 1999-2002

Health status of Army Chemical Corps Vietnam veterans who sprayed defoliant in Vietnam

Dioxin and diabetes mellitus: an analysis of the combined NIOSH and Ranch Hand data

Hormones and endocrine-disrupting chemicals: low-dose effects and nonmonotonic dose responses

Association between serum perfluorooctanoic acid (PFOA) and thyroid disease in the U.S. National Health and Nutrition Examination Survey

Exposure to polyfluoroalkyl chemicals and cholesterol, body weight, and insulin resistance in the general U.S. population

A cross-sectional study of the association between persistent organochlorine pollutants and diabetes

Chronic consumption of farmed salmon containing persistent organic pollutants causes insulin resistance and obesity in mice

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maturity-onset diabetes of the young

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