Glial tumor necrosis factor alpha (TNFα) generates metaplastic inhibition of spinal learning - Test2 - elhamkhani - TriplyDB

Glial tumor necrosis factor alpha (TNFα) generates metaplastic inhibition of spinal learning

Glial tumor necrosis factor alpha (TNFα) generates metaplastic inhibition of spinal learning

http://www.wikidata.org/entity/Q28385590

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Tumor necrosis factor alpha: a link between neuroinflammation and excitotoxicity Metaplasticity and behavior: how training and inflammation affect plastic potential within the spinal cord and recovery after injury Learning from the spinal cord: how the study of spinal cord plasticity informs our view of learning Combined SCI and TBI: recovery of forelimb function after unilateral cervical spinal cord injury (SCI) is retarded by contralateral traumatic brain injury (TBI), and ipsilateral TBI balances the effects of SCI on paw placement Regulatory effects of inhibiting the activation of glial cells on retinal synaptic plasticity Regulatory effects of intermittent noxious stimulation on spinal cord injury-sensitive microRNAs and their presumptive targets following spinal cord contusion Peripheral noxious stimulation reduces withdrawal threshold to mechanical stimuli after spinal cord injury: role of tumor necrosis factor alpha and apoptosis.Stress exacerbates neuron loss and microglia proliferation in a rat model of excitotoxic lower motor neuron injury Learning about time within the spinal cord: evidence that spinal neurons can abstract and store an index of regularity.Impact of behavioral control on the processing of nociceptive stimulation.Central nociceptive sensitization vs. spinal cord training: opposing forms of plasticity that dictate function after complete spinal cord injury Maladaptive spinal plasticity opposes spinal learning and recovery in spinal cord injury.AMPA Receptor Phosphorylation and Synaptic Colocalization on Motor Neurons Drive Maladaptive Plasticity below Complete Spinal Cord Injury.Learning about Time within the Spinal Cord II: Evidence that Temporal Regularity Is Encoded by a Spinal Oscillator.Elevated MMP-9 in the lumbar cord early after thoracic spinal cord injury impedes motor relearning in mice.Anti-Inflammatory Mechanism of Neural Stem Cell Transplantation in Spinal Cord Injury Managing inflammation after spinal cord injury through manipulation of macrophage function What Is Being Trained? How Divergent Forms of Plasticity Compete To Shape Locomotor Recovery after Spinal Cord Injury.AMPA receptor mediated behavioral plasticity in the isolated rat spinal cord.Lumbar Myeloid Cell Trafficking into Locomotor Networks after Thoracic Spinal Cord Injury.When Pain Hurts: Nociceptive Stimulation Induces a State of Maladaptive Plasticity and Impairs Recovery after Spinal Cord Injury.Acute spinal cord injury (SCI) transforms how GABA affects nociceptive sensitization.Metaplasticity within the spinal cord: Evidence brain-derived neurotrophic factor (BDNF), tumor necrosis factor (TNF), and alterations in GABA function (ionic plasticity) modulate pain and the capacity to learn.Pain Input After Spinal Cord Injury (SCI) Undermines Long-Term Recovery and Engages Signal Pathways That Promote Cell Death.

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Glial tumor necrosis factor alpha (TNFα) generates metaplastic inhibition of spinal learning

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Glial tumor necrosis factor al ...... inhibition of spinal learning

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Glial tumor necrosis factor al ...... inhibition of spinal learning

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Glial tumor necrosis factor al ...... inhibition of spinal learning

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Glial tumor necrosis factor al ...... inhibition of spinal learning

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Glial tumor necrosis factor al ...... inhibition of spinal learning

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Glial tumor necrosis factor al ...... inhibition of spinal learning

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J Russell Huie

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James W Grau

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Kuan H Lee

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Michael S Beattie

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P2860

Mice deficient in TNF receptors are protected against dopaminergic neurotoxicity: implications for Parkinson's disease

High force reaching task induces widespread inflammation, increased spinal cord neurochemicals and neuropathic pain

Effect of epidural stimulation of the lumbosacral spinal cord on voluntary movement, standing, and assisted stepping after motor complete paraplegia: a case study

Bidirectional synaptic plasticity: from theory to reality

Human tumor necrosis factor receptor p75/80 (CD120b) gene structure and promoter characterization

Evidence for the involvement of TNF and NF-kappaB in hippocampal synaptic plasticity

TNF-R1 signaling: a beautiful pathway

Differential regulation of AMPA receptor and GABA receptor trafficking by tumor necrosis factor-alpha

Metaplasticity: the plasticity of synaptic plasticity

Brain-derived neurotrophic factor promotes adaptive plasticity within the spinal cord and mediates the beneficial effects of controllable stimulation

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Adam R. Ferguson

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