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Genome-wide association study identifies chromosome 10q24.32 variants associated with arsenic metabolism and toxicity phenotypes in BangladeshArsenic exposure and hepatitis Evirus infection during pregnancyHepatocellular carcinoma and the risk of occupational exposureEnvironmental toxicants, epigenetics, and cancerArsenic exposure, dietary patterns, and skin lesion risk in bangladesh: a prospective studyArsenic Toxicity in Male Reproduction and DevelopmentRisk of hepatocellular carcinoma in workers exposed to chemicalsArsenic exposure and cancer mortality in a US-based prospective cohort: the strong heart studyAkt activation is responsible for enhanced migratory and invasive behavior of arsenic-transformed human bronchial epithelial cellsRole of pigment epithelium-derived factor (PEDF) in arsenic-induced cell apoptosis of liver and brain in a rat modelTowards prenatal biomonitoring in North Carolina: assessing arsenic, cadmium, mercury, and lead levels in pregnant womenAir pollution, UV irradiation and skin carcinogenesis: what we know, where we stand and what is likely to happen in the future?Amelioration of arsenic-induced toxic effects in mice by dietary supplementation of Syzygium cumini leaf extract.Association between genome-wide copy number variation and arsenic-induced skin lesions: a prospective study.Monomethylarsonous acid produces irreversible events resulting in malignant transformation of a human bladder cell line following 12 weeks of low-level exposureExposure to multiple metals from groundwater-a global crisis: geology, climate change, health effects, testing, and mitigation.Arsenic abrogates the estrogen-signaling pathway in the rat uterusAssociation of AS3MT polymorphisms and the risk of premalignant arsenic skin lesions.Arsenite promotes intestinal tumor cell proliferation and invasion by stimulating epithelial-to-mesenchymal transitionAssociation of arsenic exposure with lung cancer incidence rates in the United States.The broad scope of health effects from chronic arsenic exposure: update on a worldwide public health problem.Arsenic immunotoxicity: a review.Arsenic exposure, telomere length, and expression of telomere-related genes among Bangladeshi individualsReversal and prevention of arsenic-induced human bronchial epithelial cell malignant transformation by microRNA-200b.Genome-wide identification of molecular pathways and biomarkers in response to arsenic exposure in zebrafish liver.Cancer-specific mortality in chronic kidney disease: longitudinal follow-up of a large cohort.Nitric oxide donor, V-PROLI/NO, provides protection against arsenical induced toxicity in rat liver cells: requirement for Cyp1a1Trace metal imaging in diagnostic of hepatic metal disease.The Genetic Architecture of Arsenic Metabolism Efficiency:A SNP-Based Heritability Study of Bangladeshi Adults.Hepatic and Nephric NRF2 Pathway Up-Regulation, an Early Antioxidant Response, in Acute Arsenic-Exposed Mice.Analysis of the transcriptional regulation of cancer-related genes by aberrant DNA methylation of the cis-regulation sites in the promoter region during hepatocyte carcinogenesis caused by arsenic.Chronic Arsenic Poisoning Probably Caused by Arsenic-Based Pesticides: Findings from an Investigation Study of a Household.Activation of the Nrf2 pathway by inorganic arsenic in human hepatocytes and the role of transcriptional repressor Bach1In Vivo Effect of Arsenic Trioxide on Keap1-p62-Nrf2 Signaling Pathway in Mouse Liver: Expression of Antioxidant Responsive Element-Driven Genes Related to Glutathione Metabolism.Epithelial to mesenchymal transition in arsenic-transformed cells promotes angiogenesis through activating β-catenin-vascular endothelial growth factor pathway.Folate deficiency, hyperhomocysteinemia, low urinary creatinine, and hypomethylation of leukocyte DNA are risk factors for arsenic-induced skin lesionsV-PROLI/NO, a nitric oxide donor prodrug, protects liver cells from arsenic-induced toxicity.Evaluation of protective effect of N-acetyl cysteine on arsenic-induced hepatotoxicityDistinct Nrf1/2-independent mechanisms mediate As 3+-induced glutamate-cysteine ligase subunit gene expression in murine hepatocytes.Biological responses to arsenic compounds
P2860
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P2860
description
2008 nî lūn-bûn
@nan
2008 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Liver is a target of arsenic carcinogenesis
@ast
Liver is a target of arsenic carcinogenesis
@en
Liver is a target of arsenic carcinogenesis
@nl
type
label
Liver is a target of arsenic carcinogenesis
@ast
Liver is a target of arsenic carcinogenesis
@en
Liver is a target of arsenic carcinogenesis
@nl
prefLabel
Liver is a target of arsenic carcinogenesis
@ast
Liver is a target of arsenic carcinogenesis
@en
Liver is a target of arsenic carcinogenesis
@nl
P2860
P356
P1476
Liver is a target of arsenic carcinogenesis
@en
P2093
Michael P Waalkes
P2860
P356
10.1093/TOXSCI/KFN120
P407
P577
2008-09-01T00:00:00Z