Silencing of Hint1, a novel tumor suppressor gene, by promoter hypermethylation in hepatocellular carcinoma - Test2 - elhamkhani - TriplyDB

Silencing of Hint1, a novel tumor suppressor gene, by promoter hypermethylation in hepatocellular carcinoma

Silencing of Hint1, a novel tumor suppressor gene, by promoter hypermethylation in hepatocellular carcinoma

http://www.wikidata.org/entity/Q28392002

about

High-resolution X-ray structure of the rabbit histidine triad nucleotide-binding protein 1 (rHINT1)-adenosine complex at 1.10 Å resolution A new crystal form of human histidine triad nucleotide-binding protein 1 (hHINT1) in complex with adenosine 5′-monophosphate at 1.38 Å resolution Update on occult hepatitis B virus infection Axonal neuropathy with neuromyotonia: there is a HINT.Disruption of the histidine triad nucleotide-binding hint2 gene in mice affects glycemic control and mitochondrial function.Germline polymorphisms discovered via a cell-based, genome-wide approach predict platinum response in head and neck cancers DNA methylation, microRNAs, and their crosstalk as potential biomarkers in hepatocellular carcinoma.Global hypomethylation in hepatocellular carcinoma and its relationship to aflatoxin B(1) exposure.Disruption of tumor suppressor gene Hint1 leads to remodeling of the lipid metabolic phenotype of mouse liver.Molecular Liver Cancer Prevention in Cirrhosis by Organ Transcriptome Analysis and Lysophosphatidic Acid Pathway Inhibition.Dynamic interplay between locus-specific DNA methylation and hydroxymethylation regulates distinct biological pathways in prostate carcinogenesis.Hint1 Up-Regulates IκBα by Targeting the β-TrCP Subunit of SCF E3 Ligase in Human Hepatocellular Carcinoma Cells.The tumor suppressor HINT1 regulates MITF and β-catenin transcriptional activity in melanoma cells.Family history of gastric cancer correlates with decreased expression of HINT1 tumor suppressor gene in gastric mucosa of dyspeptic patients Expression, purification, crystallization and preliminary X-ray crystallographic analysis of human histidine triad nucleotide-binding protein 2 (hHINT2).Phosphoramidate hydrolysis catalyzed by human histidine triad nucleotide binding protein 1 (hHint1): a cluster-model DFT computational study.HINT1 in Neuropsychiatric Diseases: A Potential Neuroplastic Mediator.Hint1 suppresses migration and invasion of hepatocellular carcinoma cells in vitro by modulating girdin activity.Predicting hepatocellular carcinoma development for cirrhosis patients via methylation detection of heparocarcinogenesis-related genes.

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P2860

Silencing of Hint1, a novel tumor suppressor gene, by promoter hypermethylation in hepatocellular carcinoma

http://www.wikidata.org/entity/Q28392002

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2009 nî lūn-bûn

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2009 թուականի Մարտին հրատարակուած գիտական յօդուած

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2009 թվականի մարտին հրատարակված գիտական հոդված

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Silencing of Hint1, a novel tu ...... on in hepatocellular carcinoma

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Silencing of Hint1, a novel tu ...... on in hepatocellular carcinoma

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Haiyang Li

http://www.w3.org/2001/XMLSchema#string

Hui-Chen Wu

http://www.w3.org/2001/XMLSchema#string

I Bernard Weinstein

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Jing Shen

http://www.w3.org/2001/XMLSchema#string

Regina M Santella

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Yu-Jing Zhang

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P2860

Aberrant gene expression in human non small cell lung carcinoma cells exposed to demethylating agent 5-aza-2 '-deoxycytidine

The histidine triad protein Hint1 triggers apoptosis independent of its enzymatic activity

Methylation-specific PCR: a novel PCR assay for methylation status of CpG islands

HINT1 inhibits beta-catenin/TCF4, USF2 and NFkappaB activity in human hepatoma cells

Hint, Fhit, and GalT: function, structure, evolution, and mechanism of three branches of the histidine triad superfamily of nucleotide hydrolases and transferases

Aberrant Promoter Methylation Profiles of Tumor Suppressor Genes in Hepatocellular Carcinoma

A gene hypermethylation profile of human cancer

Hint1 inhibits growth and activator protein-1 activity in human colon cancer cells.

Epidemiology of primary liver cancer.

DNA hypermethylation in tumorigenesis: epigenetics joins genetics.

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