JNK and STAT3 signaling pathways converge on Akt-mediated phosphorylation of EZH2 in bronchial epithelial cells induced by arsenic
about
Current understanding of mdig/MINA in human cancersPolycomb Group (PcG) Proteins and Human Cancers: Multifaceted Functions and Therapeutic ImplicationsArsenic-induced sub-lethal stress reprograms human bronchial epithelial cells to CD61¯ cancer stem cellsReactive oxygen species contribute to arsenic-induced EZH2 phosphorylation in human bronchial epithelial cells and lung cancer cellsOxidative stress, epigenetics, and cancer stem cells in arsenic carcinogenesis and preventionCulture conditions profoundly impact phenotype in BEAS-2B, a human pulmonary epithelial modelMolecular features in arsenic-induced lung tumorsFilamin A phosphorylation by Akt promotes cell migration in response to arsenicPARP-1 overexpression contributes to Cadmium-induced death in rat proximal tubular cells via parthanatos and the MAPK signalling pathwayFeedback regulations of miR-21 and MAPKs via Pdcd4 and Spry1 are involved in arsenite-induced cell malignant transformationActivation of SAPK/JNK mediated the inhibition and reciprocal interaction of DNA methyltransferase 1 and EZH2 by ursolic acid in human lung cancer cells.MiR-148a Functions as a Tumor Suppressor by Targeting CCK-BR via Inactivating STAT3 and Akt in Human Gastric CancerJNK1/2 expression and modulation of STAT3 signaling in oral cancer.Inhibition of EZH2 via activation of SAPK/JNK and reduction of p65 and DNMT1 as a novel mechanism in inhibition of human lung cancer cells by polyphyllin I.Monoallelic loss of tumor suppressor GRIM-19 promotes tumorigenesis in mice.Regulation and role of post-translational modifications of enhancer of zeste homologue 2 in cancer developmentCarcinogenic metalloid arsenic induces expression of mdig oncogene through JNK and STAT3 activation.Update of research on the role of EZH2 in cancer progression.Gefitinib resistance resulted from STAT3-mediated Akt activation in lung cancer cells.Linking JNK-STAT3-Akt signaling axis to EZH2 phosphorylation: a novel pathway of carcinogenesisChronic arsenic intoxication diagnostic score (CAsIDS).The Role of the Histone Methyltransferase Enhancer of Zeste Homolog 2 (EZH2) in the Pathobiological Mechanisms Underlying Inflammatory Bowel Disease (IBD).Low concentration arsenite activated JAK2/STAT3 signal and increased proliferative factor expressions in SV-HUC-1cells after short and long time treatment.Melatonin inhibits tumorigenicity of glioblastoma stem-like cells via the AKT-EZH2-STAT3 signaling axis.
P2860
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P2860
JNK and STAT3 signaling pathways converge on Akt-mediated phosphorylation of EZH2 in bronchial epithelial cells induced by arsenic
description
2013 nî lūn-bûn
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2013 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2013年の論文
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2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
JNK and STAT3 signaling pathwa ...... elial cells induced by arsenic
@ast
JNK and STAT3 signaling pathwa ...... elial cells induced by arsenic
@en
JNK and STAT3 signaling pathwa ...... elial cells induced by arsenic
@nl
type
label
JNK and STAT3 signaling pathwa ...... elial cells induced by arsenic
@ast
JNK and STAT3 signaling pathwa ...... elial cells induced by arsenic
@en
JNK and STAT3 signaling pathwa ...... elial cells induced by arsenic
@nl
prefLabel
JNK and STAT3 signaling pathwa ...... elial cells induced by arsenic
@ast
JNK and STAT3 signaling pathwa ...... elial cells induced by arsenic
@en
JNK and STAT3 signaling pathwa ...... elial cells induced by arsenic
@nl
P2093
P2860
P3181
P356
P1433
P1476
JNK and STAT3 signaling pathwa ...... elial cells induced by arsenic
@en
P2093
Bailing Chen
Qingshan Chang
P2860
P304
P3181
P356
10.4161/CC.23030
P407
P577
2012-12-19T00:00:00Z