Aryl hydrocarbon receptor (AhR) regulates silica-induced inflammation but not fibrosis - Test2 - elhamkhani - TriplyDB

Aryl hydrocarbon receptor (AhR) regulates silica-induced inflammation but not fibrosis

Aryl hydrocarbon receptor (AhR) regulates silica-induced inflammation but not fibrosis

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Toward understanding the role of aryl hydrocarbon receptor in the immune system: current progress and future trends Acute Exposure to Crystalline Silica Reduces Macrophage Activation in Response to Bacterial Lipoproteins Activation of Proinflammatory Responses in Cells of the Airway Mucosa by Particulate Matter: Oxidant- and Non-Oxidant-Mediated Triggering Mechanisms Uncoupling between inflammatory and fibrotic responses to silica: evidence from MyD88 knockout mice IL-33 mediates multi-walled carbon nanotube (MWCNT)-induced airway hyper-reactivity via the mobilization of innate helper cells in the lung.Innate immune activation by inhaled lipopolysaccharide, independent of oxidative stress, exacerbates silica-induced pulmonary fibrosis in mice.Development of an integrated approach for comparison of in vitro and in vivo responses to particulate matter.3,3'-Diindolylmethane Inhibits Flt3L/GM-CSF-induced-bone Marrow-derived CD103(+) Dendritic Cell Differentiation Regulating Phosphorylation of STAT3 and STAT5.Novel cellular targets of AhR underlie alterations in neutrophilic inflammation and inducible nitric oxide synthase expression during influenza virus infection.Aryl Hydrocarbon Receptor Activation Synergistically Induces Lipopolysaccharide-Mediated Expression of Proinflammatory Chemokine (c-c motif) Ligand 20.Role of the aryl hydrocarbon receptor (AhR) in lung inflammation Early life exposure to environmental tobacco smoke alters immune response to asbestos via a shift in inflammatory phenotype resulting in increased disease development.Cytokines as molecular targets for aryl hydrocarbon receptor ligands: implications for toxicity and xenobiotic detoxification.Aryl hydrocarbon receptor mediates both proinflammatory and anti-inflammatory effects in lipopolysaccharide-activated microglia.AhR and Arnt differentially regulate NF-κB signaling and chemokine responses in human bronchial epithelial cells.2,3,7,8-tetrachlorodibenzo-p-dioxin decrease expression of aryl hydrocarbon receptor in peripheral lymphocyte of β-thalassemia major patients.Ligand-mediated cytoplasmic retention of the Ah receptor inhibits macrophage-mediated acute inflammatory responses.Aryl Hydrocarbon Receptor Activation Modulates Intestinal Epithelial Barrier Function by Maintaining Tight Junction Integrity.

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Aryl hydrocarbon receptor (AhR) regulates silica-induced inflammation but not fibrosis

http://www.wikidata.org/entity/Q28397728

description

2012 nî lūn-bûn

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2012 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի ապրիլին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

Aryl hydrocarbon receptor (AhR) regulates silica-induced inflammation but not fibrosis

@ast

Aryl hydrocarbon receptor (AhR) regulates silica-induced inflammation but not fibrosis

@en

Aryl hydrocarbon receptor (AhR) regulates silica-induced inflammation but not fibrosis

@nl

type

label

Aryl hydrocarbon receptor (AhR) regulates silica-induced inflammation but not fibrosis

@ast

Aryl hydrocarbon receptor (AhR) regulates silica-induced inflammation but not fibrosis

@en

Aryl hydrocarbon receptor (AhR) regulates silica-induced inflammation but not fibrosis

@nl

prefLabel

Aryl hydrocarbon receptor (AhR) regulates silica-induced inflammation but not fibrosis

@ast

Aryl hydrocarbon receptor (AhR) regulates silica-induced inflammation but not fibrosis

@en

Aryl hydrocarbon receptor (AhR) regulates silica-induced inflammation but not fibrosis

@nl

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Toxicological Sciences

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Aryl hydrocarbon receptor (AhR) regulates silica-induced inflammation but not fibrosis

@en

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Benjamin P Seaver

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David M Shepherd

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The serpin proteinase inhibitor 9 is an endogenous inhibitor of interleukin 1beta-converting enzyme (caspase-1) activity in human vascular smooth muscle cells

Occupational exposure to crystalline silica and risk of systemic lupus erythematosus: a population-based, case-control study in the southeastern United States

Time course of pulmonary response of rats to inhalation of crystalline silica: NF-kappa B activation, inflammation, cytokine production, and damage

Progression of lung inflammation and damage in rats after cessation of silica inhalation

The sources of inflammatory mediators in the lung after silica exposure

Diseases caused by silica: mechanisms of injury and disease development

Effect of inhaled crystalline silica in a rat model: time course of pulmonary reactions

Interleukin-10 and the interleukin-10 receptor

Pyrin-only protein 2 modulates NF-kappaB and disrupts ASC:CLR interactions

Bcl-2 and Bcl-XL regulate proinflammatory caspase-1 activation by interaction with NALP1

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554-568

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scholarly article

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4100586

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10.1093/TOXSCI/KFS024

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2

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126

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Celine A Beamer

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2012-01-23T00:00:00Z

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221774024

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22273745

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3307612

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