The mismatch repair system (mutS, mutL and uvrD genes) in Pseudomonas aeruginosa: molecular characterization of naturally occurring mutants
about
Evolution of mutation rates in bacteriaPseudomonas aeruginosa biofilms in cystic fibrosisnfxB as a novel target for analysis of mutation spectra in Pseudomonas aeruginosaGenome analysis of a transmissible lineage of pseudomonas aeruginosa reveals pathoadaptive mutations and distinct evolutionary paths of hypermutatorsGenHtr: a tool for comparative assessment of genetic heterogeneity in microbial genomes generated by massive short-read sequencing.Dynamics of mutator and antibiotic-resistant populations in a pharmacokinetic/pharmacodynamic model of Pseudomonas aeruginosa biofilm treatment.Mutator genes giving rise to decreased antibiotic susceptibility in Pseudomonas aeruginosa.Novel genetic determinants of low-level aminoglycoside resistance in Pseudomonas aeruginosaThe Pseudomonas aeruginosa pfpI gene plays an antimutator role and provides general stress protectionEpistatic roles for Pseudomonas aeruginosa MutS and DinB (DNA Pol IV) in coping with reactive oxygen species-induced DNA damage.Genetic analysis of Pseudomonas aeruginosa isolates from the sputa of Australian adult cystic fibrosis patients.Evolution and adaptation in Pseudomonas aeruginosa biofilms driven by mismatch repair system-deficient mutators.Mucoidy, quorum sensing, mismatch repair and antibiotic resistance in Pseudomonas aeruginosa from cystic fibrosis chronic airways infections.Nonmucoid Pseudomonas aeruginosa expresses alginate in the lungs of patients with cystic fibrosis and in a mouse model.Clinical significance of microbial infection and adaptation in cystic fibrosis.Efficacy and potential for resistance selection of antipseudomonal treatments in a mouse model of lung infection by hypermutable Pseudomonas aeruginosaAnalysis of the interaction interfaces of the N-terminal domain from Pseudomonas aeruginosa MutLClonal dissemination, emergence of mutator lineages and antibiotic resistance evolution in Pseudomonas aeruginosa cystic fibrosis chronic lung infection.UvrD helicase suppresses recombination and DNA damage-induced deletions.Pseudomonas aeruginosa: resistance to the maxCoexistence and within-host evolution of diversified lineages of hypermutable Pseudomonas aeruginosa in long-term cystic fibrosis infections.Genomic and proteomic evidences unravel the UV-resistome of the poly-extremophile Acinetobacter sp. Ver3.Divergent, coexisting Pseudomonas aeruginosa lineages in chronic cystic fibrosis lung infectionsIntact mutS in laboratory-derived and clinical glycopeptide-intermediate Staphylococcus aureus strainsConvergent evolution toward an improved growth rate and a reduced resistance range in Prochlorococcus strains resistant to phage.Influence of high mutation rates on the mechanisms and dynamics of in vitro and in vivo resistance development to single or combined antipseudomonal agents.Mutations in the DNA mismatch repair proteins MutS and MutL of oxazolidinone-resistant or -susceptible Enterococcus faecium.Development of resistance in wild-type and hypermutable Pseudomonas aeruginosa strains exposed to clinical pharmacokinetic profiles of meropenem and ceftazidime simulated in vitro.The cell wall amidase AmiB is essential for Pseudomonas aeruginosa cell division, drug resistance and viability.Resistance mechanisms of multiresistant Pseudomonas aeruginosa strains from Germany and correlation with hypermutationGlobal fluoroquinolone resistance epidemiology and implictions for clinical use.Pseudomonas aeruginosa RsmA plays an important role during murine infection by influencing colonization, virulence, persistence, and pulmonary inflammationRole of hypermutability in the evolution of the genus OenococcusSpontaneously arising mutL mutators in evolving Escherichia coli populations are the result of changes in repeat length.Hypermutable Pseudomonas aeruginosa in Cystic fibrosis patients from two Brazilian cities.Lytic phages obscure the cost of antibiotic resistance in Escherichia coli.The cellular, developmental and population-genetic determinants of mutation-rate evolutionDevelopment of homogeneous expression of resistance in methicillin-resistant Staphylococcus aureus clinical strains is functionally associated with a beta-lactam-mediated SOS response.Hypermutation and the preexistence of antibiotic-resistant Pseudomonas aeruginosa mutants: implications for susceptibility testing and treatment of chronic infections.Darwinism for the Genomic Age: Connecting Mutation to Diversification.
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P2860
The mismatch repair system (mutS, mutL and uvrD genes) in Pseudomonas aeruginosa: molecular characterization of naturally occurring mutants
description
2002 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի մարտին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2002
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im März 2002 veröffentlichter wissenschaftlicher Artikel
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scientific journal article
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vedecký článok (publikovaný 2002/03/01)
@sk
vědecký článek publikovaný v roce 2002
@cs
wetenschappelijk artikel (gepubliceerd op 2002/03/01)
@nl
наукова стаття, опублікована в березні 2002
@uk
مقالة علمية (نشرت في مارس 2002)
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name
The mismatch repair system (mu ...... of naturally occurring mutants
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The mismatch repair system (mu ...... of naturally occurring mutants
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The mismatch repair system (mu ...... of naturally occurring mutants
@nl
type
label
The mismatch repair system (mu ...... of naturally occurring mutants
@ast
The mismatch repair system (mu ...... of naturally occurring mutants
@en
The mismatch repair system (mu ...... of naturally occurring mutants
@nl
prefLabel
The mismatch repair system (mu ...... of naturally occurring mutants
@ast
The mismatch repair system (mu ...... of naturally occurring mutants
@en
The mismatch repair system (mu ...... of naturally occurring mutants
@nl
P2860
P1476
The mismatch repair system (mu ...... of naturally occurring mutants
@en
P2093
Antonio Oliver
Fernando Baquero
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P304
P356
10.1046/J.1365-2958.2002.02855.X
P407
P50
P577
2002-03-01T00:00:00Z