betaCaMKII controls the direction of plasticity at parallel fiber-Purkinje cell synapses
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Actin in dendritic spines: connecting dynamics to functionImpaired locomotor learning and altered cerebellar synaptic plasticity in pep-19/PCP4-null miceLong-term depression and other synaptic plasticity in the cerebellumDysfunctional cerebellar Purkinje cells contribute to autism-like behaviour in Shank2-deficient mice.Pcp4l1 contains an auto-inhibitory element that prevents its IQ motif from binding to calmodulinCaMKIIβ regulates oligodendrocyte maturation and CNS myelinationA β(IV)-spectrin/CaMKII signaling complex is essential for membrane excitability in micePurkinje cell-specific knockout of the protein phosphatase PP2B impairs potentiation and cerebellar motor learning.Distinct kinetics of synaptic structural plasticity, memory formation, and memory decay in massed and spaced learningThe gamma isoform of CaM kinase II controls mouse egg activation by regulating cell cycle resumptionActivation of sodium-dependent glutamate transporters regulates the morphological aspects of oligodendrocyte maturation via signaling through calcium/calmodulin-dependent kinase IIβ's actin-binding/-stabilizing domain.Lgr4 protein deficiency induces ataxia-like phenotype in mice and impairs long term depression at cerebellar parallel fiber-Purkinje cell synapsesPurkinje cell NMDA receptors assume a key role in synaptic gain control in the mature cerebellum.Ataxias and cerebellar dysfunction: involvement of synaptic plasticity deficits?Bidirectional control of BK channel open probability by CAMKII and PKC in medial vestibular nucleus neuronsGeneration and behavior characterization of CaMKIIβ knockout mice.Integrated plasticity at inhibitory and excitatory synapses in the cerebellar circuitGenome-wide association mapping of loci for antipsychotic-induced extrapyramidal symptoms in miceDissociation of locomotor and cerebellar deficits in a murine Angelman syndrome model.CaMKII regulation in information processing and storage.Aberrant Cerebellar Development in Mice Lacking Dual Oxidase Maturation Factors.The molecular, temporal and region-specific requirements of the beta isoform of Calcium/Calmodulin-dependent protein kinase type 2 (CAMK2B) in mouse locomotion.Calcium threshold shift enables frequency-independent control of plasticity by an instructive signalDistinct roles of α- and βCaMKII in controlling long-term potentiation of GABAA-receptor mediated transmission in murine Purkinje cells.Motor Learning Requires Purkinje Cell Synaptic Potentiation through Activation of AMPA-Receptor Subunit GluA3.Inositol 1,4,5-trisphosphate receptor-mediated calcium release in Purkinje cells: from molecular mechanism to behavior.Calcium as a trigger for cerebellar long-term synaptic depression.Distributed synergistic plasticity and cerebellar learning.Regulation of spinogenesis in mature Purkinje cells via mGluR/PKC-mediated phosphorylation of CaMKIIβ.Proteomic identification of specifically carbonylated brain proteins in APP(NLh)/APP(NLh) × PS-1(P264L)/PS-1(P264L) human double mutant knock-in mice model of Alzheimer disease as a function of ageProteomic Analysis of Postsynaptic Protein Complexes Underlying Neuronal Plasticity.Co-induction of LTP and LTD and its regulation by protein kinases and phosphatasesQuantitative proteomics analysis of CaMKII phosphorylation and the CaMKII interactome in the mouse forebrain.The bidirectionality of motor learning in the vestibulo-ocular reflex is a function of cerebellar mGluR1 receptors.Gating of long-term depression by Ca2+/calmodulin-dependent protein kinase II through enhanced cGMP signalling in cerebellar Purkinje cells.De Novo Mutations in Protein Kinase Genes CAMK2A and CAMK2B Cause Intellectual Disability.βCaMKII regulates bidirectional long-term plasticity in cerebellar Purkinje cells by a CaMKII/PP2B switch mechanism.Filamentous actin binding enables βCaMKII to regulate bidirectional plasticity in cerebellar Purkinje cells.Modulation of Murine Olivary Connexin 36 Gap Junctions by PKA and CaMKII.Non-canonical heterogeneous cellular distribution and co-localization of CaMKIIα and CaMKIIβ in the spinal superficial dorsal horn.
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P2860
betaCaMKII controls the direction of plasticity at parallel fiber-Purkinje cell synapses
description
2009 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2009
@ast
im Juli 2009 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2009/07/01)
@sk
vědecký článek publikovaný v roce 2009
@cs
wetenschappelijk artikel (gepubliceerd op 2009/07/01)
@nl
наукова стаття, опублікована в липні 2009
@uk
مقالة علمية (نشرت في يوليو 2009)
@ar
name
betaCaMKII controls the direction of plasticity at parallel fiber-Purkinje cell synapses
@ast
betaCaMKII controls the direction of plasticity at parallel fiber-Purkinje cell synapses
@en
betaCaMKII controls the direction of plasticity at parallel fiber-Purkinje cell synapses
@nl
type
label
betaCaMKII controls the direction of plasticity at parallel fiber-Purkinje cell synapses
@ast
betaCaMKII controls the direction of plasticity at parallel fiber-Purkinje cell synapses
@en
betaCaMKII controls the direction of plasticity at parallel fiber-Purkinje cell synapses
@nl
prefLabel
betaCaMKII controls the direction of plasticity at parallel fiber-Purkinje cell synapses
@ast
betaCaMKII controls the direction of plasticity at parallel fiber-Purkinje cell synapses
@en
betaCaMKII controls the direction of plasticity at parallel fiber-Purkinje cell synapses
@nl
P2093
P2860
P3181
P356
P1433
P1476
betaCaMKII controls the direction of plasticity at parallel fiber-Purkinje cell synapses
@en
P2093
Casper C. Hoogenraad
Chris I. De Zeeuw
Christian Hansel
Freek E. Hoebeek
Geeske M. van Woerden
Raghavendra Y. Nagaraja
Steven A. Kushner
Ype Elgersma
Zhenyu Gao
P2860
P2888
P304
P3181
P356
10.1038/NN.2329
P407
P577
2009-07-01T00:00:00Z
P5875
P6179
1002651257