Ihh signaling is directly required for the osteoblast lineage in the endochondral skeleton
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TGF-β and BMP signaling in osteoblast, skeletal development, and bone formation, homeostasis and diseaseThe Role of Hedgehog Signaling in Tumor Induced Bone DiseaseTGF-β and BMP signaling in osteoblast differentiation and bone formationCurrent insights on the regenerative potential of the periosteum: molecular, cellular, and endogenous engineering approachesMedication-Related Osteonecrosis of the Jaw: New Insights into Molecular Mechanisms and Cellular Therapeutic ApproachesRegulation of Long Bone Growth in Vertebrates; It Is Time to Catch UpOvergrowth of a mouse model of Simpson-Golabi-Behmel syndrome is partly mediated by Indian hedgehogInvolvement of endogenous bone morphogenetic protein (BMP) 2 and BMP6 in bone formationGli3Xt-J/Xt-J mice exhibit lambdoid suture craniosynostosis which results from altered osteoprogenitor proliferation and differentiationGli1 protein participates in Hedgehog-mediated specification of osteoblast lineage during endochondral ossificationDisruption of Scube2 Impairs Endochondral Bone FormationMolecular development of fibular reduction in birds and its evolution from dinosaursBone regeneration and stem cellsHigh frequency of cephalic neural crest cells shows coexistence of neurogenic, melanogenic, and osteogenic differentiation capacitiesAdenomatous polyposis coli-mediated control of beta-catenin is essential for both chondrogenic and osteogenic differentiation of skeletal precursorsIdentification of SOX9 interaction sites in the genome of chondrocytesdelta-EF1 is a negative regulator of Ihh in the developing growth plate.Partial rescue of postnatal growth plate abnormalities in Ihh mutants by expression of a constitutively active PTH/PTHrP receptorUp-regulation of glycolytic metabolism is required for HIF1α-driven bone formationTargeted deletion of Capn4 in cells of the chondrocyte lineage impairs chondrocyte proliferation and differentiationAdipose-derived stem cells: a review of signaling networks governing cell fate and regenerative potential in the context of craniofacial and long bone skeletal repair.A mouse model of chondrocyte-specific somatic mutation reveals a role for Ext1 loss of heterozygosity in multiple hereditary exostoses.Mutations in fam20b and xylt1 reveal that cartilage matrix controls timing of endochondral ossification by inhibiting chondrocyte maturationChondrocyte-specific knockout of Cbfβ reveals the indispensable function of Cbfβ in chondrocyte maturation, growth plate development and trabecular bone formation in mice.Selective Runx2-II deficiency leads to low-turnover osteopenia in adult mice.Gli1 haploinsufficiency leads to decreased bone mass with an uncoupling of bone metabolism in adult mice.mTORC1 signaling controls mammalian skeletal growth through stimulation of protein synthesis.Activation of the Hh pathway in periosteum-derived mesenchymal stem cells induces bone formation in vivo: implication for postnatal bone repair.The Role of RUNX2 in Osteosarcoma Oncogenesis.A new role for Hedgehogs in juxtacrine signaling.MicroRNAs involved in bone formation.Constitutive activation of Gli2 impairs bone formation in postnatal growing miceIhh and Runx2/Runx3 signaling interact to coordinate early chondrogenesis: a mouse model.Role of Indian hedgehog signaling in palatal osteogenesis.Enthesis fibrocartilage cells originate from a population of Hedgehog-responsive cells modulated by the loading environment.Stk11 (Lkb1) deletion in the osteoblast lineage leads to high bone turnover, increased trabecular bone density and cortical porosity.Site-1 protease is essential to growth plate maintenance and is a critical regulator of chondrocyte hypertrophic differentiation in postnatal miceHedgehog signaling activates a positive feedback mechanism involving insulin-like growth factors to induce osteoblast differentiation.Lrp5 and Lrp6 redundantly control skeletal development in the mouse embryoIndian Hedgehog signaling pathway members are associated with magnetic resonance imaging manifestations and pathological scores in lumbar facet joint osteoarthritis
P2860
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P2860
Ihh signaling is directly required for the osteoblast lineage in the endochondral skeleton
description
2004 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի մարտին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2004
@ast
im März 2004 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2004/03/01)
@sk
vědecký článek publikovaný v roce 2004
@cs
wetenschappelijk artikel (gepubliceerd op 2004/03/01)
@nl
наукова стаття, опублікована в березні 2004
@uk
مقالة علمية (نشرت في مارس 2004)
@ar
name
Ihh signaling is directly requ ...... e in the endochondral skeleton
@ast
Ihh signaling is directly requ ...... e in the endochondral skeleton
@en
Ihh signaling is directly requ ...... e in the endochondral skeleton
@nl
type
label
Ihh signaling is directly requ ...... e in the endochondral skeleton
@ast
Ihh signaling is directly requ ...... e in the endochondral skeleton
@en
Ihh signaling is directly requ ...... e in the endochondral skeleton
@nl
prefLabel
Ihh signaling is directly requ ...... e in the endochondral skeleton
@ast
Ihh signaling is directly requ ...... e in the endochondral skeleton
@en
Ihh signaling is directly requ ...... e in the endochondral skeleton
@nl
P2093
P921
P3181
P356
P1433
P1476
Ihh signaling is directly requ ...... e in the endochondral skeleton
@en
P2093
Andrew P. McMahon
Fanxin Long
Henry M. Kronenberg
Jill McMahon
Shinsuke Ohba
Ung-il Chung
P304
P3181
P356
10.1242/DEV.01006
P407
P577
2004-03-01T00:00:00Z