MicroRNA-17-92, a direct Ap-2α transcriptional target, modulates T-box factor activity in orofacial clefting
about
Molecular basis of cleft palates in micePitx2-microRNA pathway that delimits sinoatrial node development and inhibits predisposition to atrial fibrillationAn E2F1/MiR-17-92 Negative Feedback Loop mediates proliferation of Mouse Palatal Mesenchymal CellsTfap2a-dependent changes in mouse facial morphology result in clefting that can be ameliorated by a reduction in Fgf8 gene dosage.MicroRNA expression profiling of the developing murine upper lip.TBX1 protein interactions and microRNA-96-5p regulation controls cell proliferation during craniofacial and dental development: implications for 22q11.2 deletion syndrome.Methylated microRNA genes of the developing murine palateMolecular mechanisms of midfacial developmental defects.Yap and Taz play a crucial role in neural crest-derived craniofacial development.A new plasmid-based microRNA inhibitor system that inhibits microRNA families in transgenic mice and cells: a potential new therapeutic reagent.MicroRNA Profiling during Craniofacial Development: Potential Roles for Mir23b and Mir133b.Associations between microRNA binding site SNPs in FGFs and FGFRs and the risk of non-syndromic orofacial cleftAssessment of differentially expressed plasma microRNAs in nonsyndromic cleft palate and nonsyndromic cleft lip with cleft palate.MicroRNAs in Palatogenesis and Cleft Palate.Macro advances in microRNAs and myocardial regeneration.The Role of Noncoding Genetic Variation in Isolated Orofacial Clefts.Inhibition of the miR-17-92 Cluster Separates Stages of Palatogenesis.Differential microRNA expression in cultured palatal fibroblasts from infants with cleft palate and controls.Systems biology of facial development: contributions of ectoderm and mesenchyme.AP2α controls the dynamic balance between miR-126&126* and miR-221&222 during melanoma progression.Epigenetic mechanism causes Wnt9b deficiency and nonsyndromic cleft lip and palate in the A/WySn mouse strain.Genes and microRNAs associated with mouse cleft palate: A systematic review and bioinformatics analysis.DiGeorge-like syndrome in a child with a 3p12.3 deletion involving MIR4273 gene born to a mother with gestational diabetes mellitus.Sertoli cell-specific ablation of miR-17-92 cluster significantly alters whole testis transcriptome without apparent phenotypic effects.Integrated assessment of differentially expressed plasma microRNAs in subtypes of nonsyndromic orofacial clefts.
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P2860
MicroRNA-17-92, a direct Ap-2α transcriptional target, modulates T-box factor activity in orofacial clefting
description
2013 nî lūn-bûn
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2013 թուականին հրատարակուած գիտական յօդուած
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2013 թվականին հրատարակված գիտական հոդված
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2013年の論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年论文
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name
MicroRNA-17-92, a direct Ap-2α ...... activity in orofacial clefting
@ast
MicroRNA-17-92, a direct Ap-2α ...... activity in orofacial clefting
@en
MicroRNA-17-92, a direct Ap-2α ...... activity in orofacial clefting
@nl
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label
MicroRNA-17-92, a direct Ap-2α ...... activity in orofacial clefting
@ast
MicroRNA-17-92, a direct Ap-2α ...... activity in orofacial clefting
@en
MicroRNA-17-92, a direct Ap-2α ...... activity in orofacial clefting
@nl
prefLabel
MicroRNA-17-92, a direct Ap-2α ...... activity in orofacial clefting
@ast
MicroRNA-17-92, a direct Ap-2α ...... activity in orofacial clefting
@en
MicroRNA-17-92, a direct Ap-2α ...... activity in orofacial clefting
@nl
P2093
P2860
P3181
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MicroRNA-17-92, a direct Ap-2α ...... activity in orofacial clefting
@en
P2093
Elzbieta Klysik
James F Martin
Robert J Schwartz
Stephanie B Greene
Trevor J Williams
P2860
P304
P3181
P356
10.1371/JOURNAL.PGEN.1003785
P407
P577
2013-01-01T00:00:00Z