Response of breast cancer cells and cancer stem cells to metformin and hyperthermia alone or combined - Test2 - elhamkhani - TriplyDB

Response of breast cancer cells and cancer stem cells to metformin and hyperthermia alone or combined

Response of breast cancer cells and cancer stem cells to metformin and hyperthermia alone or combined

http://www.wikidata.org/entity/Q28539523

about

PI3K/Akt/mTOR signaling pathway in cancer stem cells: from basic research to clinical application The association between type 2 diabetes mellitus and women cancer: the epidemiological evidences and putative mechanisms Chemotherapy targeting cancer stem cells Inhibiting stemness and invasive properties of glioblastoma tumorsphere by combined treatment with temozolomide and a newly designed biguanide (HL156A).Curcumin restores sensitivity to retinoic acid in triple negative breast cancer cells Metformin enhances tamoxifen-mediated tumor growth inhibition in ER-positive breast carcinoma.Increase in apoptosis by combination of metformin with silibinin in human colorectal cancer cells Metformin ameliorates ionizing irradiation-induced long-term hematopoietic stem cell injury in mice.High mitochondrial mass identifies a sub-population of stem-like cancer cells that are chemo-resistant.Bulk pancreatic cancer cells can convert into cancer stem cells(CSCs) in vitro and 2 compounds can target these CSCs Hyperthermia enhances 17-DMAG efficacy in hepatocellular carcinoma cells with aggravated DNA damage and impaired G2/M transition In vitro comparison of conventional hyperthermia and modulated electro-hyperthermia.The future of biology in driving the field of hyperthermia.Mechanism of metformin action in MCF-7 and MDA-MB-231 human breast cancer cells involves oxidative stress generation, DNA damage, and transforming growth factor β1 induction.Human mesenchymal stem cells as a novel platform for simultaneous evaluation of cytotoxicity and genotoxicity of pharmaceuticals.Recent advances in the use of metformin: can treating diabetes prevent breast cancer?Metformin Inhibits Tumorigenesis and Tumor Growth of Breast Cancer Cells by Upregulating miR-200c but Downregulating AKT2 Expression.Enhanced Response of Metformin towards the Cancer Cells due to Synergism with Multi-walled Carbon Nanotubes in Photothermal Therapy.Electro-hyperthermia inhibits glioma tumorigenicity through the induction of E2F1-mediated apoptosis.Glyoxalase 1 sustains the metastatic phenotype of prostate cancer cells via EMT control.Microwave hyperthermia promotes caspase‑3-dependent apoptosis and induces G2/M checkpoint arrest via the ATM pathway in non‑small cell lung cancer cells.

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Response of breast cancer cells and cancer stem cells to metformin and hyperthermia alone or combined

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2014 nî lūn-bûn

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Bo-Hwa Choi

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Brent Williams

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Chang W Song

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Chang-Shin Park

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Chung K Lee

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Eun-Taex Oh

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Heon Joo Park

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Hyemi Lee

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P2860

The blooming of the French lilac

Metformin in Cancer Therapy: A New Perspective for an Old Antidiabetic Drug?

Prospective identification of tumorigenic breast cancer cells

Glioma stem cells promote radioresistance by preferential activation of the DNA damage response

TOR, a central controller of cell growth.

Metformin induces unique biological and molecular responses in triple negative breast cancer cells

Exploring the role of cancer stem cells in radioresistance

Metformin and reduced risk of cancer in diabetic patients

The response of CD24(-/low)/CD44+ breast cancer-initiating cells to radiation

Metformin: a therapeutic opportunity in breast cancer.

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