Vimar Is a Novel Regulator of Mitochondrial Fission through Miro - Test2 - elhamkhani - TriplyDB

Vimar Is a Novel Regulator of Mitochondrial Fission through Miro

Vimar Is a Novel Regulator of Mitochondrial Fission through Miro

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Understanding Miro GTPases: Implications in the Treatment of Neurodegenerative Disorders.

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Vimar Is a Novel Regulator of Mitochondrial Fission through Miro

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2016 nî lūn-bûn

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2016 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2016 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

@zh-hk

2016年論文

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2016年論文

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2016年论文

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name

Vimar Is a Novel Regulator of Mitochondrial Fission through Miro

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Vimar Is a Novel Regulator of Mitochondrial Fission through Miro

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Vimar Is a Novel Regulator of Mitochondrial Fission through Miro

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Vimar Is a Novel Regulator of Mitochondrial Fission through Miro

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Vimar Is a Novel Regulator of Mitochondrial Fission through Miro

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Vimar Is a Novel Regulator of Mitochondrial Fission through Miro

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prefLabel

Vimar Is a Novel Regulator of Mitochondrial Fission through Miro

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Vimar Is a Novel Regulator of Mitochondrial Fission through Miro

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Vimar Is a Novel Regulator of Mitochondrial Fission through Miro

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JOURNAL.PGEN.1006359

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Vimar Is a Novel Regulator of Mitochondrial Fission through Miro

@en

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Lianggong Ding

http://www.w3.org/2001/XMLSchema#string

Xunming Ji

http://www.w3.org/2001/XMLSchema#string

Yanping Han

http://www.w3.org/2001/XMLSchema#string

Yuhong Li

http://www.w3.org/2001/XMLSchema#string

P2860

PINK1 is selectively stabilized on impaired mitochondria to activate Parkin

Parkinson's disease-associated kinase PINK1 regulates Miro protein level and axonal transport of mitochondria

OPA1, encoding a dynamin-related GTPase, is mutated in autosomal dominant optic atrophy linked to chromosome 3q28

The mitochondrial phosphatase PGAM5 functions at the convergence point of multiple necrotic death pathways

Molecular cloning of the human cDNA for a stimulatory GDP/GTP exchange protein for c-Ki-ras p21 and smg p21

The mechanism of Ca2+ -dependent regulation of kinesin-mediated mitochondrial motility

Mff is an essential factor for mitochondrial recruitment of Drp1 during mitochondrial fission in mammalian cells

The atypical Rho GTPases Miro-1 and Miro-2 have essential roles in mitochondrial trafficking

Atypical Rho GTPases have roles in mitochondrial homeostasis and apoptosis

OPA1 requires mitofusin 1 to promote mitochondrial fusion

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scholarly article

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3092858

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10.1371/JOURNAL.PGEN.1006359

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2016-10-07T00:00:00Z

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27716788

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5065127

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