Thrombospondin-4 contributes to spinal sensitization and neuropathic pain states
about
Astrocytes Control Synapse Formation, Function, and EliminationCortical astrocytes rewire somatosensory cortical circuits for peripheral neuropathic painBuilding an RNA Sequencing Transcriptome of the Central Nervous SystemCurrent Status of the New Antiepileptic Drugs in Chronic PainCentral Mechanisms Mediating Thrombospondin-4-induced Pain States.Thrombospondins 1 and 2 are important for afferent synapse formation and function in the inner ear.Matricellular proteins in the trabecular meshwork: review and update.Pain regulation by non-neuronal cells and inflammation.Plasticity response in the contralesional hemisphere after subtle neurotrauma: gene expression profiling after partial deafferentation of the hippocampus.Painful nerve injury upregulates thrombospondin-4 expression in dorsal root gangliaThrombospondin-4 and excitatory synaptogenesis promote spinal sensitization after painful mechanical joint injury.Revisiting the matricellular concept.Human Umbilical Tissue-Derived Cells Promote Synapse Formation and Neurite Outgrowth via Thrombospondin Family ProteinsAstrocyte-derived thrombospondins mediate the development of hippocampal presynaptic plasticity in vitroSynaptic ultrastructure changes in trigeminocervical complex posttrigeminal nerve injury.Adolescent Intermittent Alcohol Exposure: Dysregulation of Thrombospondins and Synapse Formation are Associated with Decreased Neuronal Density in the Adult Hippocampus.Comparative proteomic analysis of normal and collagen IX null mouse cartilage reveals altered extracellular matrix composition and novel components of the collagen IX interactome.Thrombospondin-4 contributes to spinal cord injury-induced changes in nociception.Glia and pain: is chronic pain a gliopathy?Calcium channel α2δ1 proteins mediate trigeminal neuropathic pain states associated with aberrant excitatory synaptogenesis.Trigeminal nerve injury-induced thrombospondin-4 up-regulation contributes to orofacial neuropathic pain states in a rat model.Proregenerative properties of ECM molecules.Calcium-permeable ion channels in pain signaling.Astrocyte-secreted matricellular proteins in CNS remodelling during development and diseaseInjury-induced maladaptation and dysregulation of calcium channel α2 δ subunit proteins and its contribution to neuropathic pain development.Current and Future Issues in the Development of Spinal Agents for the Management of Pain.The Anti-Allodynic Gabapentinoids: Myths, Paradoxes, and Acute Effects.Painful Cervical Facet Joint Injury Is Accompanied by Changes in the Number of Excitatory and Inhibitory Synapses in the Superficial Dorsal Horn That Differentially Relate to Local Tissue Injury Severity.Astrocyte-mediated synapse remodeling in the pathological brain.Molecular mechanisms of astrocyte-induced synaptogenesis.Microarray analysis of bone marrow lesions in osteoarthritis demonstrates upregulation of genes implicated in osteochondral turnover, neurogenesis and inflammation.Thrombospondin-4 reduces binding affinity of [(3)H]-gabapentin to calcium-channel α2δ-1-subunit but does not interact with α2δ-1 on the cell-surface when co-expressed.Bortezomib-induced peripheral neuropathy: A genome-wide association study on multiple myeloma patients.Thrombospondin-4 divergently regulates voltage-gated Ca2+ channel subtypes in sensory neurons after nerve injuryAcute and chronic mu opioids differentially regulate thrombospondins 1 and 2 isoforms in astrocytes.Spinal Astrocytic Thrombospondin-4 Induced by Excitatory Neuronal Signaling Mediates Pain After Facet Capsule Injury.Increased thrombospondin-4 after nerve injury mediates disruption of intracellular calcium signaling in primary sensory neurons.Thrombospondin-4 in tissue remodeling.Short general anaesthesia induces prolonged changes in gene expression in the mouse hippocampus.Gabapentin Prevents Progressive Increases in Excitatory Connectivity and Epileptogenesis Following Neocortical Trauma.
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P2860
Thrombospondin-4 contributes to spinal sensitization and neuropathic pain states
description
2012 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի հունիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2012
@ast
im Juni 2012 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2012/06/27)
@sk
vědecký článek publikovaný v roce 2012
@cs
wetenschappelijk artikel (gepubliceerd op 2012/06/27)
@nl
наукова стаття, опублікована в червні 2012
@uk
مقالة علمية (نشرت في 27-6-2012)
@ar
name
Thrombospondin-4 contributes to spinal sensitization and neuropathic pain states
@ast
Thrombospondin-4 contributes to spinal sensitization and neuropathic pain states
@en
Thrombospondin-4 contributes to spinal sensitization and neuropathic pain states
@nl
type
label
Thrombospondin-4 contributes to spinal sensitization and neuropathic pain states
@ast
Thrombospondin-4 contributes to spinal sensitization and neuropathic pain states
@en
Thrombospondin-4 contributes to spinal sensitization and neuropathic pain states
@nl
prefLabel
Thrombospondin-4 contributes to spinal sensitization and neuropathic pain states
@ast
Thrombospondin-4 contributes to spinal sensitization and neuropathic pain states
@en
Thrombospondin-4 contributes to spinal sensitization and neuropathic pain states
@nl
P2093
P2860
P50
P921
P3181
P1476
Thrombospondin-4 contributes to spinal sensitization and neuropathic pain states
@en
P2093
Amin Boroujerdi
Ben Barres
Chun-Yi Zhou
Doo-Sik Kim
Frank Zaucke
Joshua Lee
Kang-Wu Li
Kelli Sharp
Michael Corpe
P2860
P304
P3181
P356
10.1523/JNEUROSCI.6494-11.2012
P407
P577
2012-06-01T00:00:00Z