Indoleamine 2,3-dioxygenase expression promotes renal ischemia-reperfusion injury
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Tryptophan Metabolism in Patients With Chronic Kidney Disease Secondary to Type 2 Diabetes: Relationship to Inflammatory Markers.A powerful yeast-based screening assay for the identification of inhibitors of indoleamine 2,3-dioxygenase.Increased indoleamine 2,3-dioxygenase (IDO) activity and elevated serum levels of tryptophan catabolites in patients with chronic kidney disease: a possible link between chronic inflammation and uraemic symptoms.Inhibitors of indoleamine 2,3-dioxygenase: a review of novel patented lead compounds.Necrostatin-1 attenuates ischemia injury induced cell death in rat tubular cell line NRK-52E through decreased Drp1 expression.The Mechanisms of Human Renal Epithelial Cell Modulation of Autologous Dendritic Cell Phenotype and Function.IKKα is involved in kidney recovery and regeneration of acute ischemia/reperfusion injury in mice through IL10-producing regulatory T cells.The potential role of indoleamine 2,3 dioxygenase (IDO) as a predictive and therapeutic target for diabetes treatment: a mythical truth.A combined epidemiologic and metabolomic approach improves CKD prediction.Targeting the immunoregulatory indoleamine 2,3 dioxygenase pathway in immunotherapy.High activity of indoleamine 2,3-dioxygenase is associated with renal insufficiency in Puumala hantavirus induced nephropathia epidemica.Host indoleamine 2,3-dioxygenase: contribution to systemic acquired tumor tolerance.An overview of renal metabolomics.Necroptosis: A Novel Cell Death Modality and Its Potential Relevance for Critical Care Medicine.Indoleamine 2, 3-dioxygenase (IDO) increases during renal fibrogenesis and its inhibition potentiates TGF-β 1-induced epithelial to mesenchymal transition.Metabolite markers of incident CKD riskGene therapy with adenovirus-delivered indoleamine 2,3-dioxygenase improves renal function and morphology following allogeneic kidney transplantation in rat.Inhibition of indoleamine 2, 3-dioxygenase-mediated tryptophan catabolism accelerates crescentic glomerulonephritis.Human proximal tubule epithelial cells modulate autologous B-cell function.RIPK3-mediated necroptosis promotes donor kidney inflammatory injury and reduces allograft survival.
P2860
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P2860
Indoleamine 2,3-dioxygenase expression promotes renal ischemia-reperfusion injury
description
2008 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2008
@ast
im Juli 2008 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2008/07/01)
@sk
vědecký článek publikovaný v roce 2008
@cs
wetenschappelijk artikel (gepubliceerd op 2008/07/01)
@nl
наукова стаття, опублікована в липні 2008
@uk
مقالة علمية (نشرت في يوليو 2008)
@ar
name
Indoleamine 2,3-dioxygenase expression promotes renal ischemia-reperfusion injury
@ast
Indoleamine 2,3-dioxygenase expression promotes renal ischemia-reperfusion injury
@en
Indoleamine 2,3-dioxygenase expression promotes renal ischemia-reperfusion injury
@nl
type
label
Indoleamine 2,3-dioxygenase expression promotes renal ischemia-reperfusion injury
@ast
Indoleamine 2,3-dioxygenase expression promotes renal ischemia-reperfusion injury
@en
Indoleamine 2,3-dioxygenase expression promotes renal ischemia-reperfusion injury
@nl
prefLabel
Indoleamine 2,3-dioxygenase expression promotes renal ischemia-reperfusion injury
@ast
Indoleamine 2,3-dioxygenase expression promotes renal ischemia-reperfusion injury
@en
Indoleamine 2,3-dioxygenase expression promotes renal ischemia-reperfusion injury
@nl
P2093
P2860
P1476
Indoleamine 2,3-dioxygenase expression promotes renal ischemia-reperfusion injury
@en
P2093
Andrew L Mellor
Anthony M Jevnikar
Hongtao Sun
Qiunong Guan
Shuang Wang
P2860
P304
P356
10.1152/AJPRENAL.00567.2007
P577
2008-05-14T00:00:00Z