CCR2 expression determines T1 versus T2 polarization during pulmonary Cryptococcus neoformans infection
about
Aspergillus antigen induces robust Th2 cytokine production, inflammation, airway hyperreactivity and fibrosis in the absence of MCP-1 or CCR2Inflammatory monocytes and the pathogenesis of viral encephalitisMolecular control of monocyte developmentAdaptive immunity to fungiImmunoregulation in Fungal DiseasesType I interferons promote fatal immunopathology by regulating inflammatory monocytes and neutrophils during Candida infectionsMonocyte recruitment during infection and inflammationSignificant involvement of CCL2 (MCP-1) in inflammatory disorders of the lung.An association between pulmonary Mycobacterium avium-intracellulare complex infections and biomarkers of Th2-type inflammation.Accumulation of CD11b+ lung dendritic cells in response to fungal infection results from the CCR2-mediated recruitment and differentiation of Ly-6Chigh monocytes.The gamma interferon receptor is required for the protective pulmonary inflammatory response to Cryptococcus neoformans.Antigen-presenting dendritic cells rescue CD4-depleted CCR2-/- mice from lethal Histoplasma capsulatum infection.Critical protective role for MCP-1 in pneumonic Burkholderia mallei infection.Paracoccidioides brasilinsis-induced migration of dendritic cells and subsequent T-cell activation in the lung-draining lymph nodes.Regulation of inflammatory monocyte/macrophage recruitment from the bone marrow during murine cytomegalovirus infection: role for type I interferons in localized induction of CCR2 ligandsTLR9 signaling is required for generation of the adaptive immune protection in Cryptococcus neoformans-infected lungs.Monocyte trafficking to hepatic sites of bacterial infection is chemokine independent and directed by focal intercellular adhesion molecule-1 expressionMonocyte-mediated defense against microbial pathogens.Ontogeny of myeloid cells.Mycobacterium tuberculosis in chemokine receptor 2-deficient mice: influence of dose on disease progressionImmune modulation mediated by cryptococcal laccase promotes pulmonary growth and brain dissemination of virulent Cryptococcus neoformans in mice.Inflammatory monocytes promote progression of Duchenne muscular dystrophy and can be therapeutically targeted via CCR2.IL-23 induces atopic dermatitis-like inflammation instead of psoriasis-like inflammation in CCR2-deficient miceChemokine control of West Nile virus infection.Chemokine receptor 2-mediated accumulation of fungicidal exudate macrophages in mice that clear cryptococcal lung infection.Successful protection against tularemia in C57BL/6 mice is correlated with expansion of Francisella tularensis-specific effector T cells.Chemokine receptor CCR2 is not essential for the development of experimental cerebral malaria.Cryptococcal infection and Th1-Th2 cytokine balance.Type II cytokines impair host defense against an intracellular fungal pathogen by amplifying macrophage generation of IL-33.Impaired lung dendritic cell activation in CCR2 knockout mice.Urease expression by Cryptococcus neoformans promotes microvascular sequestration, thereby enhancing central nervous system invasion.Adaptive immunity to fungi.CCR2 expressing CD4+ T lymphocytes are preferentially recruited to the ileum in Crohn's disease.Mononuclear phagocyte-mediated antifungal immunity: the role of chemotactic receptors and ligands.Cryptococcal heat shock protein 70 homolog Ssa1 contributes to pulmonary expansion of Cryptococcus neoformans during the afferent phase of the immune response by promoting macrophage M2 polarizationType I IFN Induction via Poly-ICLC Protects Mice against Cryptococcosis.The Granuloma Response Controlling Cryptococcosis in Mice Depends on the Sphingosine Kinase 1-Sphingosine 1-Phosphate Pathway.Early induction of CCL7 downstream of TLR9 signaling promotes the development of robust immunity to cryptococcal infection.IL-23 dampens the allergic response to Cryptococcus neoformans through IL-17-independent and -dependent mechanisms.A macrophage subpopulation recruited by CC chemokine ligand-2 clears apoptotic cells in noninfectious lung injury
P2860
Q24796466-C48B8098-4670-4C01-88E7-D91170966A8EQ26861256-D271379E-9C51-49D0-968B-92AFC957DCB9Q27011850-58D216B1-AA41-418D-A43F-722A72F45B92Q27025806-44F92985-B937-466C-80F8-B9ACADAB9E5BQ28074776-624B6923-B75E-4E41-B352-0BAFDBA2F6B9Q28481589-C1A00282-64F5-461C-9CA3-542D241847BAQ29620067-54068C73-3AA0-4DBD-9BFD-A7FA95B03CF8Q30311027-D7265B3D-3BE2-41B8-AEEE-B2CE1F461588Q33691367-90014CA2-0102-42F4-8F3B-10CFF7E276C7Q33705456-739654A0-EE92-4A66-9F83-70C8BD287345Q33716138-5BDB2A3C-F28B-4014-9502-D51DF5C8F459Q33826072-35926B0E-3488-4AB9-9D69-5AEB08DE2BC0Q33886336-54CAFBC2-22C7-46E1-967D-D8DEB030C3F9Q33911255-6B54DDA5-91B6-4743-9F1C-D2288E856802Q34024156-49CF961C-43AA-4039-A657-C5C0BD4816E6Q34033551-A4BF288E-5D01-4A42-BBDD-71465E3AEF23Q34065838-30EC0090-1DD3-4E31-985A-B7F494190912Q34065898-4F2FBB66-61BD-4A36-BA09-0DE2F3DD3280Q34127088-4A370C7B-A2A7-46BD-994A-DA3626AAE209Q34182051-8B70B62D-7805-4982-9E9D-080E3CEB50D9Q34460637-12DE3858-1EA1-4294-B597-07830C152367Q34542698-D6569BE0-BA7D-4070-BA4A-592D1CF38EBCQ34614791-014105A8-E4B0-4EB4-A1C4-0B53F6EA5A94Q34632168-17CC1572-B8E0-4515-AFEC-9ACC65B58F93Q34756043-FF02F7B7-750B-4500-9AB2-DE77D8792D1DQ34784967-0BCAB042-8AE6-4DDE-9740-1225F8C7C5D3Q35011260-B16B2548-B64C-4EEE-A0ED-814CDB65C3E1Q35027538-F75C202C-4A36-42A4-8AE9-58167BC470BBQ35079476-ED73F3D3-66EF-46FE-AC4A-F55E2251E47AQ35080114-CF84129E-1D85-43EE-AC57-492CB93DFB36Q35098130-90F0AB3F-6C66-4FAD-9E94-A90E97F13AA3Q35164257-5C79309F-0AE8-4D93-95A7-1E8CAEC477D0Q35596811-26C502F6-142A-4679-A431-570B9B2F713CQ35602489-AA0B2FF0-8D89-4851-8594-0020232C20BDQ35691068-DF57F4FF-25F4-4F27-9DE7-BCD0BE8621D9Q35739585-D33437E8-7F5C-40CD-9F13-C9F39B1A776FQ35745536-3D53365E-AA31-489F-9B7B-DC58599718C9Q35884062-FD4E390F-ED66-414B-B495-5A2AA94D75ABQ35952109-CC8B4589-01C1-4A8E-8FF5-E10DFA0D8A35Q35994491-11C8C0D4-6715-4BB1-9D1B-CDD94D8EE233
P2860
CCR2 expression determines T1 versus T2 polarization during pulmonary Cryptococcus neoformans infection
description
2000 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2000
@ast
im Februar 2000 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2000/02/15)
@sk
vědecký článek publikovaný v roce 2000
@cs
wetenschappelijk artikel (gepubliceerd op 2000/02/15)
@nl
наукова стаття, опублікована в лютому 2000
@uk
مقالة علمية (نشرت في 15-2-2000)
@ar
name
CCR2 expression determines T1 ...... ptococcus neoformans infection
@ast
CCR2 expression determines T1 ...... ptococcus neoformans infection
@en
CCR2 expression determines T1 ...... ptococcus neoformans infection
@nl
type
label
CCR2 expression determines T1 ...... ptococcus neoformans infection
@ast
CCR2 expression determines T1 ...... ptococcus neoformans infection
@en
CCR2 expression determines T1 ...... ptococcus neoformans infection
@nl
prefLabel
CCR2 expression determines T1 ...... ptococcus neoformans infection
@ast
CCR2 expression determines T1 ...... ptococcus neoformans infection
@en
CCR2 expression determines T1 ...... ptococcus neoformans infection
@nl
P2093
P3181
P1476
CCR2 expression determines T1 ...... ptococcus neoformans infection
@en
P2093
G. B. Huffnagle
G. B. Toews
T. R. Traynor
W. A. Kuziel
P304
P3181
P356
10.4049/JIMMUNOL.164.4.2021
P407
P577
2000-02-15T00:00:00Z