Clustered environments and randomized genes: a fundamental distinction between conventional and genetic epidemiology
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Fetal alcohol exposure and IQ at age 8: evidence from a population-based birth-cohort studyFADS2 polymorphisms modify the effect of breastfeeding on child IQMendelian randomisation and causal inference in observational epidemiologyIs replication the gold standard for validating genome-wide association findings?Placing epidemiological results in the context of multiplicity and typical correlations of exposuresMR-PheWAS: hypothesis prioritization among potential causal effects of body mass index on many outcomes, using Mendelian randomizationStrengthening the reporting of genetic risk prediction studies (GRIPS): explanation and elaboration.Assessment of vibration of effects due to model specification can demonstrate the instability of observational associationsStrengthening the reporting of genetic risk prediction studies (GRIPS): explanation and elaborationSystematic identification of interaction effects between genome- and environment-wide associations in type 2 diabetes mellitusSex hormone-binding globulin and risk of type 2 diabetes in women and menIntegrated associations of genotypes with multiple blood biomarkers linked to coronary heart disease riskEpigenome-wide Association Studies and the Interpretation of Disease -OmicsAnalytical Complexity in Detection of Gene Variant-by-Environment Exposure Interactions in High-Throughput Genomic and Exposomic ResearchThe exposome: from concept to utilityMendelian randomization: genetic anchors for causal inference in epidemiological studiesUse of the "exposome" in the practice of epidemiology: a primer on -omic technologiesMendelian Randomization and Type 2 DiabetesA non-synonymous variant in ADH1B is strongly associated with prenatal alcohol use in a European sample of pregnant womenAssessing causality in the association between child adiposity and physical activity levels: a Mendelian randomization analysisEffects of BMI, fat mass, and lean mass on asthma in childhood: a Mendelian randomization studyCommon variants associated with plasma triglycerides and risk for coronary artery diseaseThe role of adiposity in cardiometabolic traits: a Mendelian randomization analysisPrenatal alcohol exposure and childhood balance ability: findings from a UK birth cohort studyThe role of longitudinal cohort studies in epigenetic epidemiology: challenges and opportunitiesMendelian randomization in cardiometabolic disease: challenges in evaluating causality.Body mass index: Has epidemiology started to break down causal contributions to health and disease?Apolipoprotein E genotype, cardiovascular biomarkers and risk of stroke: systematic review and meta-analysis of 14,015 stroke cases and pooled analysis of primary biomarker data from up to 60,883 individuals.Best (but oft-forgotten) practices: the design, analysis, and interpretation of Mendelian randomization studies.Association of lactase persistence genotype with milk consumption, obesity and blood pressure: a Mendelian randomization study in the 1982 Pelotas (Brazil) Birth Cohort, with a systematic review and meta-analysisMendelian randomization analysis of a time-varying exposure for binary disease outcomes using functional data analysis methods.Exploring the developmental overnutrition hypothesis using parental-offspring associations and FTO as an instrumental variableDetermining origins and causes of childhood obesity via Mendelian randomization analysis.Does high C-reactive protein concentration increase atherosclerosis? The Whitehall II Study.The association of C-reactive protein and CRP genotype with coronary heart disease: findings from five studies with 4,610 cases amongst 18,637 participantsThe association between BMI and mortality using offspring BMI as an indicator of own BMI: large intergenerational mortality studyUnraveling the directional link between adiposity and inflammation: a bidirectional Mendelian randomization approach.Vitamin C and InfectionsEpigenetic epidemiology of common complex disease: prospects for prediction, prevention, and treatment.Association between common variation at the FTO locus and changes in body mass index from infancy to late childhood: the complex nature of genetic association through growth and development.
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P2860
Clustered environments and randomized genes: a fundamental distinction between conventional and genetic epidemiology
description
2007 nî lūn-bûn
@nan
2007 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
name
Clustered environments and ran ...... ional and genetic epidemiology
@ast
Clustered environments and ran ...... ional and genetic epidemiology
@en
Clustered environments and ran ...... ional and genetic epidemiology
@en-gb
Clustered environments and ran ...... ional and genetic epidemiology
@nl
type
label
Clustered environments and ran ...... ional and genetic epidemiology
@ast
Clustered environments and ran ...... ional and genetic epidemiology
@en
Clustered environments and ran ...... ional and genetic epidemiology
@en-gb
Clustered environments and ran ...... ional and genetic epidemiology
@nl
prefLabel
Clustered environments and ran ...... ional and genetic epidemiology
@ast
Clustered environments and ran ...... ional and genetic epidemiology
@en
Clustered environments and ran ...... ional and genetic epidemiology
@en-gb
Clustered environments and ran ...... ional and genetic epidemiology
@nl
P2093
P2860
P50
P3181
P1433
P1476
Clustered environments and ran ...... ional and genetic epidemiology
@en
P2093
Nic Timpson
Shah Ebrahim
P2860
P3181
P356
10.1371/JOURNAL.PMED.0040352
P407
P577
2007-12-01T00:00:00Z