Toll-like receptor 2 impairs host defense in gram-negative sepsis caused by Burkholderia pseudomallei (Melioidosis) - Test2 - elhamkhani - TriplyDB

Toll-like receptor 2 impairs host defense in gram-negative sepsis caused by Burkholderia pseudomallei (Melioidosis)

Toll-like receptor 2 impairs host defense in gram-negative sepsis caused by Burkholderia pseudomallei (Melioidosis)

http://www.wikidata.org/entity/Q28757205

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Mechanisms of Disease: Host-Pathogen Interactions between Burkholderia Species and Lung Epithelial Cells Development of Burkholderia mallei and pseudomallei vaccines Glyburide reduces bacterial dissemination in a mouse model of melioidosis Post-exposure therapeutic efficacy of COX-2 inhibition against Burkholderia pseudomallei The urokinase receptor (uPAR) facilitates clearance of Borrelia burgdorferi Toll-like receptor 4 region genetic variants are associated with susceptibility to melioidosis.The role of NOD2 in murine and human melioidosis Activation of Toll-like receptors by Burkholderia pseudomallei.MyD88 dependent signaling contributes to protective host defense against Burkholderia pseudomallei Expression and function of macrophage migration inhibitory factor (MIF) in melioidosis.Osteopontin impairs host defense during established gram-negative sepsis caused by Burkholderia pseudomallei (melioidosis).A nonsense mutation in TLR5 is associated with survival and reduced IL-10 and TNF-α levels in human melioidosis.Genome wide transcriptome profiling of a murine acute melioidosis model reveals new insights into how Burkholderia pseudomallei overcomes host innate immunity The Burkholderia pseudomallei type III secretion system and BopA are required for evasion of LC3-associated phagocytosis The genetic and molecular basis of O-antigenic diversity in Burkholderia pseudomallei lipopolysaccharide.Overexpression of the endothelial protein C receptor is detrimental during pneumonia-derived gram-negative sepsis (Melioidosis).Divergent functions of Toll-like receptors during bacterial lung infections.Survey of innate immune responses to Burkholderia pseudomallei in human blood identifies a central role for lipopolysaccharide.A thrombomodulin mutation that impairs active protein C generation is detrimental in severe pneumonia-derived gram-negative sepsis (melioidosis).Development of vaccines against burkholderia pseudomallei.NLRC4 and TLR5 each contribute to host defense in respiratory melioidosis.Burkholderia pseudomallei-induced expression of a negative regulator, sterile-alpha and Armadillo motif-containing protein, in mouse macrophages: a possible mechanism for suppression of the MyD88-independent pathway.MyD88-dependent recruitment of monocytes and dendritic cells required for protection from pulmonary Burkholderia mallei infection.T-Cell Responses Are Associated with Survival in Acute Melioidosis Patients.Characterization of the murine macrophage response to infection with virulent and avirulent Burkholderia species.Role of Nucleotide-Binding Oligomerization Domain-Containing (NOD) 2 in Host Defense during Pneumococcal Pneumonia.Differential Toll-Like Receptor-Signalling of Burkholderia pseudomallei Lipopolysaccharide in Murine and Human Models.The Role of TLR2 in Infection and Immunity Burkholderia pseudomallei triggers altered inflammatory profiles in a whole-blood model of type 2 diabetes-melioidosis comorbidity Triggering Receptor Expressed on Myeloid Cells (TREM)-2 Impairs Host Defense in Experimental Melioidosis Toll-Like Receptor Signalling Is Not Involved in Platelet Response to Streptococcus pneumoniae In Vitro or In Vivo.The Blood Transcriptome of Experimental Melioidosis Reflects Disease Severity and Shows Considerable Similarity with the Human Disease The gut microbiota as a modulator of innate immunity during melioidosis Bacterial lipoprotein TLR2 agonists broadly modulate endothelial function and coagulation pathways in vitro and in vivo.Structural diversity of Burkholderia pseudomallei lipopolysaccharides affects innate immune signaling.Neutrophil extracellular traps in the host defense against sepsis induced by Burkholderia pseudomallei (melioidosis)Host gene expression analysis in Sri Lankan melioidosis patients.Sensing gram-negative bacterial lipopolysaccharides: a human disease determinant?Impaired TLR5 functionality is associated with survival in melioidosis Burkholderia pseudomallei Capsule Exacerbates Respiratory Melioidosis but Does Not Afford Protection against Antimicrobial Signaling or Bacterial Killing in Human Olfactory Ensheathing Cells.

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Toll-like receptor 2 impairs host defense in gram-negative sepsis caused by Burkholderia pseudomallei (Melioidosis)

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2007 nî lūn-bûn

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2007 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2007年の論文

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JOURNAL.PMED.0040248

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Toll-like receptor 2 impairs h ...... ria pseudomallei (Melioidosis)

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Alex F de Vos

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Catharina W Wieland

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Mark C Dessing

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Masja Leendertse

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Narisara Chantratita

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Nicholas White

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W Joost Wiersinga

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Wirongrong Chierakul

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Melioidosis: epidemiology, pathophysiology, and management.

Relative quantification of 40 nucleic acid sequences by multiplex ligation-dependent probe amplification

Negative regulation of Toll-like receptor 4 signaling by the Toll-like receptor homolog RP105

Pathogen recognition and innate immunity

Cutting edge: Toll-like receptor 4 (TLR4)-deficient mice are hyporesponsive to lipopolysaccharide: evidence for TLR4 as the Lps gene product

Lipopolysaccharide rapidly traffics to and from the Golgi apparatus with the toll-like receptor 4-MD-2-CD14 complex in a process that is distinct from the initiation of signal transduction

Lipopolysaccharides of Bacteroides fragilis, Chlamydia trachomatis and Pseudomonas aeruginosa signal via toll-like receptor 2

Melioidosis: insights into the pathogenicity of Burkholderia pseudomallei

Cutting edge: TLR2-deficient and MyD88-deficient mice are highly susceptible to Staphylococcus aureus infection

2001 SCCM/ESICM/ACCP/ATS/SIS International Sepsis Definitions Conference

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e248

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Sandrine Florquin

Nicholas P J Day

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Tom van der Poll

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2007-07-31T00:00:00Z

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Burkholderia pseudomallei

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