Neuroinflammatory component of gray matter pathology in multiple sclerosis
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Translocator Protein-18 kDa (TSPO) Positron Emission Tomography (PET) Imaging and Its Clinical Impact in Neurodegenerative DiseasesNeuroinflammation, neurodegeneration and regeneration in multiple sclerosis: intercorrelated manifestations of the immune response.MRI evidence of acute inflammation in leukocortical lesions of patients with early multiple sclerosis.MRI evaluation of thalamic volume differentiates MS from common mimics.The Diverse Roles of Microglia in the Neurodegenerative Aspects of Central Nervous System (CNS) Autoimmunity.Imaging of glia activation in people with primary lateral sclerosis.Heterogeneous pathological processes account for thalamic degeneration in multiple sclerosis: Insights from 7 T imaging.The Effect of Propofol vs. Isoflurane Anesthesia on Postoperative Changes in Cerebrospinal Fluid Cytokine Levels: Results from a Randomized Trial.Reconceptualization of translocator protein as a biomarker of neuroinflammation in psychiatry.Neuroinflammation and its relationship to changes in brain volume and white matter lesions in multiple sclerosis.PET Imaging of Human Brown Adipose Tissue with the TSPO Tracer [(11)C]PBR28.Mapping of thalamic magnetic susceptibility in multiple sclerosis indicates decreasing iron with disease duration: A proposed mechanistic relationship between inflammation and oligodendrocyte vitality.The Role of Advanced Magnetic Resonance Imaging Techniques in Multiple Sclerosis Clinical Trials.[11 C]PBR-28 positron emission tomography in multiple sclerosis: Neuroinflammation or otherwise?Progressive multiple sclerosis patients show substantial lesion activity that correlates with clinical disease severity and sex: a retrospective autopsy cohort analysis.Neuroinflammation of the spinal cord and nerve roots in chronic radicular pain patients.Molecular Magnetic Resonance Imaging of Endothelial Activation in the Central Nervous System.Evaluation of Microglial Activation in Multiple Sclerosis Patients Using Positron Emission Tomography.Microglial activation, white matter tract damage, and disability in MS.Much, if not all, of the cortical damage in MS can be attributed to the microglial cell - Yes.Differential Gray Matter Vulnerability in the 1 Year Following a Clinically Isolated Syndrome
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Neuroinflammatory component of gray matter pathology in multiple sclerosis
description
2016 nî lūn-bûn
@nan
2016 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2016 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
name
Neuroinflammatory component of gray matter pathology in multiple sclerosis
@ast
Neuroinflammatory component of gray matter pathology in multiple sclerosis
@en
type
label
Neuroinflammatory component of gray matter pathology in multiple sclerosis
@ast
Neuroinflammatory component of gray matter pathology in multiple sclerosis
@en
prefLabel
Neuroinflammatory component of gray matter pathology in multiple sclerosis
@ast
Neuroinflammatory component of gray matter pathology in multiple sclerosis
@en
P2093
P2860
P50
P356
P1433
P1476
Neuroinflammatory component of gray matter pathology in multiple sclerosis
@en
P2093
Carolina Ionete
Caterina Mainero
Ciprian Catana
Constantina A Treaba
Céline Louapre
David Izquierdo-Garcia
Elena Herranz
Eric C Klawiter
Gabriel Mangeat
Marco L Loggia
P2860
P304
P356
10.1002/ANA.24791
P577
2016-10-25T00:00:00Z