FOXO1/3 and PTEN Depletion in Granulosa Cells Promotes Ovarian Granulosa Cell Tumor Development.
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Insights into granulosa cell tumors using spontaneous or genetically engineered mouse modelsAmplification of R-spondin1 signaling induces granulosa cell fate defects and cancers in mouse adult ovary.Constitutively active transforming growth factor β receptor 1 in the mouse ovary promotes tumorigenesisMolecular analyses of juvenile granulosa cell tumors bearing AKT1 mutations provide insights into tumor biology and therapeutic leads.Constitutive Activation of PI3K in Oocyte Induces Ovarian Granulosa Cell Tumors.Emx2 as a novel tool to suppress glioblastoma.Effects of PTEN gene silencing on invasion and EMT in oral squamous carcinoma Tca8113 cells.SMAD3 Activation: A Converging Point of Dysregulated TGF-Beta Superfamily Signaling and Genetic Aberrations in Granulosa Cell Tumor Development?Genetics and genomics of ovarian sex cord-stromal tumors.Understanding follicle growth in vitro: Are we getting closer to obtaining mature oocytes from in vitro-grown follicles in human?Primary ovarian insufficiency with t(5;13): a case report and literature review on disrupted genes.G protein-coupled receptors (GPCRs) That Signal via Protein Kinase A (PKA) Cross-talk at Insulin Receptor Substrate 1 (IRS1) to Activate the phosphatidylinositol 3-kinase (PI3K)/AKT Pathway.Lentiviral-Mediated Short Hairpin RNA Knockdown of MTDH Inhibits Cell Growth and Induces Apoptosis by Regulating the PTEN/AKT Pathway in Hepatocellular CarcinomaTGFβ Controls Ovarian Cancer Cell ProliferationSOX9 is a proliferation and stem cell factor in hepatocellular carcinoma and possess widespread prognostic significance in different cancer types.Disruption of postnatal folliculogenesis and development of ovarian tumor in a mouse model with aberrant transforming growth factor beta signaling.MicroRNA expression profiles in non‑epithelial ovarian tumors.The molecular mechanism of ovarian granulosa cell tumors.Global or Granulosa Cell-Specific Pten Mutations in Combination with Elevated FSH Levels Fail to Cause Ovarian Tumours in Mice.Endogenous c-Jun N-terminal kinase (JNK) activity marks the boundary between normal and malignant granulosa cells.MicroRNA-10a promotes granulosa cells tumor development via PTEN-AKT/Wnt regulatory axis
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P2860
FOXO1/3 and PTEN Depletion in Granulosa Cells Promotes Ovarian Granulosa Cell Tumor Development.
description
2015 nî lūn-bûn
@nan
2015 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2015 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
name
FOXO1/3 and PTEN Depletion in ...... nulosa Cell Tumor Development.
@ast
FOXO1/3 and PTEN Depletion in ...... nulosa Cell Tumor Development.
@en
type
label
FOXO1/3 and PTEN Depletion in ...... nulosa Cell Tumor Development.
@ast
FOXO1/3 and PTEN Depletion in ...... nulosa Cell Tumor Development.
@en
prefLabel
FOXO1/3 and PTEN Depletion in ...... nulosa Cell Tumor Development.
@ast
FOXO1/3 and PTEN Depletion in ...... nulosa Cell Tumor Development.
@en
P2093
P2860
P356
P1476
FOXO1/3 and PTEN Depletion in ...... nulosa Cell Tumor Development.
@en
P2093
Diego H Castrillon
Jaye Adams
JoAnne S Richards
Zhilin Liu
P2860
P304
P356
10.1210/ME.2015-1103
P577
2015-06-10T00:00:00Z