Influenza H5N1 and H1N1 virus replication and innate immune responses in bronchial epithelial cells are influenced by the state of differentiation.
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Use of ex vivo and in vitro cultures of the human respiratory tract to study the tropism and host responses of highly pathogenic avian influenza A (H5N1) and other influenza virusesChanges to the dynamic nature of hemagglutinin and the emergence of the 2009 pandemic H1N1 influenza virus.Gene expression signature-based screening identifies new broadly effective influenza a antiviralsApoptosis and the airway epitheliumEarly host responses of seasonal and pandemic influenza A viruses in primary well-differentiated human lung epithelial cellsThe immune profile associated with acute allergic asthma accelerates clearance of influenza virusTranscriptomic characterization of the novel avian-origin influenza A (H7N9) virus: specific host response and responses intermediate between avian (H5N1 and H7N7) and human (H3N2) viruses and implications for treatment optionsHighly pathogenic avian influenza A H5N1 and pandemic H1N1 virus infections have different phenotypes in Toll-like receptor 3 knockout mice.Innate immune sensing and response to influenza.Polymerase discordance in novel swine influenza H3N2v constellations is tolerated in swine but not human respiratory epithelial cellsValidation of normal human bronchial epithelial cells as a model for influenza A infections in human distal tracheaPandemic Influenza A (H1N1) Virus Infection Increases Apoptosis and HIV-1 Replication in HIV-1 Infected Jurkat Cells.A spatial model of the efficiency of T cell search in the influenza-infected lung.1918 Influenza receptor binding domain variants bind and replicate in primary human airway cells regardless of receptor specificity.Inefficient control of host gene expression by the 2009 pandemic H1N1 influenza A virus NS1 proteinHighly pathogenic avian influenza H5N1 virus delays apoptotic responses via activation of STAT3.PB2-Q591K Mutation Determines the Pathogenicity of Avian H9N2 Influenza Viruses for Mammalian SpeciesComparison of pro-inflammatory cytokine expression and cellular signal transduction in human macrophages infected with different influenza A viruses.Higher level of replication efficiency of 2009 (H1N1) pandemic influenza virus than those of seasonal and avian strains: kinetics from epithelial cell culture and computational modelingApoptosis, cytokine and chemokine induction by non-structural 1 (NS1) proteins encoded by different influenza subtypesComparable fitness and transmissibility between oseltamivir-resistant pandemic 2009 and seasonal H1N1 influenza viruses with the H275Y neuraminidase mutation.An overview of the highly pathogenic H5N1 influenza virus.Reassortment between Avian H5N1 and human influenza viruses is mainly restricted to the matrix and neuraminidase gene segments.Jaagsiekte sheep retrovirus infects multiple cell types in the ovine lungEGFR activation suppresses respiratory virus-induced IRF1-dependent CXCL10 productionAvian influenza viruses infect primary human bronchial epithelial cells unconstrained by sialic acid α2,3 residues.The SARS coronavirus E protein interacts with PALS1 and alters tight junction formation and epithelial morphogenesis.Neurovirulence of H5N1 infection in ferrets is mediated by multifocal replication in distinct permissive neuronal cell regionsAmino acid residues 253 and 591 of the PB2 protein of avian influenza virus A H9N2 contribute to mammalian pathogenesis.H5N1 influenza virus pathogenesis in genetically diverse mice is mediated at the level of viral load.Host regulatory network response to infection with highly pathogenic H5N1 avian influenza virus.Comprehensive evaluation of poly(I:C) induced inflammatory response in an airway epithelial modelInterferon Lambda Upregulates IDO1 Expression in Respiratory Epithelial Cells After Influenza Virus Infection.Response of primary human airway epithelial cells to influenza infection: a quantitative proteomic studyCalcium-activated chloride channel TMEM16A modulates mucin secretion and airway smooth muscle contractionInnate immunity to H5N1 influenza viruses in humans.Novel Highly Pathogenic Avian A(H5N2) and A(H5N8) Influenza Viruses of Clade 2.3.4.4 from North America Have Limited Capacity for Replication and Transmission in Mammals.Moving H5N1 studies into the era of systems biology.The differentiated airway epithelium infected by influenza viruses maintains the barrier function despite a dramatic loss of ciliated cells.Cells and Culture Systems Used to Model the Small Airway Epithelium.
P2860
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P2860
Influenza H5N1 and H1N1 virus replication and innate immune responses in bronchial epithelial cells are influenced by the state of differentiation.
description
2010 nî lūn-bûn
@nan
2010 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
name
Influenza H5N1 and H1N1 virus ...... the state of differentiation.
@ast
Influenza H5N1 and H1N1 virus ...... the state of differentiation.
@en
type
label
Influenza H5N1 and H1N1 virus ...... the state of differentiation.
@ast
Influenza H5N1 and H1N1 virus ...... the state of differentiation.
@en
prefLabel
Influenza H5N1 and H1N1 virus ...... the state of differentiation.
@ast
Influenza H5N1 and H1N1 virus ...... the state of differentiation.
@en
P2093
P2860
P1433
P1476
Influenza H5N1 and H1N1 virus ...... the state of differentiation.
@en
P2093
Carol C C Ho
John M Nicholls
Kit M Yuen
Michael C W Chan
Renee W Y Chan
Wendy C L Yu
P2860
P356
10.1371/JOURNAL.PONE.0008713
P407
P50
P577
2010-01-15T00:00:00Z