Dengue Virus Infection Is through a Cooperative Interaction between a Mannose Receptor and CLEC5A on Macrophage as a Multivalent Hetero-Complex.
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Dengue Virus Glycosylation: What Do We Know?Human macrophages differentiated in the presence of vitamin D3 restrict dengue virus infection and innate responses by downregulating mannose receptor expression.Flexible Signaling of Myeloid C-Type Lectin Receptors in Immunity and Inflammation.Innate immune receptors drive dengue virus immune activation and disease.Flavivirus Receptors: Diversity, Identity, and Cell Entry
P2860
Dengue Virus Infection Is through a Cooperative Interaction between a Mannose Receptor and CLEC5A on Macrophage as a Multivalent Hetero-Complex.
description
2016 nî lūn-bûn
@nan
2016 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2016 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
name
Dengue Virus Infection Is thro ...... a Multivalent Hetero-Complex.
@ast
Dengue Virus Infection Is thro ...... a Multivalent Hetero-Complex.
@en
type
label
Dengue Virus Infection Is thro ...... a Multivalent Hetero-Complex.
@ast
Dengue Virus Infection Is thro ...... a Multivalent Hetero-Complex.
@en
prefLabel
Dengue Virus Infection Is thro ...... a Multivalent Hetero-Complex.
@ast
Dengue Virus Infection Is thro ...... a Multivalent Hetero-Complex.
@en
P2093
P2860
P1433
P1476
Dengue Virus Infection Is thro ...... s a Multivalent Hetero-Complex
@en
P2093
Chi-Huey Wong
Jia-Huei Lyu
Tsui-Ling Hsu
Yen-Lung Lo
P2860
P304
P356
10.1371/JOURNAL.PONE.0166474
P407
P577
2016-11-10T00:00:00Z