Reversal of neuropathic pain in diabetes by targeting glycosylation of Ca(V)3.2 T-type calcium channels
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The Physiology, Pathology, and Pharmacology of Voltage-Gated Calcium Channels and Their Future Therapeutic PotentialTargeting of CaV3.2 T-type calcium channels in peripheral sensory neurons for the treatment of painful diabetic neuropathyNew Horizons in Diabetic Neuropathy: Mechanisms, Bioenergetics, and Pain.Is Diabetic Nerve Pain Caused by Dysregulated Ion Channels in Sensory Neurons?Painful Diabetic Neuropathy: Prevention or Suppression?Characterization of novel cannabinoid based T-type calcium channel blockers with analgesic effects.Inflammatory mediator bradykinin increases population of sensory neurons expressing functional T-type Ca(2+) channels.Neuronal calcium signaling in chronic pain.Modulation of nociceptive ion channels and receptors via protein-protein interactions: implications for pain relief.Regulation of the T-type Ca(2+) channel Cav3.2 by hydrogen sulfide: emerging controversies concerning the role of H2 S in nociception.Alleviation of pain in painful diabetic neuropathy.Activity-dependent regulation of T-type calcium channels by submembrane calcium ionsRecent advances in the development of T-type calcium channel blockers for pain intervention.Modulation of Cav3.2 T-type calcium channel permeability by asparagine-linked glycosylation.Location, location, location?: is the pain of diabetic neuropathy generated by hyperactive sensory neurons?Constitutive activity of the Ghrelin receptor reduces surface expression of voltage-gated Ca2+ channels in a CaVβ-dependent manner.Noradrenaline upregulates T-type calcium channels in rat pinealocytes.The tao of IGF-1: insulin-like growth factor receptor activation increases pain by enhancing T-type calcium channel activity.Cooperative roles of glucose and asparagine-linked glycosylation in T-type calcium channel expression.Stroke-Like Episodes and Cerebellar Syndrome in Phosphomannomutase Deficiency (PMM2-CDG): Evidence for Hypoglycosylation-Driven Channelopathy.Continuous subcutaneous insulin infusion preserves axonal function in type 1 diabetes mellitus.Drosophila Insulin receptor regulates the persistence of injury-induced nociceptive sensitization.A Crude 1-DNJ Extract from Home Made Bombyx Batryticatus Inhibits Diabetic Cardiomyopathy-Associated Fibrosis in db/db Mice and Reduces Protein N-Glycosylation Levels.
P2860
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P2860
Reversal of neuropathic pain in diabetes by targeting glycosylation of Ca(V)3.2 T-type calcium channels
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2013 nî lūn-bûn
@nan
2013 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2013年の論文
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2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Reversal of neuropathic pain i ...... (V)3.2 T-type calcium channels
@ast
Reversal of neuropathic pain i ...... (V)3.2 T-type calcium channels
@en
type
label
Reversal of neuropathic pain i ...... (V)3.2 T-type calcium channels
@ast
Reversal of neuropathic pain i ...... (V)3.2 T-type calcium channels
@en
prefLabel
Reversal of neuropathic pain i ...... (V)3.2 T-type calcium channels
@ast
Reversal of neuropathic pain i ...... (V)3.2 T-type calcium channels
@en
P2093
P2860
P50
P356
P1433
P1476
Reversal of neuropathic pain i ...... (V)3.2 T-type calcium channels
@en
P2093
Douglas F Covey
Jeonghan Lee
Jung-Ha Lee
Katiresan Krishnan
Kirstin E Rose
Megan O Jacus
Miljen M Jagodic
Nathan Poiro
Paula Q Barrett
Peihan Orestes
P2860
P304
P356
10.2337/DB13-0813
P407
P577
2013-07-08T00:00:00Z