Interaction between nitric oxide signaling and gap junctions: effects on vascular function
about
Cellular and molecular mechanisms of endothelial ischemia/reperfusion injury: perspectives and implications for postischemic myocardial protectionPannexin channel and connexin hemichannel expression in vascular function and inflammationModulating Vascular Hemodynamics With an Alpha Globin Mimetic Peptide (HbαX).Nitric oxide pretreatment enhances atheroma component highlighting in vivo with intercellular adhesion molecule-1-targeted echogenic liposomes.Traumatic brain injury in vivo and in vitro contributes to cerebral vascular dysfunction through impaired gap junction communication between vascular smooth muscle cellsMutations in cardiovascular connexin genes.Ginkgolide B Inhibits JAM-A, Cx43, and VE-Cadherin Expression and Reduces Monocyte Transmigration in Oxidized LDL-Stimulated Human Umbilical Vein Endothelial Cells.Regulation of gap junction channels and hemichannels by phosphorylation and redox changes: a revision.Association of connexin gene polymorphism with essential hypertension in Kazak and Han Chinese in Xinjiang, China.Regulation of gap junctions by nitric oxide influences the generation of arrhythmias resulting from acute ischemia and reperfusion in vivo.Regulation of cardiovascular connexins by mechanical forces and junctions.Targeting endothelial connexin40 inhibits tumor growth by reducing angiogenesis and improving vessel perfusion.Role of CAPE on cardiomyocyte protection via connexin 43 regulation under hypoxia.Local effects of pregnancy on connexin proteins that mediate Ca2+-associated uterine endothelial NO synthesis.Endothelial control of vasodilation: integration of myoendothelial microdomain signalling and modulation by epoxyeicosatrienoic acids.Connexins in lymphatic vessel physiology and disease.Pannexin-1 channels and their emerging functions in cardiovascular diseases.Compartmentalized nitric oxide signaling in the resistance vasculaturePannexin hemichannels: A novel promising therapy target for oxidative stress related diseases.Communication Is Key: Mechanisms of Intercellular Signaling in Vasodilation.Nitric oxide, PKC-ε, and connexin43 are crucial for ischemic preconditioning-induced chemical gap junction uncoupling.Targeting Pulmonary Endothelial Hemoglobin α Improves Nitric Oxide Signaling and Reverses Pulmonary Artery Endothelial Dysfunction.Identification of Connexin43 Phosphorylation and S-Nitrosylation in Cultured Primary Vascular Cells.Hyperglycaemia disrupts conducted vasodilation in the resistance vasculature of db/db mice.
P2860
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P2860
Interaction between nitric oxide signaling and gap junctions: effects on vascular function
description
2011 nî lūn-bûn
@nan
2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Interaction between nitric oxi ...... : effects on vascular function
@ast
Interaction between nitric oxi ...... : effects on vascular function
@en
type
label
Interaction between nitric oxi ...... : effects on vascular function
@ast
Interaction between nitric oxi ...... : effects on vascular function
@en
prefLabel
Interaction between nitric oxi ...... : effects on vascular function
@ast
Interaction between nitric oxi ...... : effects on vascular function
@en
P2093
P2860
P1476
Interaction between nitric oxi ...... : effects on vascular function
@en
P2093
A C Straub
B E Isakson
R C Looft-Wilson
P2860
P304
P356
10.1016/J.BBAMEM.2011.07.031
P407
P577
2011-07-28T00:00:00Z